Авторы

  • Sarvinoz Fozilova
    Kimyo International University in Tashkent, Uzbekistan
  • Sitora Ozodbekova
    Kimyo International University in Tashkent, Uzbekistan
  • Abdujabbor Adilov
    Kimyo International University in Tashkent, Uzbekistan
  • Behruz Saidmuratov
    Kimyo International University in Tashkent, Uzbekistan
  • Shahlo Musaeva
    Scientific adviser: Kimyo International University in Tashkent, Uzbekistan

DOI:

https://doi.org/10.71337/inlibrary.uz.ejmns.135141

Ключевые слова:

Photodynamic therapy (PDT) 5-aminolevulinic acid (ALA) fiber-optic light cancer treatment cytotoxicity lymphocyte culture Ehrlich tumor cells red light irradiation photosensitizer in vitro model cell viability trypan blue assay non-coherent light phototoxic effect.

Аннотация

The increasing incidence of malignant neoplasms of various localizations is making them an increasingly serious problem for modern medicine, both in clinical practice and in prevention. Unfortunately, the proportion of advanced cases remains high, and tumor recurrences are often poorly responsive to repeated treatment and are accompanied by significant complications. This underscores the urgent need to improve methods for the diagnosis and treatment of cancer


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EURASIAN JOURNAL OF MEDICAL AND

NATURAL SCIENCES

Innovative Academy Research Support Center

IF = 7.921

www.in-academy.uz

Volume 5 Issue 8, August 2025 ISSN 2181-287X

Page 75

EVALUATION OF THE EFFECTIVENESS OF

PHOTODYNAMIC THERAPY USING A NON-COHERENT

LIGHT SOURCE WITH A WAVELENGTH OF 660 NM IN AN

EXPERIMENT

Fozilova Sarvinoz Tursunboy Qizi

E-mail: vionadark84@gmail.com

Ozodbekova Sitora Alisher qizi

E-mail: Sozodbekova@gmail.com

Adilov Abdujabbor Abdukayumovich

E-mail: abdujabboradilov1@gmail.com

Saidmuratov Behruz Salaydin O`g`li

E-mail: Behruzsaidmurodov030@gmail.com

Kimyo International University in Tashkent, Uzbekistan

Musaeva Shahlo Najatovna

Scientific adviser:

E-mail: musayeva.shahlo.83@mail.ru

Kimyo International University in Tashkent, Uzbekistan

https://doi.org/10.5281/zenodo.16942003

ARTICLE INFO

ABSTRACT

Received: 18

th

August 2025

Accepted: 24

th

August 2025

Online: 25

th

August 2025

The increasing incidence of malignant neoplasms of various

localizations is making them an increasingly serious problem
for modern medicine, both in clinical practice and in
prevention. Unfortunately, the proportion of advanced cases
remains high, and tumor recurrences are often poorly
responsive to repeated treatment and are accompanied by
significant complications. This underscores the urgent need to
improve methods for the diagnosis and treatment of cancer.

KEYWORDS

Photodynamic

therapy

(PDT), 5-aminolevulinic acid
(ALA),

fiber-optic

light,

cancer

treatment,

cytotoxicity,

lymphocyte

culture, Ehrlich tumor cells,
red

light

irradiation,

photosensitizer, in vitro
model, cell viability, trypan
blue assay, non-coherent
light, phototoxic effect.

Photodynamic therapy (PDT) is a promising and rapidly developing approach to cancer

treatment. Its progress directly depends on the development of specialized systems generating
light in the 600–660 nm range. An important role is also played by both already known and
new, more effective and accessible photosensitizers, as well as in-depth scientific studies aimed
at revealing the mechanisms of the photodynamic effect. Evaluating the effectiveness of PDT is
complicated by the fact that the photodynamic effect itself manifests minimally, and the death
of tumor cells becomes evident only after a significant period of time.

In cancer treatment, photodynamic therapy (PDT) can be used both radically, with the

goal of complete tumor destruction, and palliatively, to improve the patient's quality of life. This
method is characterized by high selectivity, allowing healthy organs and tissues to be


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preserved, as well as providing a good cosmetic effect. In addition, PDT can be performed
repeatedly without the risk of serious local or systemic complications. Photochemotherapy
represents another promising direction in the diagnosis and treatment of various types of
cancer. This approach uses photosensitizers (external or internal), which, when activated by
light, initiate chemical reactions in biological tissues

Research Objective:

To develop a laboratory (in vitro) model based on cell cultures for studying the effect of

light exposure transmitted through fiber-optic systems in combination with a photosensitizer
– a 10% solution of 5-aminolevulinic acid.

Materials and Methods

Due to the significant number of variables that can influence the course of photodynamic

therapy (PDT), appropriate experimental models were selected. To assess the effectiveness of
PDT, studies were conducted in two directions: in vivo and in vitro. Peripheral blood obtained
from healthy individuals was used as a biological substrate, and isolated cells of Ehrlich's mouse
ascitic tumor were also involved. A specialized device for photodynamic therapy was used to
carry out the therapeutic exposure. The most important component determining the
mechanism of action is the absorption and excitation spectrum of the photosensitizer used.

To create a lymphocyte culture, peripheral blood samples were taken from healthy

volunteers. The culturing process was carried out using the whole blood method, following the
technique developed by Arakaki D.T. and Sparkes R.S. in 1963. The main principle of this
method is as follows: lymphocytes in peripheral blood culture are activated by
phytohemagglutinin (PHA) – a purified mitogen derived from beans. Under the influence of
PHA, lymphocytes begin to actively divide (enter the mitotic cycle) each day. The greatest
number of dividing cells (mitoses) is observed 72 hours after the start of culturing. After this
stage, the cells were incubated with the photosensitizer, 5-aminolevulinic acid, at a
temperature of 37°C for three hours. They were then irradiated with a non-coherent light
source with a wavelength of 660 nm for 30 minutes. At the same time, isolated cells of Ehrlich's
mouse ascitic tumor were irradiated for 20 minutes.

To determine the quantitative indicator of cytotoxicity, the cells were stained with trypan

blue. Then, using light microscopy with 400x magnification, the number of stained (i.e., dead)
cells was counted. For this, the cell suspension was applied as a thick drop onto a microscope
slide and mixed with an equal volume of a 0.1% trypan blue solution.

The value of cytotoxic activity (CTA) was determined using the following formula:

CTA (%) =

𝑨

𝑩

× 100

,

Where

A

– number of dead cells;

B

– total number of examined cells.

Statistical data processing.

Statistical data are presented as M±m (where M – arithmetic mean; m – standard

deviation). For statistical analysis, the software package Statistica 5.0 was used. Comparison of
independent groups was carried out using the Mann–Whitney test (T-test), and for the analysis
of relative indicators, Fisher’s t-test was applied. Differences between groups were considered
statistically significant at a probability level of p < 0.05.


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Research results and their discussion.

In the course of the study, it was found that photodynamic therapy (PDT) causes cell

death, including cancer cells, through direct phototoxic action. To implement this approach, a
portable device using red radiation was developed. Its purpose is the destruction of malignant
tumor cells as part of a comprehensive cancer treatment. The therapeutic effect is achieved by
activating a photosensitized reaction that is triggered by photons of light from the optical
emitter.
The initial study of the new laser device using fiber-optic technology was aimed at studying its
effect on a lymphocyte cell culture from healthy volunteers. It was found that the simultaneous
use of a photosensitizer (5-aminolevulinic acid) and irradiation with non-coherent light at a
wavelength of 660 nm causes a pronounced cytotoxic reaction (56%). This effect was
statistically significantly higher than with the separate application of the photosensitizer (25%)
or light (20%), as well as compared to the control (18%).

Table 1. Exposure to a non-coherent light source with a wavelength of 660 nm on a

culture of lymphocytes from healthy donors

Exposure

Number of viable

cells

Number of dead

cells

Cytotoxic effect

(CTE), %

Control, n = 100

82

18

18 ± 3.86

Photosensitizer (PS), n

= 100

75

25

25 ± 4.35

PS + light irradiation,

n = 100

44

56

56 ± 5.0*

Light irradiation,

n = 100

80

20

20 ± 4.02

*- р<0.05

At the second stage, mouse Ehrlich ascites tumor cells were incubated for 3 hours with a

photosensitizer (5-aminolevulinic acid) at 37°C. Then the cells were subjected to 20 minutes of
irradiation with non-coherent light at a wavelength of 660 nm (see Table 2).

Table 2.Exposure to a non-coherent light source with a wavelength of 660 nm on

isolated Ehrlich ascites tumor cells.

Exposure

Number of viable

cells

Number of dead

cells

Cytotoxic effect

(CTE), %

Control, n = 100

79

21

21 ± 4.1

Photosensitizer (PS), n

= 100

73

27

27 ± 4.5

PS + light irradiation,

n = 100

38

62

62 ± 4.9*

Light irradiation,

n = 100

84

16

16 ± 3.7

*- р<0.05

During the analysis of the effect of non-coherent radiation with a wavelength of 660 nm

and 5-aminolevulinic acid on Ehrlich ascites tumor cells, it was found that monotherapy with


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each of these factors caused minimal toxic response. The combined use of the photosensitizer
and irradiation resulted in a pronounced cytotoxic effect, reaching 62%.

In the next stage of the study, a model of an experimental Ehrlich sarcoma strain in mice

was used. Three animals with implanted Ehrlich tumors were intraperitoneally injected with
0.5 ml of a 5-aminolevulinic acid solution (concentration of 30 µg/ml in sterile water for
injection) 3 and 20 hours before the start of irradiation (see Tables 3 and 4)

Table 3. Exposure to a non-coherent light source with a wavelength of 660 nm on

Ehrlich ascites tumor cells 3 hours after the administration of the photosensitizer.

Exposure

Number of

viable cells

Number of dead

cells

Cytotoxic effect

(CTE), %

Control, n = 100

97

3

3 ± 1.7

PS + light irradiation, n =

300 cell/ 3 animals

101

199

66.3 ± 2.7*

*- р<0.05
Table 4. Exposure to a non-coherent light source with a wavelength of 660 nm on

Ehrlich ascites tumor cells 20 hours after the administration of the photosensitizer.

Exposure

Number of

viable cells

Number of dead

cells

Cytotoxic effect

(CTE), %

Control, n = 100

96

4

4 ± 3.9

PS + light irradiation, n =

300 cell/ 3 animals

117

183

61.0 ± 2.8*

*- р<0.05

It was found that exposure to a non-coherent light source (660 nm, 30 minutes) and 5-

aminolevulinic acid had a significant toxic effect on Ehrlich ascites tumor cells, causing the
death of 66.3% and 61.0% of the cells, respectively. The duration of the preliminary exposure
to the photosensitizer (3 or 20 hours) did not affect the degree of toxicity.

Conclusions

1.The experiment on lymphocyte culture showed that monotherapy with the

photosensitizer (5-aminolevulinic acid) or standalone exposure to a non-coherent light source
with a wavelength of 660 nm caused minimal toxicity. Combined exposure to the
photosensitizer and light resulted in 56% cytotoxicity.

2.The experiment with Ehrlich ascites tumor cells revealed that both the use of 5-

aminolevulinic acid as a photosensitizer and irradiation with non-coherent light at 660 nm
individually caused minimal toxic effects. However, combined exposure to the photosensitizer
and light led to a cytotoxic effect of 62%.

3.The study demonstrated that 30-minute exposure to non-coherent light with a

wavelength of 660 nm, in combination with the photosensitizer 5-aminolevulinic acid, caused
significant toxicity (66.3% and 61.0%, respectively) in Ehrlich ascites tumor cells. This effect
was observed regardless of whether the cells were incubated with the photosensitizer for 3 or
20 hours.


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