Authors

  • Nozimakhon Saidmukhtorova
    Tashkent Medical University
  • Khurshida Maksudova
    Tashkent Medical University

DOI:

https://doi.org/10.71337/inlibrary.uz.ijms.114420

Abstract

Acute cerebrovascular accident (CVA) continues to be one of the leading causes of disability and mortality in the world. Along with the restoration of brain perfusion, the urgent task is to protect neurons from secondary damage. The article discusses modern approaches to neuroprotective therapy, including drugs with proven efficacy, new pharmacological targets, as well as promising areas such as the use of neuropeptides, antioxidants and cellular technologies. Particular attention is paid to multifactorial therapy and individualization of treatment for patients with CVA. The review is based on recent data, including recommendations from international communities.

 

 

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NEW APPROACHES TO NEUROPROTECTIVE THERAPY OF ACUTE

CEREBROVASCULAR ACCIDENT

Saidmukhtorova Nozimakhon Azizkhon kizi,

Maksudova Khurshida Nabievna

Tashkent Medical University

Abstract:

Acute cerebrovascular accident (CVA) continues to be one of the leading causes

of disability and mortality in the world. Along with the restoration of brain perfusion, the

urgent task is to protect neurons from secondary damage. The article discusses modern

approaches to neuroprotective therapy, including drugs with proven efficacy, new

pharmacological targets, as well as promising areas such as the use of neuropeptides,

antioxidants and cellular technologies. Particular attention is paid to multifactorial therapy

and individualization of treatment for patients with CVA. The review is based on recent data,

including recommendations from international communities.

Keywords:

stroke, neuroprotection, CVA, cerebroprotection, antioxidants, recovery,

neuroplasticity.

Introduction

Acute cerebrovascular accident (CVA) is not only a medical problem, but also a socio-

economic problem due to the high rate of disability and mortality [1]. Despite advances in

the development of emergency care methods, including thrombolysis and mechanical

thrombectomy, the limited "therapeutic window" and the impossibility of using these

methods in most patients make the development of neuroprotective strategies relevant [2].

Neuroprotection in stroke is aimed at preventing secondary neuronal damage resulting from

ischemia, reperfusion syndrome, inflammation and oxidative stress. The modern concept of

CVA therapy includes not only the restoration of blood flow, but also the protection of

neurons, oligodendrocytes, endothelial cells and glial matrix [3].

Modern directions of neuroprotection

Modern approaches to neuroprotective therapy for CVA are aimed at multicomponent

intervention in the cascade of pathophysiological processes initiated by ischemia or

hemorrhage. The main directions are:

• Decreasing the excitability of the glutamate system - due to NMDA receptor antagonists,

such as memantine, which prevents calcium overload of neurons [3];

• Antioxidant protection - drugs that neutralize free radicals (for example,

ethylmethylhydroxypyridine succinate) reduce oxidative stress and damage to cell

membranes [4];


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• Modulation of apoptosis - the use of agents that affect the expression of anti-apoptotic

proteins and mitochondrial function, such as citicoline [5];

• Anti-inflammatory effect - suppression of proinflammatory cytokines and microglia

activation (for example, with edarvon or glimepirides) can prevent remote neuronal damage

[6];

• Stabilization of the blood-brain barrier - prevention of cerebral edema with osmotic

diuretics and steroids can significantly reduce mortality in hemorrhagic strokes [7].

In addition, the role of neurohormonal factors such as erythropoietin, as well as stem cells

capable of inducing the restoration of damaged brain structures, is being actively studied.

Clinical data on the effectiveness of new neuroprotectors

In recent years, new drugs with neuroprotective action aimed at improving outcomes in

patients with stroke have been actively used and studied in clinical practice. The following

drugs have the greatest evidence base in this area:

• Citicoline is a metabolic neuroprotector involved in the synthesis of phosphatidylcholine, a

key component of the cell membrane. Multicenter studies, including ICTUS and COBRIT,

have demonstrated a moderate but significant improvement in cognitive and motor functions

in patients after ischemic stroke with long-term use of citicoline [8].

• Actovegin is a deproteinized hemodialysate of calf blood with antihypoxic and antioxidant

effects. It is used in the acute and recovery period of stroke, helping to reduce the severity of

neurological deficits [9]. • Semax is a synthetic analogue of adrenocorticotropic hormone,

exhibiting neuromodulatory, antioxidant and anti-inflammatory effects. It is used in the

complex therapy of ischemic strokes in Russia, showing encouraging results in terms of

restoring cognitive function and rehabilitation [10].

• Gliatilin (choline alfoscerate) - improves phospholipid metabolism in neuronal membranes

and has a moderate anticholinesterase effect. In combination with rehabilitation measures, it

helps to accelerate the restoration of neuropsychological functions [11].

• Mildronate (meldonium) is a drug with a pronounced metabolic effect, improves the

energy supply of neurons in ischemic conditions, although its effectiveness in strokes

remains a subject of debate [12].

Promising results have also been demonstrated by combination therapy regimens, including

the use of antioxidants, nootropics, as well as the use of hypothermia and early rehabilitation

methods.

Prognosis and rehabilitation of patients


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The prognosis for stroke directly depends on the timeliness of care, the extent of brain tissue

damage, the patient's age and concomitant diseases. Despite advances in neuroprotection,

mortality remains high - up to 20-30% for ischemic stroke and over 40% for hemorrhagic

stroke [13].

Early and comprehensive rehabilitation in combination with neuroprotective therapy plays a

key role in improving outcomes. The main areas of rehabilitation include:

• Physical therapy and kinesitherapy aimed at restoring motor activity;

• Speech therapy correction of speech disorders;

• Psychological and cognitive rehabilitation;

• Neuropsychological support and correction of mood disorders, including treatment of post-

stroke depression.

The effectiveness of rehabilitation increases with the use of modern technologies: robotic

systems, functional electrical stimulation and telerehabilitation.

Conclusion

Modern neuroprotective therapy for acute cerebrovascular accident is based on a

pathogenetically substantiated approach and includes the use of drugs with antioxidant, anti-

apoptotic, anti-inflammatory and metabolic effects. Despite the presence of a large number

of clinical studies, not all methods have a high evidence base.

Further randomized studies are needed to study the efficacy and safety of neuroprotective

drugs in the acute period of stroke, as well as the integration of pharmacotherapy with

individualized rehabilitation programs.

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3. Rothman S.M., Olney J.W. Glutamate and the pathophysiology of hypoxic–ischemic

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Вестник восстановительной медицины. — 2020. — № 4. — С. 14–18.

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13. Feigin V.L., Norrving B., Mensah G.A. Global burden of stroke // Circ Res. — 2017. —

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References

Бабияк В.В., Демченко И.А. Инсульт: современные представления о патогенезе и лечении. — М.: ГЭОТАР-Медиа, 2020. — 304 с.

Камчатнов П.Р. Нейропротекция: перспективы и ограничения // Вопросы современной медицины. — 2021. — № 3. — С. 25–31.

Rothman S.M., Olney J.W. Glutamate and the pathophysiology of hypoxic–ischemic brain damage // Annals of Neurology. — 1986. — Vol. 19, № 2. — P. 105–111.

Simonyan E.Yu. Оксидативный стресс при инсульте и возможности его коррекции // Неврологический журнал. — 2020. — Т. 25, № 1. — С. 12–17.

Secades J.J. Citicoline: pharmacological and clinical review, 2010 update // Revista de Neurología. — 2011. — Vol. 52, Suppl 2. — P. S1–S62.

Shichita T., Sakaguchi R., Suzuki M., Yoshimura A. Post-ischemic inflammation in the brain // Frontiers in Immunology. — 2012. — Vol. 3. — P. 132.

Иванов Д.А., Куценко А.И. Нарушения ГЭБ при инсультах и возможности фармакологической коррекции // Журнал неврологии и психиатрии. — 2021. — № 7. — С. 34–38.

Martí-Vilalta J.L., Katsnelson M. Cognitive function in ischemic stroke: the role of citicoline // Revista de Neurología. — 2006. — Vol. 42, Suppl 3. — P. S59–S63.

Захарова Н.Е. Актовегин в лечении постинсультных когнитивных нарушений // Вестник восстановительной медицины. — 2020. — № 4. — С. 14–18.

Гуляев С.А., Мартынов М.Ю. Применение семакса при ишемическом инсульте // Неврология, нейропсихиатрия, психосоматика. — 2019. — Т. 11, № 2. — С. 56–61.

Fabrizi G.M., Cavallaro T., Angeleri F. Choline alfoscerate (GPC) in cognitive decline and stroke recovery // Clin Ther. — 2005. — Vol. 27. — P. 292–302.

Vasiuk I., Dushanin S., Butko Y. Meldonium in stroke rehabilitation: clinical experience // Acta Medica. — 2019. — Vol. 65, № 4. — P. 170–175.

Feigin V.L., Norrving B., Mensah G.A. Global burden of stroke // Circ Res. — 2017. — Vol. 120, № 3. — P. 439–448.