Authors

  • Bolta A. Kahorov
    National University of Uzbekistan named after Mirzo Ulugbek, University str., 4, 100174, Tashkent, Uzbekistan
  • Sevara L. Rasulova
    National University of Uzbekistan named after Mirzo Ulugbek, University str., 4, 100174, Tashkent, Uzbekistan

DOI:

https://doi.org/10.37547/ajbspi/Volume03Issue12-09

Keywords:

Toxicity side effects treatment of cancer

Abstract

The results showed that immunomodulin has a moderate regulatory effect on the spontaneous blast transformation of lymphocytes in the in vitro system. The costimulatory effect of immunomodulin in the reaction of PHA-induced blast transformation of T-lymphocytes was revealed. The data we obtained on the rather pronounced interferon-inducing properties of Sanogen developed by us and its combinations with well-known inductors - Cycloferon and Betaleukin, can be of practical use in the treatment of infectious pathology, especially viral hepatitis, when the combination of hepatoprotective, anti-inflammatory, antiviral, immunomodulatory and detoxification mechanisms of action, in the absence of toxicity and side effects, will ensure the development of sanogenetic processes in the patient's body.


background image

Volume 03 Issue 12-2023

48


American Journal Of Biomedical Science & Pharmaceutical Innovation
(ISSN

2771-2753)

VOLUME

03

ISSUE

12

P

AGES

:

48-55

SJIF

I

MPACT

FACTOR

(2021:

5.

705

)

(2022:

5.

705

)

(2023:

6.534

)

OCLC

1121105677















































Publisher:

Oscar Publishing Services

Servi

ABSTRACT

The results showed that immunomodulin has a moderate regulatory effect on the spontaneous blast transformation

of lymphocytes in the in vitro system. The costimulatory effect of immunomodulin in the reaction of PHA-induced

blast transformation of T-lymphocytes was revealed. The data we obtained on the rather pronounced interferon-

inducing properties of Sanogen developed by us and its combinations with well-known inductors - Cycloferon and

Betaleukin, can be of practical use in the treatment of infectious pathology, especially viral hepatitis, when the

combination of hepatoprotective, anti-inflammatory, antiviral, immunomodulatory and detoxification mechanisms of

action, in the absence of toxicity and side effects, will ensure the development of sanogenetic processes in the

patient's div.

KEYWORDS

Toxicity, side effects, treatment of cancer, cells.

INTRODUCTION

Research Article

INFLUENCE OF MODIFIED PEPTIDES FROM THE FETAL THYMUS ON THE
ACTIVITY OF T-LYMPHOCYTES AND NATURAL KILLERS IN
EXPERIMENTAL VIRAL HEPATITIS

Submission Date:

December 15, 2023,

Accepted Date:

December 20, 2023,

Published Date:

December 25, 2023

Crossref doi:

https://doi.org/10.37547/ajbspi/Volume03Issue12-09


Bolta A. Kahorov

National University of Uzbekistan named after Mirzo Ulugbek, University str., 4, 100174, Tashkent, Uzbekistan

Sevara L. Rasulova

National University of Uzbekistan named after Mirzo Ulugbek, University str., 4, 100174, Tashkent, Uzbekistan

Journal

Website:

https://theusajournals.
com/index.php/ajbspi

Copyright:

Original

content from this work
may be used under the
terms of the creative
commons

attributes

4.0 licence.


background image

Volume 03 Issue 12-2023

49


American Journal Of Biomedical Science & Pharmaceutical Innovation
(ISSN

2771-2753)

VOLUME

03

ISSUE

12

P

AGES

:

48-55

SJIF

I

MPACT

FACTOR

(2021:

5.

705

)

(2022:

5.

705

)

(2023:

6.534

)

OCLC

1121105677















































Publisher:

Oscar Publishing Services

Servi

Thymus extracts, incl. thymosin is used in the

treatment of cancer, autoimmune diseases, many

chronic infectious processes, etc. [1,2,10, 28, 37]. The

most important mechanism of action of thymic

peptides is the enhancement of the functional activity

of T-lymphocytes, however, the multi-stage process of

developing an immune response includes the

activation of not only cellular but also humoral

immunity factors, contributing to an increase in the

production

of

specific

antibodies,

cytokines,

inflammatory factors, etc. [7, 16, 22 ]. Natural immunity

is largely determined by killer cells (NK), which play a

decisive protective role in the early stages of viral

aggression [26]. Among the known thymic peptides, a

drug obtained from fetal sheep thymus is particularly

interesting. It consists of 15 peptides. Its

immunocorrective effect was shown in experiments

and clinical observations, in connection with which it

received the name "Immunomodulin". It is mass-

produced in the state of emergency "Immunomed"

(Tashkent) and approved for medical use in Uzbekistan

and Kazakhstan [2].

To increase the immunobiological activity of thymic

peptides, we attempted to combine them with metal

ions, as is the case in thymulin, which circulates in the

bloodstream as a nanopeptide combined with zinc.

The aim of the work is to study the effect of zinc-

modified thymus peptides on the functional activity of

T-lymphocytes and human natural killer cells in the in

vitro system and to evaluate the effectiveness of

interferonogenesis and antiviral action under the

influence of Sanogen, Betaleukin, Cycloferon and their

combinations in the experiment.

METHODS

Determination of the effect of drugs on the

proliferative response of T-lymphocytes. The material

for the study of lymphocyte blastogenesis was

peripheral blood mononuclear cells of 32 patients with

chronic viral hepatitis B aged 20-49 years.

Phytohemagglutinin (PHA) (Sigma) and concanavalin A

(Con A) (Pharmacia) at suboptimal concentrations (10

µg/mL) were used as RBTL activators. Modified and

unmodified peptides were added to the lymphocytes

(1 million/ml) at a final concentration of 0.01 μg/ml in

the test samples. The tablet was incubated at 37ºC for

1 hour, after which the corresponding mitogen was

added to the wells. Only mitogen was added to control

lymphocyte samples. Mitogen was not used in the

study of spontaneous blast transformation of

lymphocytes. After 48 hours, 3H-thymidine was added

to the samples at a concentration of 1 μCi/ml. The

results of the reaction were taken into account 72

hours after the start of cultivation.

To quantify the effect of immunomodulin on the

proliferative response of T-lymphocytes, the impact

index (IV) was used, which was calculated by the

formula:

IV = (Iо –

Ik)/Ik • 10

0%,


background image

Volume 03 Issue 12-2023

50


American Journal Of Biomedical Science & Pharmaceutical Innovation
(ISSN

2771-2753)

VOLUME

03

ISSUE

12

P

AGES

:

48-55

SJIF

I

MPACT

FACTOR

(2021:

5.

705

)

(2022:

5.

705

)

(2023:

6.534

)

OCLC

1121105677















































Publisher:

Oscar Publishing Services

Servi

where: Io - the number of pulses per minute (imp/min)

in the experiment;

Ik - the number of pulses/min in the control.

RESULTS AND DISCUSSION

It was found that the average value of spontaneous

RBTL in patients with hepatitis in control group 1

(without

incubation

with

mitogen

and

immunomodulin) was 280±14 imp/min with a range of

individual fluctuations from 153 to 404 imp/min (Fig. 1).

In the presence of immunomodulin (control 2), the

indicators of spontaneous blastogenesis significantly

increased on average in the group up to 351 ± 26

imp/min with a range of individual fluctuations from

207 to 673 imp/min. The index of drug effect on

spontaneous RBTL was +25.3% (P<0.05). The

introduction of the zinc-modified peptide into the

culture increased the rates of spontaneous

transformation of T-lymphocytes to an average of

415±36 pulses/min. With a range of individual

fluctuations from 248 to 650 imp/min. The index of

drug effect on spontaneous RBTL was +48% (P<0.05

with control 2 and P<0.001 with control 1).

We also studied the functional activity of T-

lymphocytes in terms of their ability to enter the

mitotic cycle under the influence of PHA. It was

established that under the influence of lectin, the blast

transformation of lymphocytes in general in the

control group 1 is (51.4±3.3) x 103 imp/min with

individual fluctuations in indicators from 41 to 74

thousand imp/min.

In control group 2, the mean value of this indicator did

not differ significantly from control 1 and amounted to

57.0 ± 2.4 thousand imp/min with individual values from

42 to 77 thousand imp/min. The impact index of

immunomodulin on average for the group was +11%.

In the main group, the average value of this indicator

significantly differed from control 1 and control 2 and

amounted to 65.0 ± 2.4 thousand imp/min with

individual values from 48 to 86 thousand imp/min. The

impact index of modified immunomodulin averaged

+27% per group (P<0.05 with control 2 and P<0.001 with

control 1).

In the experiment with pre-treatment with the drug

only mononuclear cells, similar results were obtained.

Thus, the incubation of effector cells with

immunomodulin (without target cells) showed a

significant stimulation of the membrane toxic activity

of natural killer cells in all studied groups. In this group

of experiments, 2 controls were used: preliminary

parallel incubation of effector cells only in a nutrient

medium (control 1) and with a peptide (control 2). In

the experimental group, the metallopeptide was

evaluated. Thus, in healthy donors, the EC cytotoxicity

index was 51.2±1.9%; incubation with the modified

peptide increases these values to 65.7±1.6%;

preincubation with immunomodulin activates them up

to 58.3±1.7%. The difference between the experimental


background image

Volume 03 Issue 12-2023

51


American Journal Of Biomedical Science & Pharmaceutical Innovation
(ISSN

2771-2753)

VOLUME

03

ISSUE

12

P

AGES

:

48-55

SJIF

I

MPACT

FACTOR

(2021:

5.

705

)

(2022:

5.

705

)

(2023:

6.534

)

OCLC

1121105677















































Publisher:

Oscar Publishing Services

Servi

values and controls was significant (P<0.05 with

control 2 and P<0.001 with control 1).

Quantitative assessment of the induction of 1PM by

Sanogen, Betaleukin and Cycloferon with separate and

combined administration to experimental animals.

It has been established that Betaleukin induces IFN

production within 96 hours (observation period); the

maximum titer was noted after 12 hours and reached

512 units. The average titer was 118±16 units. (Fig.1, 3).

Sanogen also had quite pronounced interferonogenic

properties - the maximum titer was 128 units. after 24

hours with an average value of 47 ± 4 units (Fig. 1, 3).

When using monopreparations, Cycloferon was the

most effective - the maximum IFN was 1024 units. after

48 hours. Under his influence, the average titer during

a 120-hour observation was 295 ± 35 units. (fig.3)

Fig.1. Dynamics of serum interferon activity in mice after a single separate intraperitoneal injection of Sanogen (C),

Betaleukin (B) and Cycloferon (Cy) in effective doses: 2 μg/kg; 10 ng/kg and 4 µg/kg, respectively.

The introduction of Betaleukin with Cycloferon sharply

increased the activity of serum IFN to an average of

1060 ± 80 units. The expected increase was to be 395

units. (100 units of Betaleukin + 295 units of

Cycloferon), i.e. it turned out to be 2.7 times higher

than the additive value. Synergy was also manifested in

the accumulation of the maximum titer up to 2048

units, which is 1.3 times higher than expected (512 units

+ 1024 units = 1536 units). There was also a shift in the

peak of IFN production for a period of 48 hours. Even

4 hours after the combined administration of inducers,

the IFN activity was high and amounted to 256 units,

while the serum activity remained significant and after

120 hours was 512 units. The synergistic effect for this

period was 32 times higher than the additive one.

0

200

400

600

800

1000

1200

4

12

24

48

72

96

120

Activity titer IFN, units

B

C

Cy


background image

Volume 03 Issue 12-2023

52


American Journal Of Biomedical Science & Pharmaceutical Innovation
(ISSN

2771-2753)

VOLUME

03

ISSUE

12

P

AGES

:

48-55

SJIF

I

MPACT

FACTOR

(2021:

5.

705

)

(2022:

5.

705

)

(2023:

6.534

)

OCLC

1121105677















































Publisher:

Oscar Publishing Services

Servi

The simultaneous administration of Sanogen and

Cycloferon sharply increased the activity of serum IFN

to an average of 805 ± 75 units, since the expected

average increase was to be 323 units. (28 Sanogen units

+ 295 Cycloferon units), it turned out to be 2.5 times

higher than the additive value (Fig. 3). Synergy also

manifested itself in the accumulation of the maximum

IFN titer up to 2048 units, which is 1.8 times higher than

expected (128 units + 1024 units). The peak of IFN

production was recorded for a period of 48 hours,

which corresponded to the period of maximum serum

activity with the introduction of Cycloferon. It should

be noted that already 4 hours after the combined

administration of these inducers, the IFN activity was

the highest - 256 units. (Fig. 2). Despite the early and

very pronounced induction, serum activity remained

significant throughout the entire observation period,

and even after 120 hours it was 256 units. (Fig. 2),

whereas with the separate administration of Sanogen

or Cycloferon, the titers were only 2 units each. and 16

units. (Fig. 1), i.e. the synergistic effect was 14 times

higher than the additive one.

Therefore, with the simultaneous administration of

Sanogen and Cycloferon, a pronounced synergism was

found in the induction of endogenous IFN, since IFN

synthesis was noted, the high activity of which is

recorded in the circulating blood from 4 to 120 hours

with a maximum of 2048 units 48 hours after injection,

1.8 times higher than additive action when used

separately.

To quantify the activity of interferonogenesis under

the influence of monodrugs and their combinations,

we calculated the average values for the duration of

the study for 5 days for each option (Fig. 3). It has been

established that Cycloferon (295±36 units) is the most

active

among

monopreparations.

All

used

combinations of drugs were more powerful

interferonogens compared to it. Under the influence of

Sanogen with Betaleukin, the average activity of

interferonogenesis was 457±45 units, Sanogen with

Cycloferon - 805±56 units, and Betaleukin with

Cycloferon -1060±52 units.


background image

Volume 03 Issue 12-2023

53


American Journal Of Biomedical Science & Pharmaceutical Innovation
(ISSN

2771-2753)

VOLUME

03

ISSUE

12

P

AGES

:

48-55

SJIF

I

MPACT

FACTOR

(2021:

5.

705

)

(2022:

5.

705

)

(2023:

6.534

)

OCLC

1121105677















































Publisher:

Oscar Publishing Services

Servi

Fig.

3. Average values of serum interferon titers for 5 days after separate and combined

administration of Sanogen (C), Betaleukin (B), and Cycloferon (Cy) to mice.

CONCLUSIONS

The fundamental feature of the action of fetal thymus

peptides is the dependence of the severity and

direction of their effects on the initial state of

regulated cells, which contributes to the normalization

of processes that are out of balance.

-thymic peptides combined with zinc have a regulatory

effect on the proliferative activity of T-lymphocytes

through the interaction of their cell receptors with

mitogen and thymus peptides, which ultimately leads

to a cascade synthesis of cytokines, which in turn

modulates the proliferation of T-cells and cytotoxic

activity of natural killers.

- pronounced interferon-inducing properties of

Sanogen developed by us and its combinations with

well-known inductors - Cycloferon and Betaleukin, can

have practical application in the treatment of

infectious pathology, especially viral hepatitis, when

the

combination

of

hepatoprotective,

anti-

inflammatory, antiviral, immunomodulatory and

detoxification mechanisms of action

REFERENCES

1.

Арион В.Я. Иммунологически активные факторы

тимуса // Итоги науки и техники. Серия.

2.

Иммунология.

-

М.

- 1991.-

№9.

-

С. 10

-50.

3.

Гариб Ф.Ю., Гариб В.Ф.

Иммуномодулин.

Ташкент, Из

-

во мед.литературы им. Абу Али ибн

Сино. 2000, 240 с.

4.

Гариб Ф.Ю., Ризопулу А.П., Петрова Т.А.,

Кахоров Б.А., Оценка иммуностимулирующий

эффективности

синтетической

фракции

0

200

400

600

800

1000

1200

C

B

Cy

C+B

C+Cy

B+Cy

Activity titer IFN, units

C

B

Cy

C+B

C+Cy

B+Cy


background image

Volume 03 Issue 12-2023

54


American Journal Of Biomedical Science & Pharmaceutical Innovation
(ISSN

2771-2753)

VOLUME

03

ISSUE

12

P

AGES

:

48-55

SJIF

I

MPACT

FACTOR

(2021:

5.

705

)

(2022:

5.

705

)

(2023:

6.534

)

OCLC

1121105677















































Publisher:

Oscar Publishing Services

Servi

иммуномодулина.

Ж.

«Медицинская

иммунология».

-

Материалы

6

научной

конференции с международным участием «Дни

иммунологии в Санкт

-

Петербурге»

- 2002.-

т.4.

№2. С. 356.

5.

Денеш Л., Хайош, Зпорни Л., Штауб М. Изучение

действия фрагментов тимопоэтина на старых

мышей// Иммунология.

- 1989. -

№3.

- 58-61.-

С

6.

Ершов Ф.И.,

Парфенов В.В. Методические

указания

по

изучению

специфической

активности

интерферонов

и

индукторов

интерферона.

В

кн.

Руководство

по

экспериментальному

(доклиническому)

изучению новых фармакологических веществ.

М.: Ремедиум .

-

2000, С. 281

-286.

7.

Ершов Ф.И., Тазулахова Э.Б. Индукторы

интерферона

-

новое

поколение

иммуномодуляторов//ТеггаМеdica (11са.

-1998.-

2.-

С. 2

-7.

8.

Зозуля А., Клошник Т., Корнеева Р. Клеточная

терапия в косметологии:белково

-

пептидные

комплексы фетальных тканей как действующее

звено

anti

-

age

therapy

в

косметических

средствах // Косметика и медицина.

-2001.-

№4(23).

-

С.32

-39.

9.

Кетлинский С.А., Симбирцев А.С., Воробьев А.А.

Эндогенные

иммуномодуляторы.

-

СПБ:

Гиппократ, 1992.

10.

А.П.Лыков, В.А.Козлов Натуральные киллеры и

гемопоэз //Иммунология.

-2001.-

№1.

-

С. 10

-14.

11.

Чекнев С.Б. Фенотипическая и функциональная

гетерогенность

циркулирующего

пула

естественных киллеров// Иммунология.

- 1999.-

№4.

-

С. 24

-33.

12.

Хаитов Р.М., Гущин И.С., Пинегин Б.В., Зебрев

А.И. Методические указания по изучению

иммунотропноп активности фармакологических

веществ

в

кн.

«Руководство

по

экспериментальному

(доклиническому)

изучению новых фармакологическх веществ.

Москва.

-2000.-

С.257

-263.

13.

Хаитов

Р.М.,

Пинегин

Б.В.

Основные

представления

об

иммунотропных

лекарственных средствах//

Иммунология.

- 1996.

-

№6.

-

С.4

-9.

14.

Хаитов Р.М. Иммунология. Учебник для

студентов/ Хаитов Р.М.2

-

е изд., перераб. И доп..

-

Москва: ГЭОТАР –

Медиа, 2011.

-

528 с.

15.

Хаитов Р.М. Иммунология. Учебник для

студентов/ Хаитов Р.М.2

-

е изд., перераб. И доп..

-

Москва: ГЭОТАР –

Медиа, 2011.

-

528 с.

16.

Хаитов Р.М.,Ильиной Н.И. Аллергология и

иммунология. Национальное руководство / Под

ред.

-

М.: ГЭОТАР

-

Медиа, 2009.

-

659 с.

17.

Хаитов Р.М., Ярилин А.А., Пинегин Б.В.

Иммунология. Атлас: учебное пособие. 2011.

-

624 с.: ил.

18.

Ярилин А.А. Иммунология.

-

М.: ГЭОТАР

-

Медиа,

2010. -

748 с.


background image

Volume 03 Issue 12-2023

55


American Journal Of Biomedical Science & Pharmaceutical Innovation
(ISSN

2771-2753)

VOLUME

03

ISSUE

12

P

AGES

:

48-55

SJIF

I

MPACT

FACTOR

(2021:

5.

705

)

(2022:

5.

705

)

(2023:

6.534

)

OCLC

1121105677















































Publisher:

Oscar Publishing Services

Servi

19.

Bernabei Р, Allione А, Rigamonti L. Regulation of

interferongammareceptor (IFN-Gammar) chains: a

peculiar way to rule the life and death of human

lymphocytes. Eur. Cytokine Nwek. -2001.-12:6-14.

20.

Durantel D, Carrouee-Durantel S, Branza-Nichita N,

Dwek RA, Zitzmann N. Expression of IFN-gamma

and receptor alpha in the peripheral blood of

patients with hronic hepatitis. C.Chin Med J

(Engl).2004 Jan.; 117(1): 79-82.

21.

Hadden J.W Immunostimulation // Immunology

Today.1993.-p Vol.14.-

Р.275

-280.

22.

Goldstein A.L. Clinical potential and application of

thymosin peptides. International Journal on

Immunorehabilitation 5: 9, 1997.

23.

Karelin A.A., Blishchenko E. Yu., Ivanov V.T. A novel

system of peptideric regulation // FEBS Lett. 1998

Vol./-428.N 1-

2.р. 7

-12.

24.

Kuby J.Immunology. Freeman a. Company, New

York, 1997, 664 р.

25.

Low T.L.K., Goldstein A.L. Thymosin fraction 5 and

5A// Methods in enzymology/ Ed.S.P.Golowick,

N.O. Caplan/ Acad Press.- 1985. -

У.Пб.

-

Р.219.

26.

Rinaldi G.C., Rosaria T.M., Jezzi T. Receptors for

thymosin alfa-1 on mouse thymocytes// Cell.

Immunol 1985.Vol. 91..Р

-.289-293.

27.

http://www.rji.ru/immweb.htm

28.

http://immunology.ru

29.

http://www.ncbi.nim.nih.gov

References

Арион В.Я. Иммунологически активные факторы тимуса // Итоги науки и техники. Серия.

Иммунология.- М.- 1991.- №9.- С. 10-50.

Гариб Ф.Ю., Гариб В.Ф. Иммуномодулин. Ташкент, Из-во мед.литературы им. Абу Али ибн Сино. 2000, 240 с.

Гариб Ф.Ю., Ризопулу А.П., Петрова Т.А., Кахоров Б.А., Оценка иммуностимулирующий эффективности синтетической фракции иммуномодулина. Ж. «Медицинская иммунология». -Материалы 6 научной конференции с международным участием «Дни иммунологии в Санкт-Петербурге» - 2002.-т.4. №2. С. 356.

Денеш Л., Хайош, Зпорни Л., Штауб М. Изучение действия фрагментов тимопоэтина на старых мышей// Иммунология.- 1989. - №3. - 58-61.-С

Ершов Ф.И., Парфенов В.В. Методические указания по изучению специфической активности интерферонов и индукторов интерферона. В кн. Руководство по экспериментальному (доклиническому) изучению новых фармакологических веществ. М.: Ремедиум . - 2000, С. 281-286.

Ершов Ф.И., Тазулахова Э.Б. Индукторы интерферона - новое поколение иммуномодуляторов//ТеггаМеdica (11са.-1998.-2.-С. 2-7.

Зозуля А., Клошник Т., Корнеева Р. Клеточная терапия в косметологии:белково-пептидные комплексы фетальных тканей как действующее звено anti-age therapy в косметических средствах // Косметика и медицина.-2001.-№4(23).-С.32-39.

Кетлинский С.А., Симбирцев А.С., Воробьев А.А. Эндогенные иммуномодуляторы.- СПБ: Гиппократ, 1992.

А.П.Лыков, В.А.Козлов Натуральные киллеры и гемопоэз //Иммунология.-2001.-№1.-С. 10-14.

Чекнев С.Б. Фенотипическая и функциональная гетерогенность циркулирующего пула естественных киллеров// Иммунология.- 1999.-№4.-С. 24-33.

Хаитов Р.М., Гущин И.С., Пинегин Б.В., Зебрев А.И. Методические указания по изучению иммунотропноп активности фармакологических веществ в кн. «Руководство по экспериментальному (доклиническому) изучению новых фармакологическх веществ. Москва.-2000.-С.257-263.

Хаитов Р.М., Пинегин Б.В. Основные представления об иммунотропных лекарственных средствах// Иммунология.- 1996. - №6. - С.4-9.

Хаитов Р.М. Иммунология. Учебник для студентов/ Хаитов Р.М.2-е изд., перераб. И доп..- Москва: ГЭОТАР – Медиа, 2011.- 528 с.

Хаитов Р.М. Иммунология. Учебник для студентов/ Хаитов Р.М.2-е изд., перераб. И доп..- Москва: ГЭОТАР – Медиа, 2011.- 528 с.

Хаитов Р.М.,Ильиной Н.И. Аллергология и иммунология. Национальное руководство / Под ред.- М.: ГЭОТАР-Медиа, 2009. - 659 с.

Хаитов Р.М., Ярилин А.А., Пинегин Б.В. Иммунология. Атлас: учебное пособие. 2011. - 624 с.: ил.

Ярилин А.А. Иммунология. - М.: ГЭОТАР-Медиа, 2010. - 748 с.

Bernabei Р, Allione А, Rigamonti L. Regulation of interferongammareceptor (IFN-Gammar) chains: a peculiar way to rule the life and death of human lymphocytes. Eur. Cytokine Nwek. -2001.-12:6-14.

Durantel D, Carrouee-Durantel S, Branza-Nichita N, Dwek RA, Zitzmann N. Expression of IFN-gamma and receptor alpha in the peripheral blood of patients with hronic hepatitis. C.Chin Med J (Engl).2004 Jan.; 117(1): 79-82.

Hadden J.W Immunostimulation // Immunology Today.1993.-p Vol.14.-Р.275-280.

Goldstein A.L. Clinical potential and application of thymosin peptides. International Journal on Immunorehabilitation 5: 9, 1997.

Karelin A.A., Blishchenko E. Yu., Ivanov V.T. A novel system of peptideric regulation // FEBS Lett. 1998 Vol./-428.N 1-2.р. 7-12.

Kuby J.Immunology. Freeman a. Company, New York, 1997, 664 р.

Low T.L.K., Goldstein A.L. Thymosin fraction 5 and 5A// Methods in enzymology/ Ed.S.P.Golowick, N.O. Caplan/ Acad Press.- 1985. -У.Пб.-Р.219.

Rinaldi G.C., Rosaria T.M., Jezzi T. Receptors for thymosin alfa-1 on mouse thymocytes// Cell. Immunol 1985.Vol. 91..Р-.289-293.