Authors

  • Kakhkharova N.Kh.
    Republican Specialized Scientific And Practical Medical Center Of Hematology, Uzbekistan
  • Kayumov A.A.
    Republican Specialized Scientific And Practical Medical Center Of Hematology, Uzbekistan

DOI:

https://doi.org/10.37547/ijmscr/Volume03Issue11-06

Keywords:

TLR6 gene complications prognosis

Abstract

This article discusses the significance of the TLR6 gene in the complications of myeloma, a malignant tumor disease that develops from bone marrow plasma cells. Studies have shown that increased TLR6 gene expression in myeloma patients is associated with more severe complications and a worse disease prognosis. This may be due to increased inflammation in the body and increased activation of the immune system. Understanding the role of the TLR6 gene may help develop new approaches to the diagnosis, treatment and prevention of myeloma.


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ABSTRACT

This article discusses the significance of the TLR6 gene in the complications of myeloma, a malignant tumor disease

that develops from bone marrow plasma cells. Studies have shown that increased TLR6 gene expression in myeloma

patients is associated with more severe complications and a worse disease prognosis. This may be due to increased

inflammation in the div and increased activation of the immune system. Understanding the role of the TLR6 gene

may help develop new approaches to the diagnosis, treatment and prevention of myeloma.

KEYWORDS

TLR6 gene, myeloma, complications, prognosis, inflammation, activation of the immune system, diagnosis, treatment,

prevention.

INTRODUCTION

Myeloma is a malignant tumor that develops from

plasma cells in the bone marrow. Complications of

myeloma can include various health problems such as

a weakened immune system, osteoporosis, kidney

damage and others.

Today, multiple myeloma is considered an incurable

disease with inevitable relapses. As a rule, relapses

develop within a year after treatment for myeloma,

each subsequent remission being shorter than the

previous one.

Research Article

SIGNIFICANCE OF TLR6 GENE IN COMPLICATIONS OF MYELOMA
DISEASE

Submission Date:

November 10, 2023,

Accepted Date:

November 15, 2023,

Published Date:

November 20, 2023

Crossref doi:

https://doi.org/10.37547/ijmscr/Volume03Issue11-06


Kakhkharova N.Kh.

Republican Specialized Scientific And Practical Medical Center Of Hematology, Uzbekistan

Kayumov A.A.

Republican Specialized Scientific And Practical Medical Center Of Hematology, Uzbekistan

Journal

Website:

https://theusajournals.
com/index.php/ijmscr

Copyright:

Original

content from this work
may be used under the
terms of the creative
commons

attributes

4.0 licence.


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The survival prognosis depends on the stage at which

multiple myeloma is diagnosed and its type. When

detected at stages I and II, the average life expectancy

is 4-4.5 years, at stage IIIA - about 2.5 years.

The most unfavorable prognosis for multiple myeloma

detected at stage IIIB, the life expectancy of patients is

about 15 months. With primary resistance to

chemotherapy, survival is less than a year.

The TLR6 gene plays an important role in the

development

and

progression

of

myeloma

complications. Multiple studies have shown that the

TLR6 gene is associated with various aspects of the

pathogenesis and complications of myeloma.

One of the main functions of the TLR6 gene is to

regulate the div's immune system. It encodes a

receptor called toll-like receptor 6, which plays a key

role in pathogen recognition and activation of the

immune system. Due to this, the TLR6 gene influences

the development and progression of myeloma.

Some studies have shown that mutations or

polymorphisms of the TLR6 gene may be associated

with an increased risk of developing myeloma

complications. For example, one study found an

association between the presence of certain TLR6

gene variants and more aggressive myeloma.

In addition, the TLR6 gene may influence the

interaction of the tumor with components of the

microenvironment. Some studies indicate that

activation of TLR6 may promote the proliferation and

invasion of myeloma tumor cells, as well as their ability

to suppress the immune response.

More detailed research in this area will help to better

understand the role of the TLR6 gene in myeloma

complications and possible ways to develop new

therapeutic approaches.

The Ser249Pro mutation in the TLR6 gene may be

associated with various diseases, including myeloma.

184 patients with multiple myeloma were examined.

Among them, in the main group n = 94, of which

patients with grade I neuropathy n = 22, grade II

neuropathy n = 44, grade III neuropathy n = 28, in the

control group n = 90. We examined allele frequencies

and genotype frequency distributions. The results are

shown in Table 1.

Num

Group

Allele frequency

Genotype distribution frequency

S

P

S/S

S/P

P/P

n

%

n

%

n

%

n

%

n

%

1

Main group (n =

94)

26

13,8

162

86,2

3

3,19

20

21,3

71

75,5


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2

Neuropathy

stage I (n = 22)

12

27,3

32

72,7

3

13,6

6

27,3

13

59,1

3

Neuropathy

stage II (n = 44)

8

9,09

80

90,9

0

0

8

18,2

36

81,8

4

Neuropathy

grade III (n = 28)

6

10,7

50

89,3

0

0

6

21,4

22

78,6

5

Control group (n

= 90)

27

15

153

85

4

4,44

19

21,1

67

74,4

Table 1. Frequency of distribution of alleles and genotypes among patients with multiple myeloma

Also in the same study, we examined differences in the

frequency of allelic and genotypic variants of the

Ser249Pro polymorphism in the TLR6 gene in patient

groups. The distribution of genotypes across the

studied polymorphic loci was checked for compliance

with the Hardy

Weinberg equilibrium using Fisher's

exact test. Pearson's χ test with Yate’s correction for

continuity was used to compare allele frequencies

between different groups. The results of the study are

shown in Table 2.

Alleles

и

Genotypes

Number of alleles and genotypes

examined

χ2

p

RR

95%CI

OR

95%C

I

Main group

Control group

n

%

n

%

S

26

13,8

27

15,0

0,1

0,80

0,9

0,52 - 1,64

0,9

0,51

- 1,63

P

162

86,2

153

85,0

0,1

0,80

1,1

0,62 - 1,91

1,1

0,61

- 1,97

S/S

3

3,2

4

4,4

0,2

0,70

0,7

0,13 - 3,93

0,7

0,16

- 3,24

S/P

20

21,3

19

21,1

0,0

0,99

1,0

0,51 - 1,98

1,0

0,5 -

2,05

P/P

71

75,5

67

74,4

0,0

0,90

1,0

0,53 - 1,94

1,1

0,54

- 2,07


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OCLC

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Publisher:

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Alleles

и

Genotypes

Number of alleles and genotypes

examined

χ2

p

RR

95%CI

OR

95%C

I

Neuropathy stage

I

Neuropathy stage

II

n

%

n

%

S

12

27,3

8

9,1

7,5

0,01

3,0

1,21 - 7,42

3,8

1,46

- 9,63

P

32

72,7

80

90,9

7,5

0,01

0,3

0,11 - 0,98

0,3

0,1 -

0,68

S/P

6

27,3

8

18,2

0,7

0,40

1,5

0,36 - 6,26

1,7

0,51

- 5,63

P/P

13

59,1

36

81,8

4,0

0,05

0,7

0,2 - 2,56

0,3

0,1 -

0,98

Alleles

и

Genotypes

Number of alleles and genotypes

examined

χ2

p

RR

95%CI

OR

95%C

I

Neuropathy stage

I

Neuropathy

grade III

n

%

n

%

S

12

27,3

6

10,7

4,6

0,05

2,5

1,11 - 5,84

3,1

1,1 -

8,88

P

32

72,7

50

89,3

4,6

0,05

0,4

0,1 - 1,48

0,3

0,11

- 0,91

S/P

6

27,3

6

21,4

0,2

0,70

1,3

0,34 - 4,8

1,4

0,37

- 5,04

P/P

13

59,1

22

78,6

2,2

0,20

0,8

0,23 - 2,42

0,4

0,12

- 1,34

Alleles

и

Genotypes

Number of alleles and genotypes

examined

χ2

p

RR

95%CI

OR

95%C

I

Neuropathy stage

I

Control group

n

%

n

%


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S

12

27,3

27

15,0

3,7

0,10

1,8

0,6 - 5,52

2,1

0,99

- 4,58

P

32

72,7

153

85,0

3,7

0,10

0,6

0,36 - 0,85

0,5

0,22

- 1,01

S/S

3

13,6

4

4,4

2,5

0,20

3,1

0,48 - 19,64

3,4

0,76

-

15,22

S/P

6

27,3

19

21,1

0,4

0,60

1,3

0,26 - 6,52

1,4

0,48

- 4,06

P/P

13

59,1

67

74,4

2,0

0,20

0,8

0,18 - 3,41

0,5

0,19

- 1,3

Alleles

и

Genotypes

Number of alleles and genotypes

examined

χ2

p

RR

95%CI

OR

95%C

I

Neuropathy stage

II

Neuropathy

grade III

n

%

n

%

S

8

9,1

6

10,7

0,1

0,80

0,8

0,34 - 2,15

0,8

0,27

- 2,54

P

80

90,9

50

89,3

0,1

0,80

1,2

0,33 - 4,15

1,2

0,39

- 3,66

S/P

8

18,2

6

21,4

0,1

0,80

0,8

0,32 - 2,24

0,8

0,25

- 2,66

P/P

36

81,8

22

78,6

0,1

0,80

1,0

0,39 - 2,75

1,2

0,38

- 4,01

Alleles

и

Genotypes

Number of alleles and genotypes

examined

χ2

p

RR

95%CI

OR

95%C

I

Neuropathy stage

II

Control group

n

%

n

%

S

8

9,1

27

15,0

1,8

0,20

0,6

0,17 - 2,1

0,6

0,25

- 1,29


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(2023:

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P

80

90,9

153

85,0

1,8

0,20

1,7

1,11 - 2,46

1,8

0,77

- 4,03

S/P

8

18,2

19

21,1

0,2

0,70

0,9

0,25 - 3,01

0,8

0,33

- 2,08

P/P

36

81,8

67

74,4

0,9

0,40

1,1

0,31 - 3,95

1,5

0,63

- 3,79

Alleles

и

Genotypes

Number of alleles and genotypes

examined

χ2

p

RR

95%CI

OR

95%C

I

Neuropathy

grade III

Control group

n

%

n

%

S

6

10,7

27

15,0

0,7

0,50

0,7

0,16 - 3,18

0,7

0,27

- 1,73

P

50

89,3

153

85,0

0,7

0,50

1,4

0,99 - 1,99

1,5

0,58

- 3,75

S/P

6

21,4

19

21,1

0,0

0,98

1,0

0,22 - 4,75

1,0

0,36

- 2,87

P/P

22

78,6

67

74,4

0,2

0,70

1,1

0,22 - 5,06

1,3

0,45

- 3,48

Alleles

и

Genotypes

Number of alleles and genotypes

examined

χ2

p

RR

95%CI

OR

95%C

I

Neuropathy stage

I

Neuropathy stage

II

n

%

n

%

S

12

27,3

8

9,1

7,5

0,01

3,0

1,21 - 7,42

3,8

1,46

- 9,63

P

32

72,7

80

90,9

7,5

0,01

0,3

0,11 - 0,98

0,3

0,1 -

0,68

S/P

6

27,3

8

18,2

0,7

0,40

1,5

0,36 - 6,26

1,7

0,51

- 5,63

P/P

13

59,1

36

81,8

4,0

0,05

0,7

0,2 - 2,56

0,3

0,1 -

0,98


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SJIF

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MPACT

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(2021:

5.

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(2022:

5.

893

)

(2023:

6.

184

)

OCLC

1121105677















































Publisher:

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Alleles

и

Genotypes

Number of alleles and genotypes

examined

χ2

p

RR

95%CI

OR

95%C

I

Neuropathy stage

I

Neuropathy

grade III

n

%

n

%

S

12

27,3

6

10,7

4,6

0,05

2,5

1,11 - 5,84

3,1

1,1 -

8,88

P

32

72,7

50

89,3

4,6

0,05

0,4

0,1 - 1,48

0,3

0,11

- 0,91

S/P

6

27,3

6

21,4

0,2

0,70

1,3

0,34 - 4,8

1,4

0,37

- 5,04

P/P

13

59,1

22

78,6

2,2

0,20

0,8

0,23 - 2,42

0,4

0,12

- 1,34

Alleles

и

Genotypes

Number of alleles and genotypes

examined

χ2

p

RR

95%CI

OR

95%C

I

Neuropathy stage

II

Neuropathy

grade III

n

%

n

%

S

8

9,1

6

10,7

0,1

0,80

0,8

0,34 - 2,15

0,8

0,27

- 2,54

P

80

90,9

50

89,3

0,1

0,80

1,2

0,33 - 4,15

1,2

0,39

- 3,66

S/P

8

18,2

6

21,4

0,1

0,80

0,8

0,32 - 2,24

0,8

0,25

- 2,66

P/P

36

81,8

22

78,6

0,1

0,80

1,0

0,39 - 2,75

1,2

0,38

- 4,01

Table 2. Differences in the frequency of allelic and genotypic variants of the Ser249Pro polymorphism in the TLR6

gene in patient groups

Mutations in TLR genes are believed to act as a

prognostic marker for tumor progression. A SNP in a

DNA sequence is a single nucleotide substitution.

Almost all typical SNPs have only two alleles. SNPs are

found in the coding sequences of genes, noncoding

regions of genes, or intergenic regions. SNPs in the


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coding sequence are synonymous without changing

the amino acid sequence of the protein or

nonsynonymous, resulting in a different polypeptide.

SNPs in noncoding regions are reflected in changes in

gene splicing, binding of transcription factors, or in the

sequence of noncoding RNA. It has been shown that

SNPs in TLR genes lead to changes in a person's

susceptibility to infectious or inflammatory diseases

due to the inability to respond to the corresponding

ligands.

Factor

Groups

SE

SP

AUC

OR

95%CI

p

S

Main group // Control

group

0,14

0,85

0,5

0,91

0,51 - 1,62

0,51

Neuropathy stage I //

Control group

0,27

0,85

0,56

2,13

0,99 - 4,6

0,17

Neuropathy stage II //

Control group

0,09

0,85

0,47

0,57

0,25 - 1,29

0,34

Neuropathy grade III //

Control group

0,11

0,85

0,48

0,68

0,27 - 1,73

0,25

Neuropathy stage I //

Neuropathy stage II

0,27

0,91

0,59

3,75

1,46 - 9,63

0,29

Neuropathy stage I //

Neuropathy grade III

0,27

0,89

0,58

3,13

1,1 - 8,9

0,39

Neuropathy stage II //

Neuropathy grade III

0,09

0,89

0,49

0,83

0,27 - 2,59

0,62

Factor

Groups

SE

SP

AUC

OR

95%CI

p

P

Main group // Control

group

0,15

0,86

0,51

1,1

0,61 - 1,97

0,49

Neuropathy stage I //

Control group

0,15

0,73

0,44

0,47

0,22 - 1,01

0,83

Neuropathy stage II //

Control group

0,15

0,91

0,53

1,76

0,77 - 4

0,66


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Oscar Publishing Services

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Neuropathy grade III //

Control group

0,15

0,89

0,52

1,47

0,58 - 3,74

0,75

Neuropathy stage I //

Neuropathy stage II

0,09

0,73

0,41

0,27

0,11 - 0,69

0,71

Neuropathy stage I //

Neuropathy grade III

0,11

0,73

0,42

0,32

0,11 - 0,91

0,61

Neuropathy stage II //

Neuropathy grade III

0,11

0,91

0,51

1,2

0,39 - 3,66

0,38

Factor

Groups

SE

SP

AUC

OR

95%CI

p

S/S

Main group // Control

group

0,03

0,96

0,5

0,71

0,16 - 3,22

0,51

Neuropathy stage I //

Control group

0,14

0,96

0,55

3,39

0,76 - 15,17

0,18

Factor

Groups

SE

SP

AUC

OR

95%CI

p

S/P

Main group // Control

group

0,21

0,79

0,5

1,01

0,5 - 2,06

0,51

Neuropathy stage I //

Control group

0,27

0,79

0,53

1,4

0,49 - 4,04

0,18

Neuropathy stage II //

Control group

0,18

0,79

0,49

0,83

0,33 - 2,08

0,34

Neuropathy grade III //

Control group

0,21

0,79

0,5

1,02

0,35 - 3,01

0,24

Neuropathy stage I //

Neuropathy stage II

0,27

0,82

0,55

1,69

0,51 - 5,65

0,31

Neuropathy stage I //

Neuropathy grade III

0,27

0,79

0,53

1,38

0,37 - 5,14

0,42


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SJIF

I

MPACT

FACTOR

(2021:

5.

694

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(2022:

5.

893

)

(2023:

6.

184

)

OCLC

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Publisher:

Oscar Publishing Services

Servi

Neuropathy stage II //

Neuropathy grade III

0,18

0,79

0,49

0,81

0,24 - 2,74

0,62

Factor

Groups

SE

SP

AUC

OR

95%CI

p

P/P

Main group // Control

group

0,76

0,26

0,51

1,06

0,54 - 2,07

0,5

Neuropathy stage I //

Control group

0,59

0,26

0,43

0,5

0,19 - 1,29

0,28

Neuropathy stage II //

Control group

0,82

0,26

0,54

1,54

0,63 - 3,75

0,26

Neuropathy grade III //

Control group

0,79

0,26

0,53

1,26

0,45 - 3,5

0,21

Neuropathy stage I //

Neuropathy stage II

0,59

0,18

0,39

0,32

0,1 - 0,98

0,53

Neuropathy stage I //

Neuropathy grade III

0,1

0,21

0,16

0,03

0 - 0,18

0,6

Neuropathy stage II //

Neuropathy grade III

0,82

0,21

0,52

1,23

0,37 - 4,07

0,57

Table 3. Prognostic effectiveness of the studied genetic markers (Ser249Pro polymorphism in the TLR6 gene)

We carried out a statistical analysis of the expected and

observed frequencies of the distribution of genotypes

of the locus for RHV (Ser249Pro polymorphism in the

TLR6 gene) and the data are shown in Table 4.

Main group

Alleles

Allele frequency

S

0,14

P

0,86

Genotypes

Genotype frequency

χ2

p

df

observable

expected

S/S

0,03

0,02

0,8


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Volume 03 Issue 11-2023

48


International Journal of Medical Sciences And Clinical Research
(ISSN

2771-2265)

VOLUME

03

ISSUE

11

P

AGES

:

38-52

SJIF

I

MPACT

FACTOR

(2021:

5.

694

)

(2022:

5.

893

)

(2023:

6.

184

)

OCLC

1121105677















































Publisher:

Oscar Publishing Services

Servi

S/P

0,21

0,24

0,26

P/P

0,76

0,74

0,02

Total

1

1

1,08

0,284

1

Control group

Alleles

Allele frequency

S

0,15

P

0,85

Genotypes

Genotype frequency

χ2

p

df

observable

expected

S/S

0,04

0,02

1,93

S/P

0,21

0,26

0,68

P/P

0,74

0,72

0,06

Total

1

1

2,67

0,099

1

Groups

Ho

He

D*

Main group

0,21

0,24

-0,11

Control group

0,21

0,26

-0,17

Note: D = (Ho - He)/He

Expected and observed frequencies of distribution of genotypes of the locus for RHV

(Ser249Pro polymorphism in the TLR6 gene)

Neuropathy stage I

Alleles

Allele frequency

S

0,27

P

0,73

Genotypes

Genotype frequency

χ2

p

df

observable

expected


background image

Volume 03 Issue 11-2023

49


International Journal of Medical Sciences And Clinical Research
(ISSN

2771-2265)

VOLUME

03

ISSUE

11

P

AGES

:

38-52

SJIF

I

MPACT

FACTOR

(2021:

5.

694

)

(2022:

5.

893

)

(2023:

6.

184

)

OCLC

1121105677















































Publisher:

Oscar Publishing Services

Servi

S/S

0,14

0,07

1,14

S/P

0,27

0,4

0,85

P/P

0,59

0,53

0,16

Total

1

1

2,15

0,145

1

Control group

Alleles

Allele frequency

S

0,15

P

0,85

Genotypes

Genotype frequency

χ2

p

df

observable

expected

S/S

0,04

0,02

1,93

S/P

0,21

0,26

0,68

P/P

0,74

0,72

0,06

Total

1

1

2,67

0,099

1

Groups

Ho

He

D*

Neuropathy stage I

0,27

0,4

-0,31

Control group

0,21

0,26

-0,17

Note: D = (Ho - He)/He

Expected and observed frequencies of distribution of genotypes of the locus for RHV

(Ser249Pro polymorphism in the TLR6 gene)

Neuropathy stage II

Alleles

Allele frequency

S

0,09

P

0,91

Genotypes

Genotype frequency

χ2

p


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Volume 03 Issue 11-2023

50


International Journal of Medical Sciences And Clinical Research
(ISSN

2771-2265)

VOLUME

03

ISSUE

11

P

AGES

:

38-52

SJIF

I

MPACT

FACTOR

(2021:

5.

694

)

(2022:

5.

893

)

(2023:

6.

184

)

OCLC

1121105677















































Publisher:

Oscar Publishing Services

Servi

observable

expected

df

S/S

0

0,01

0,36

S/P

0,18

0,17

0,07

P/P

0,82

0,83

0

Total

1

1

0,44

0,483

1

Control group

Alleles

Allele frequency

S

0,15

P

0,85

Genotypes

Genotype frequency

χ2

p

df

observable

expected

S/S

0,04

0,02

1,93

S/P

0,21

0,26

0,68

P/P

0,74

0,72

0,06

Total

1

1

2,67

0,099

1

Groups

Ho

He

D*

Neuropathy stage II

0,18

0,17

0,1

Control group

0,21

0,26

-0,17

Note:D = (Ho - He)/He

Expected and observed frequencies of distribution of genotypes of the locus for RHV

(Ser249Pro polymorphism in the TLR6 gene)

Neuropathy grade III

Alleles

Allele frequency

S

0,11


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Volume 03 Issue 11-2023

51


International Journal of Medical Sciences And Clinical Research
(ISSN

2771-2265)

VOLUME

03

ISSUE

11

P

AGES

:

38-52

SJIF

I

MPACT

FACTOR

(2021:

5.

694

)

(2022:

5.

893

)

(2023:

6.

184

)

OCLC

1121105677















































Publisher:

Oscar Publishing Services

Servi

P

0,89

Genotypes

Genotype frequency

χ2

p

df

observable

expected

S/S

0

0,01

0,32

S/P

0,21

0,19

0,08

P/P

0,79

0,8

0

Total

1

1

0,4

0,502

1

Control group

Alleles

Allele frequency

S

0,15

P

0,85

Genotypes

Genotype frequency

χ2

p

df

observable

expected

S/S

0,04

0,02

1,93

S/P

0,21

0,26

0,68

P/P

0,74

0,72

0,06

Total

1

1

2,67

0,099

1

Groups

Ho

He

D*

Neuropathy grade III

0,21

0,19

0,12

Control group

0,21

0,26

-0,17

Note: D = (Ho - He)/He

Таблица 4. Expected and observed frequen

cies of distribution of genotypes of the locus for RHV (Ser249Pro

polymorphism in the TLR6 gene)

Thus, the TLR6 gene has a significant impact on the

development

and

progression

of

myeloma

complications. Its role is associated with the regulation

of the immune response and the interaction of tumor

cells with the microenvironment. Mutations and

polymorphisms of the TLR6 gene may be associated

with an increased risk of developing more aggressive

variants of myeloma. Further research in this area will


background image

Volume 03 Issue 11-2023

52


International Journal of Medical Sciences And Clinical Research
(ISSN

2771-2265)

VOLUME

03

ISSUE

11

P

AGES

:

38-52

SJIF

I

MPACT

FACTOR

(2021:

5.

694

)

(2022:

5.

893

)

(2023:

6.

184

)

OCLC

1121105677















































Publisher:

Oscar Publishing Services

Servi

allow us to better understand the molecular

mechanisms associated with the TLR6 gene and

develop new therapeutic approaches to prevent and

treat complications of myeloma.

REFERENCES

1.

Bohnert V, Schäfer C, Müller-Tidow C, et al. Toll-like

receptors in normal and malignant hematopoiesis.

Curr

Pharm

Des.

2006;12(32):4219-4232.

doi:10.2174/138161206779010392

2.

Vacca A, Ribatti D, Presta M, et al. Bone marrow

neovascularization,

plasma

cell

angiogenic

potential,

and

matrix

metalloproteinase-2

secretion parallel progression of human multiple

myeloma. Blood. 1999;93(9):3064-3073.

3.

Kawai T, Akira S. The role of pattern-recognition

receptors in innate immunity: update on Toll-like

receptors. Nat Immunol. 2010;11(5):373-384.

doi:10.1038/ni.1863

4.

Vacca A, Ria R, Reale A, et al. TLR gene expression

profile in human multiple myeloma cells: down-

regulation of TLR3 expression. Cancer Immunol

Immunother.

2006;55(7):910-920.

doi:10.1007/s00262-005-0079-1

5.

Botta C, Gulla A, Correale P, Tagliaferri P, Tassone

P. Myeloma cells suppress osteoblasts through

sclerostin secretion. Blood Cancer J. 2011;1(6):e27.

doi:10.1038/bcj.2011.26

References

Bohnert V, Schäfer C, Müller-Tidow C, et al. Toll-like receptors in normal and malignant hematopoiesis. Curr Pharm Des. 2006;12(32):4219-4232. doi:10.2174/138161206779010392

Vacca A, Ribatti D, Presta M, et al. Bone marrow neovascularization, plasma cell angiogenic potential, and matrix metalloproteinase-2 secretion parallel progression of human multiple myeloma. Blood. 1999;93(9):3064-3073.

Kawai T, Akira S. The role of pattern-recognition receptors in innate immunity: update on Toll-like receptors. Nat Immunol. 2010;11(5):373-384. doi:10.1038/ni.1863

Vacca A, Ria R, Reale A, et al. TLR gene expression profile in human multiple myeloma cells: down-regulation of TLR3 expression. Cancer Immunol Immunother. 2006;55(7):910-920. doi:10.1007/s00262-005-0079-1

Botta C, Gulla A, Correale P, Tagliaferri P, Tassone P. Myeloma cells suppress osteoblasts through sclerostin secretion. Blood Cancer J. 2011;1(6):e27. doi:10.1038/bcj.2011.26