Authors

  • Dadajonov Sh.N.
    Main Medical Directorate Under The Administration Of The President Of The Republic Of Uzbekistan, Central Clinical Hospital N. 2, Tashkent, Uzbekistan
  • Usmanov Z.H.
    Main Medical Directorate Under The Administration Of The President Of The Republic Of Uzbekistan, Central Clinical Hospital N. 2, Tashkent, Uzbekistan
  • Quziev Z.N.
    Main Medical Directorate Under The Administration Of The President Of The Republic Of Uzbekistan, Central Clinical Hospital N. 2, Tashkent, Uzbekistan

DOI:

https://doi.org/10.37547/ijmscr/Volume03Issue11-10

Keywords:

Diabetes diabetic ketoacidosis intensive care

Abstract

The aim of our work was to develop a comprehensive, pathogenetic substantiated intensive care in patients with diabetic ketoacidosis who are in critical condition. The development of complex pathogenetic substantiated intensive care in patients with diabetic ketoacidosis who are in critical condition is one of the urgent problems of intensive care. It should be noted that the proposed intensive care with solutions containing succinate and potassium in the complex intensive care of diabetic ketoacidosis is one of the promising directions for optimizing the treatment of patients. The intensive care option we proposed contributed over 3 days in 95% of patients to the elimination of diabetic hyperglycemic ketoacidosis.


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Volume 03 Issue 11-2023

96


International Journal of Medical Sciences And Clinical Research
(ISSN

2771-2265)

VOLUME

03

ISSUE

11

P

AGES

:

96-102

SJIF

I

MPACT

FACTOR

(2021:

5.

694

)

(2022:

5.

893

)

(2023:

6.

184

)

OCLC

1121105677















































Publisher:

Oscar Publishing Services

Servi

ABSTRACT

The aim of our work was to develop a comprehensive, pathogenetic substantiated intensive care in patients with

diabetic ketoacidosis who are in critical condition. The development of complex pathogenetic substantiated intensive

care in patients with diabetic ketoacidosis who are in critical condition is one of the urgent problems of intensive care.

It should be noted that the proposed intensive care with solutions containing succinate and potassium in the complex

intensive care of diabetic ketoacidosis is one of the promising directions for optimizing the treatment of patients. The

intensive care option we proposed contributed over 3 days in 95% of patients to the elimination of diabetic

hyperglycemic ketoacidosis.

KEYWORDS

Diabetes, diabetic ketoacidosis, intensive care, succinate.

INTRODUCTION

Research Article

SOME FEATURES OF INTENSIVE THERAPY FOR DIABETIC KETOACIDOSIS

Submission Date:

November 18, 2023,

Accepted Date:

November 23, 2023,

Published Date:

November 28, 2023

Crossref doi:

https://doi.org/10.37547/ijmscr/Volume03Issue11-10


Dadajonov Sh.N.

Main Medical Directorate Under The Administration Of The President Of The Republic Of Uzbekistan, Central
Clinical Hospital N. 2, Tashkent, Uzbekistan


Usmanov Z.H.

Main Medical Directorate Under The Administration Of The President Of The Republic Of Uzbekistan, Central
Clinical Hospital N. 2, Tashkent, Uzbekistan

Quziev Z.N.

Main Medical Directorate Under The Administration Of The President Of The Republic Of Uzbekistan, Central
Clinical Hospital N. 2, Tashkent, Uzbekistan

Journal

Website:

https://theusajournals.
com/index.php/ijmscr

Copyright:

Original

content from this work
may be used under the
terms of the creative
commons

attributes

4.0 licence.


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Diabetes mellitus (DM) is one of the most common

endocrine diseases worldwide. One of the severe

complications of diabetes mellitus is diabetic

ketoacidosis and hyperglycemic ketoacidotic coma,

which require a special approach, both due to the

severity of the course and due to high mortality. High

mortality in hyperglycemic ketoacidotic coma is

associated with insulin insufficiency (1,4,6), tissue

hypoxia, endogenous intoxication, water-electrolyte

imbalance, metabolic disorders, multiple organ

dysfunction (2,3,5). Most clinicians distinguish

hypovolemia and concomitant systemic hypoperfusion

as one of the leading links in the pathogenesis of

systemic and organ insufficiency in patients with

diabetic ketoacidosis and hyperglycemic ketoacidotic

coma (2,6).

The leading role in the correction of hypovolemia is

played by infusion therapy aimed at a rapid increase in

the volume of circulating blood, cardiac output,

oxygen delivery and its consumption by div tissues.

Its optimal choice, along with insulin therapy and other

means of pharmacological correction, can prevent the

development of a critical condition and multiple organ

dysfunction in patients with diabetic ketoacidosis and

hyperglycemic ketoacidotic coma. All of the above

indicates the relevance and clinical significance of the

problem under consideration, since timely diagnosis

and adequate intensive therapy of diabetic

ketoacidosis and hyperglycemic ketoacidotic coma can

not only significantly improve the results of treatment,

but also the outcome of the disease as a whole. The

aim of our work is to develop a comprehensive, well-

founded intensive therapy in patients with diabetes

mellitus complicated by diabetic ketoacidosis and

ketoacidotic coma.

Materials and methods of research. 65 patients with

diabetes mellitus, in critical condition, complicated by

diabetic ketoacidosis and hyperglycemic ketoacidotic

coma, who underwent inpatient treatment in the

intensive care unit, were examined. The study included

patients aged 35 to 60 years (average age 55 + 1.2

years). Of these, 37 female and 28 male patients. The

duration of the disease ranged from 3 to 15 years. To

determine the severity of the patients' condition, the

parameters of the central hemodynamics of volemic

parameters (Heart rate, Systemic Scleroderma, Stroke

Volume (SV), Cardiac index CI, Volume of circulating

blood VCB, volume of circulating plasma (VCP) and

volume of circulating erythrocytes (VCE) were studied.

To optimize infusion therapy, the main and control

groups of patients were formed. In the main group, in

accordance with the severity of the patients' condition

(n=35), pathogenetic-based infusion programs were

used, balanced in qualitative and quantitative

composition. The volume and optimal ratio of saline,

salt-free and colloidal solutions calculated according to

the clinical severity of the condition and the div

weight of patients were selected. In patients of the


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control group (n=30), a generally accepted classical

infusion program was used as part of infusion therapy.

All clinical and laboratory studies were carried out at

the following stages before the start of therapy, after

2 hours, 6 hours, on the 2nd, 3rd day.

Results of research patients with diabetes mellitus in

critical condition complicated by diabetic ketoacidosis

and hyperglycemic ketoacidotic coma revealed

changes in central hemodynamics, indicating a

breakdown of compensatory mechanisms of the

circulatory system, a sharp increase in cardiovascular

insufficiency due to a combination of low cardiac

output, hypovolemia and dehydration. Tachycardia

was observed in patients with stable arterial pressure,

with an average intake of 90.2+1.2 beats/min, ADP

(average dynamic pressure) 110 + 1.7 mmHg, CI (cardiac

index) 24.4+1.2 ml x m, SI (cardiac index) 3.0+0.8 l l x

min. In 97% of patients, there was a decrease in the

volume of circulating blood (VCB) due to a decrease in

the volume of circulating plasma (VCP) and a decrease

in the volume of circulating erythrocytes (VCE).

A significant decrease in VCB was observed by 45.7%

(p<0.05). When patients breathed, the smell of

acetone was clearly detected, hard breathing without

wheezing. In 59% (38) of patients with palpation of the

abdomen, pain was observed during palpation. The

urine test for acetone in all patients was positive and

sharply positive. Decompensated metabolic acidosis

was detected in all patients, with an average blood pH

of 7.22 +0.3 in all patients. Modern intensive therapy

for diabetes mellitus in a critical condition complicated

by

diabetic

ketoacidosis

and

hyperglycemic

ketoacidotic

coma

includes

two

mandatory

components. This is a subsidy of fluid with

compensation for its deficiency and correction of

current pathological losses of the water-electrolyte

composition of the blood, as well as insulin therapy

(Alexandrovich Yu.S.). Patients of the main and control

groups underwent standard insulin therapy to correct

blood glycemia. The dose of insulin and the rate of its

administration are selected in such a way that the rate

of glucose reduction does not exceed 3.5-5.5 mmol / l /

hour or 10% of the initial values. Rehydration therapy

with salt and salt-free solutions is carried out strictly

under the control of CVP and hourly diuresis. Patients

of the control group received standard infusion

therapy with saline solutions: 0.9% sodium chloride

solution or Ringer's solution, 0.45% sodium chloride

solution were used. The concentration of sodium

chloride (0.9 or 0.45%) was selected depending on the

concentration of sodium in the blood plasma. With

normonatremia,

0.9%

are

used,

and

with

hypernatremia, 0.45%.

It was mandatory in infusion therapy to pay attention

to the correction of potassium deficiency, since its

deficiency can lead to the development of cardiac

arrhythmias. Correction of potassium deficiency was

carried out by intravenous drip administration of


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potassium chloride solution. Polycomponent and

multifunctional crystalloid Succinasol solution was

used in patients of the main group in the intensive care

complex in order to correct acidosis and detoxification.

Succinasol is based on sodium succinate, which

supplies succinic acid anion - a substrate of the Krebs

cycle and a supplier of energy-rich compounds, with a

pH of 6.97. Its main property is the ability to influence

the electrolyte composition and volume of the

extracellular and intracellular fluid, as well as the acid-

base state (ABS). This solution is able to have an effect

on not only hemodynamics, water-electrolyte

composition and ABS, but also improves blood

microcirculation,

activates

energy

metabolism,

improves the function of the heart muscle increases

energy production by including fumarate ion in the

Krebs cycle. To correct severe hypovolemia, we

prescribed this solution 200-400 ml, 1-2 times a day.

One of the problems in the correction of diabetic

ketoacidosis is the stabilization of blood levels of

potassium and glucose. After lowering blood glucose

to 14-16 mmol / l, Cadence solution was included in the

infusion in order to provide parenteral carbohydrate

nutrition and regulation of salt balance with the

addition of short-acting insulin (SAI) against the

background of insulin therapy. Since potassium

deficiency occurs especially in diabetes mellitus

complicated

by

diabetic

ketoacidosis

and

hyperglycemic ketoacidotic coma. The use of ready-

made potassium solutions reduces the likelihood of

errors in the dosage of potassium, which can be

observed when preparing solutions from ampoule and

pharmacy concentrates, thereby preventing the risk of

potassium overdose and the development of

hyperkalemia. The ability of this infusion drug to

increase potassium levels without the risk of

hyperkalemia distinguishes it from other potassium-

containing drugs. Dosage: 10-20 mmol of potassium

(0.5-1.0 L), administered within 1-3 hours. The dosage

of Cadence solution depends on the age, div weight,

severity of the clinical condition of the patient and the

indications of laboratory tests. When conducting a

rehydration program in patients in the main group, an

improvement in the indicators of the water-electrolyte

balance of the blood was revealed. As can be seen from

the table, patients of the main group had an increase in

the Na+ content in blood plasma by 3.6% on day 1, and

by 6.5% on day 2. No change in Na+ was observed in

blood plasma in patients of the control group on days

1 and 2. Such normalization of the electrolyte

composition of the blood in patients of the main group

led to an improvement in the indicators of central

hemodynamics.

In the main group, the values of the parameters of

central hemodynamics improved in the first days. This

was confirmed by a significant decrease in heart rate

by 5% after 1 day, SSD by 13%, an increase in CI by 24%,

SV by 14%. It should be noted that the Specific

peripheral resistance SPR decreased by 50%. On the


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second day of observation, there was an improvement

in CG indicators, which are reflected in the table. In

patients of the control group, against the background

of treatment according to the standard algorithm, the

parameters of the CI, SV did not differ significantly

from the initial values. The heart rate remained at the

same level. The indicators of the study results

presented in Table c show that, in the control group,

against the background of traditional infusion therapy,

achieving the target level of glycaemia was

accompanied by a significant decrease in this indicator

on day 1 to 52.4%, and on day 2 to 48.8%. In patients of

the main group, patients have a smooth decrease in

glycaemia, by 60 and 68%. According to the data

obtained, patients with hyperglycemic ketoacidosis in

critical condition have volemic disorders associated

with a decrease in VCB, VCP and a decrease in VCE.

Despite the therapy, during rehydration therapy, the

control group patients showed an increase in VCB by

only 15%, VCP by 24%, and VCE did not undergo

significant changes. In the patients of the main group,

there was an increase in VCB by 39% on day 1, and on

day 2 by 60%, while the increase in VCP by 2 times,

respectively by 54.5% and 71%, indicating the

effectiveness of the therapy. This allowed to correct

the VCB deficiency, stabilize blood circulation, creating

optimal conditions for circulation.

It should be noted that the recovery of pH was observed on the 2nd day of treatment, in 42% of patients of the main

group, this indicator began to increase. And on the 2nd day of treatment, 89% began to meet the norm. There was a

connection between changes in the pH of blood and urine during treatment. Elimination of diabetic hyperglycemic

0%

10%

20%

30%

40%

50%

60%

70%

80%

90%

100%

Na+,

mmol/l

К+,

mmol/l

Glucose,

mmol/l

Heart

rate,

min"1

SV,

mmHg.

CI, ml x

m"2

CI, l x

min"1

х

m"2

SPR,

din/s

х

sm"5

х

m"2

VCB,

ml/kg

VCP,

ml/kg

VCE,

ml/kg

Hemodynamic parameters in patients in the control

group

Control group In admission

Control group After 24 hours

Control group After 48 hours


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ketoacidosis was observed in all patients of the main group within 3 days. 5% of patients in the main group had a

relapse of diabetic ketoacidosis.

The cause was chronic renal failure, pneumonia was

observed in 7% of patients. Mortality of patients in the

main group was not observed. All patients of the main

group were transferred to the endocrinological and

therapeutic departments. The average duration of stay

of patients of the main group in the ICU was 2.65 ± 0.12

days, in patients of the control group 3.45 ± 0.18 days.

Thus, based on the above, it can be assumed that the

developed intensive therapy of solutions containing

succinate and Cadence in the complex therapy of

diabetic ketoacidosis is one of the promising directions

for optimizing the treatment of patients. And also

significantly contribute to reducing the length of stay

of patients in the ICU and reducing the mortality rate.

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Aleksandrovich YU.S. Intensivnaya terapiya

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SJIF

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MPACT

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Kriticheskiye sostoyaniya v klinicheskoy praktike. // S. A. Rumyantseva, V. A. Stupin,V. V. Afanas'yev, A. I. Fedin, Ye. V. Silina — M.: MIG «Meditsinskaya kniga»;2010. — 640 s. Critical conditions in clinical practice. // S. A. Rumyantseva, V. A. Stupin, V. V. Afanasiev, A. I. Fedin, E. V. Silina - M .: MIG "Medical Book"; 2010. — 640 p.

Potemkin, V.V.3. Diabeticheskiy ketoatsidoz / V.V. Potemkin, Ye.G. Starostina // Neotlozhnaya endokrinologiya: rukovodstvo dlya vrachey. — M.: Meditsinskoye informatsionnoye agentstvo, 2008. — S.11—125, 365—387. Potemkin, V.V.3. Diabetic ketoacidosis / V.V. Potemkin, E.G. Starostina // Urgent endocrinology: a guide for physicians. - M .: Medical Information Agency, 2008. - P. 11-125, 365-387.

Rabochaya gruppa po diabetu i SSZ Yevropeyskogo obshchestva kardiologov i Yevropeyskoy assotsiatsii po izucheniyu sakharnogo diabeta, 2007 g. Rekomendatsii po lecheniyu sakharnogo diabeta, prediabeta i serdechno-sosudistykh zabolevaniy // Sakharnyy diabet, 2008. №1. S. 86-92. Diabetes and CVD Working Group of the European Society of Cardiology and the European Association for the Study of Diabetes Mellitus, 2007. Recommendations for the treatment of diabetes mellitus, prediabetes and cardiovascular diseases // Diabetes mellitus, 2008. No. 1. pp. 86-92.

Brown, T.B.4. Cerebral oedema in childhood diabetic ketoacidosis: Is treatment a factor? / T.B. Brown // Emerg. Med. J. — 2004. — № 21. — Р.141—144.

Wolfsdorf, J.5. Diabetic ketoacidosis in infants, children and adolescents: A consensus statement from the American Diabetes Association / J. Wolfsdorf, N. Glazer, M.A. Sperling // Diabetes Care. — 2006. — № 29. — Р.1150—1159.