Authors

  • Zokhirov Adkhamjon Rafiqovich
    Assistant Of General Surgery No.2, Tashkent Medical Academy, Tashkent City, Uzbekistan
  • Abdukarimov Sarvar Azizbek Ogli
    3rd Year Student Of Faculty Of Medicine, Tashkent Medical Academy, Tashkent City, Uzbekistan
  • Khakimova Dilyora Ulugbekovna
    4th Year Student Of Faculty Of General Medicine Kimyo International University In Tashkent, Tashkent City, Uzbekistan
  • Solijonova Umidaxon Ilhomjonovna
    3rd Year Student Of Faculty Of General Medicine Kimyo International University In Tashkent, Tashkent City, Uzbekistan
  • Sherkulov Bobur Bahodir Ogli
    3rd Year Student Of Faculty Of General Medicine Kimyo International University In Tashkent, Tashkent City, Uzbekistan
  • Fayzullayeva Dilnoza Baxtiyor Qizi
    2nd Year Student Of Faculty Of General Medicine Kimyo International University In Tashkent, Tashkent City, Uzbekistan

DOI:

https://doi.org/10.37547/ijmscr/Volume04Issue09-07

Keywords:

diabetic foot syndrome cytokine system complex treatment

Abstract

In patients with purulent-necrotic processes in diabetes mellitus, the condition of the IL-1B, IL-2, IL-4, IL-6, IL-8, IL-10, TNF-α, INF system based on the dynamics of cytokine levels was studied. Complex surgical treatment of the drug Reomannisol corrects the balance of cytokines, allowing them to have an antioxidant effect on the cytokine system.


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ABSTRACT

In patients with purulent-necrotic processes in diabetes mellitus, the condition of the IL-1B, IL-2, IL-4, IL-6, IL-8, IL-10,

TNF-

α, INF system based on the dynamics of cytokine levels was studied. Complex surgical treatmen

t of the drug

Reomannisol corrects the balance of cytokines, allowing them to have an antioxidant effect on the cytokine system.

Research Article

OPTIMIZATION OF COMPLEX TREATMENT OF PURULENT-NECROTIC
WOUNDS ON THE FOOT IN DIABETES MELLITUS

Submission Date:

Sep 19, 2024,

Accepted Date:

Sep 24, 2024,

Published Date:

Sep 29, 2024

Crossref doi:

https://doi.org/10.37547/ijmscr/Volume04Issue09-07


Zokhirov Adkhamjon Rafiqovich

Assistant Of General Surgery No.2, Tashkent Medical Academy, Tashkent City, Uzbekistan

Abdukarimov Sarvar Azizbek Ogli

3rd Year Student Of Faculty Of Medicine, Tashkent Medical Academy, Tashkent City, Uzbekistan

Khakimova Dilyora Ulugbekovna

4th Year Student Of Faculty Of General Medicine Kimyo International University In Tashkent, Tashkent City,
Uzbekistan

Solijonova Umidaxon Ilhomjonovna

3rd Year Student Of Faculty Of General Medicine Kimyo International University In Tashkent, Tashkent City,
Uzbekistan

Sherkulov Bobur Bahodir Ogli

3rd Year Student Of Faculty Of General Medicine Kimyo International University In Tashkent, Tashkent City,
Uzbekistan

Fayzullayeva Dilnoza Baxtiyor Qizi

2nd Year Student Of Faculty Of General Medicine Kimyo International University In Tashkent, Tashkent City,
Uzbekistan


Journal

Website:

https://theusajournals.
com/index.php/ijmscr

Copyright:

Original

content from this work
may be used under the
terms of the creative
commons

attributes

4.0 licence.


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KEYWORDS

diabetic foot syndrome, cytokine system, complex treatment, Rheomannisol.

INTRODUCTION

Diabetes mellitus (DM) is a chronic autoimmune

disease. When purulent-necrotic lesions of diabetic

heel syndrome appear, an inflammatory reaction

appears and manifests itself in the form of a local

reaction, which can lead to a systemic response. As a

result of bacterial action, under the influence of

humoral factors, activation occurs in the main

phagocytosis cells

neutrophils (polymorphonuclear

leukocytes), monocytes (macrophages) and platelets.

Activated cells begin to produce inflammatory

mediators (cytokines) into the blood, which are

involved in the regulation of vascular tone, hemostasis,

and cell proliferation. Atherosclerosis of blood vessels

in patients with diabetes occurs 8-10 years earlier than

in the general population. Therefore, an inflammatory

reaction occurs against the background of changes in

metabolism

and

circulatory

disorders.

The

pathogenetic mechanism of diabetes is complex, but

the latest research data suggests that the participation

of cytokines plays a large role in this disease. Cytokines

are conditionally anti-inflammatory: natural immune

regulators involved in inflammatory formation (IL-1, IL-

2, IL-6, IL-12, TNF-

α) and anti

-inflammatory by

pancreatic β

-cells that depressant to insulin

production:

special

immune

regulators

of

inflammatory response (IL-4, IL-10, IL-13, INF-

γ) these

cytokines have protective and antidiabetic effects. In

addition, there are cytokines that regulate specific

immune reactions (IL-2 and IL-4, transformative

growth factor (TGF-

β va et al.). It is involved in protein

activation, differentiation and growth of mature

lymphocytes. A number of cytokines (IL-1, IL-6, TNF-

α,

erythropoietin) can also have effects such as

distension, which is simple hormones. In addition, a

similar single cell can secrete several different

cytokines and the same cytokines at the same time, or

different cells can produce several different or

identical cytokines. Therefore, the importance of

cytokines in the formation of diabetes mellitus, on the

one hand, and in the formation of an inflammatory

reaction, on the other, is interesting to obtain

information about the state of the cytokine in patients

with DFS. The state of the cytokine system at the time

of the development of purulent-necrotic processes in

patients with DFS is completely unclear. Given the lack

of research, DFS is solved by the dynamic state of the

cytokine system in patients with purulent-necrotic

processes and recurrence of purulent-necrotic

processes. It was decided to study the dynamics of

cytokine levels in the blood serum of anti -


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inflammatory cytokines-interleukins IL-

1β, IL

-2, IL-6, IL-

8, tumor necrosis factor-TNF-

α, as well as gamma

-

interferon (IFN-

γ) and anti

-inflammatory interleukins

IL-4, IL-10 patients. In our opinion, perhaps the

imbalance of cytokines will help predict the course of

the purulent-necrotic process and determine the

tactics of surgical treatment. In addition, the state of

the cytokine balance is of interest in patients with

recurrence of purulent-necrotic complications of DFS.

Cytokine management balance is currently seen as a

new direction of immunotherapeutic effects in the

treatment of patients with purulent-necrotic lesions of

DFS.

The purpose of the work. By studying the dynamics of

anti-inflammatory cytokine levels: analysis of the need

and possibility of immunocorrection by studying

cytokine status in IL-

1β, IL

-2, IL-4, IL-6, IL-8, IL-10, TNF-

α,

INF-

γ patients on the development and recurrence of

purulent-necrotic processes of DFS.

Table 1. In normal patients, the amount of cytokines and the number of

cells in the peripheral blood serum

Cytokines

Concentration of

cytokines in

blood plasma,

pg/ml

Production of cells in blood culture,

pg/ml

Spontaneous

Induced

IL-1β

0-50

0-50

1000-5000

IL-2

0

0

2-10 ED/ml

IL-4

0-50

30-50

100-400

IL-6

0-50

30-50

1000-3000

IL-8

0-50

30-100

1000-5000

IL-10

0-50

30-50

1000-3000

TNF-α

0-50

30-50

500-3000

INF-γ

0-10

10-100

1000-5000


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Table 2. Characteristics of operations in group I

Type of operation

Frequency of operations (n=60)

abs.

%

Amputations at the hip

level

20

33,3

Amputations at the

lower leg level

6

10

Amputations at the foot

level

12

20

Amputations at the

level of the fingers

11

18,3

Amputations at the

level of the fingers

7

11,7

Necrectomy

4

6,7

MATERIALS AND METHODS

The concentration of cytokines in the blood plasma

was determined using the StatFax 2100 immunological

complex (manufactured by Cytokin LLC, St.

Petersburg) by enzyme immunoassay (IFA). The

obtained results are carried out using descriptive and

non-parametric statistical methods on a personal

computer with mathematical processing using

Statistica 6.0 software (Table 1). Control parameters of

the manufacturer's cytokine norms were used - the

normative parameters obtained as a result of testing

the donors were obtained from the manufacturer

Cytokin LLC, St. Petersburg, Russian Federation.

Plasma collection and cytokine level testing were

performed on day 1 before surgery and on days 3, 5, 7,

and 10 after surgery. The study was conducted in three

phases and, accordingly, the patients were divided into

3 research groups: Group I (control group) - blood

serum of patients with purulent-necrotic lesions of

diabetes for the study of the cytokine system; II group

(comparison group) - blood serum obtained during the


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treatment of patients with purulent-necrotic lesions of

diabetes with ozone therapy and peosorbilact - 200.0

ml (YURIYA-FARM LLC, Ukraine); Group III (main

group) - blood sera obtained during the treatment of

patients with purulent-necrotic lesions of diabetes

with ozone therapy and reomannisol - 200.0 ml (REKA-

MED FARM LLC, Uzbekistan).

The first stage: to determine the imbalance of

cytokines, it consists in taking the blood plasma of 60

patients with purulent-necrotic lesions of diabetes and

determining the amount of cytokines in it. 60 patients

were treated at various levels in the Yiringli surgical

department of the Yakkasaroy District Medical

Association between 2021 and 2023. The age range of

the patients was between 48-60 years. Neuropathic

form of DFS - 25 people (type 1 DM - 2 patients, type 2

DM - 23): 12 were men and 13 women, and

neuroischemic form of DFS - 35 patients (all with type 2

DM): 15 men and consisted of 20 women. The

characteristics of operations performed in this group

of patients are presented in Table 2. Most often,

amputations were performed at the thigh level - 33.3%,

heel area - 20%, finger level - 18.3%, necrotomy - 6.7%,

sequestrectomy - 11.7%. Upper amputations were also

performed as the main option, as a result of the failure

of the approach to save vital organs: at the thigh level

- in 33.3% of cases, at the heel level - in 20%.

Results and discussions in the first group. The dynamics

of the level of cytokines in the first group is presented

in Table 3. In the analysis of the indicators of the level

of cytokines, the picture of growth was revealed:

inflammatory cytokines: IL-

1β, IL

-2, IL-6, IL-8, TNF-

α and

anti-inflammatory cytokines: IL-4, IL- 10, IFN-

γ.

IL-

1β: ranged from 7.25±0.24 to 2.9±0.1 pg/ml, but all

changes were within the acceptable range. IL-1b

appears in the blood plasma when a pathogenic agent

enters the div for the first time or in autoimmune

type 1 DM. Perhaps, with the first purulent-septic injury

of DFS, the biological activity of this cytokine does not

increase, that is, due to the insufficient activity of

macrophages, monocytes, lymphocytes due to the

endothelial reaction, as well as the functional activity

of fibroblasts decreases.

IL-2: ranges from 6.97±0.13 to 1.09±0.48 pg/ml, which is

3-2 times higher than normal values, especially in

lymphocytes responsible for the immune system is

explained by activation. In the postoperative period,

the cytokine level decreases, but it is higher than the

norm during discharge from the hospital, which allows

us to consider this decrease as a residual reaction of

the immune system, which can be associated with

positive clinical dynamics.

IL-6: ranged from 90.1±0.76 to 12.59±1.9 pg/ml. A high

level of cytokines on the first day, a decrease to normal

by the 7th day, an acute phase with a decrease after

the clinical relief of the inflammatory process, as well


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as an indirect reaction of the endothelial system in the

purulent-necrotic focus indicate high protein activity.

IL-8: ranged from 129±0.13 to 579±0.11 pg/ml by day 7

and decreased to 51.8±0.11 pg/ml at hospital discharge,

suggesting that indicates the presence of an acute

purulent-necrotic process and the possibility of

chronicity, since cytokine levels remain at higher levels

during clinical recovery.

Table 3. Level of cytokines (in pg/ml) in patients of the first group

Cytokin

es/

Days

Day 1

Day 3

Day 5

Day 7

Day 10

Discharge (11-

12 days)

IL-

7,25±0,24

*

5,67±0,1

3*

4,56±0,1

*

4,14±0,

14*

2,9 ±0,1*

6,2±0,7*

IL-2

6,22±0,3

*

6,97±0,1

3*

6,1±0,2

*

6,5±0,1

5*

5,1±0,11*

1,09±0,48*

IL-4

235±0,13

*

255±0,5

*

280±0,1

2*

199±0,1

6*

101±0,1*

45±0,2*

IL-6

90,1±0,7

6*

80,5±0,

6*

60±0,12

*

16,02±0,

23*

13±0,5*

12,59±1,9*

IL-8

129±0,13

*

199,3±0,

11*

245,5±0,

1*

579±0,

11*

65,7±0,1

5*

50,8±0,11*

IL-10

59,08±0,7

2*

58,12±0,

3*

50±0,4

*

51,68±0,

73*

36±0,15

*

6,65±1,33*

TFN-

α

51,51±0,7

*

55±0,1*

56±0,14

*

51,01±0,

94*

40±0,16

*

2,2±0,52*

INF-

γ

68,3±0,1

*

105,1±0,

14*

101±0,11

*

45,8±0,

13*

40±0,11

*

10,4±0,14*

Note:*

р

<0,05.


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TNF-

α: ranged to 56±0.14 pg/ml by day 5 and decreased

to 2.2±0.52 pg/ml at hospital discharge. It shows the

immunomodulatory and anti-inflammatory effects of

the cytokine caused by the activation of macrophages,

neutrophils, eosinophils and endothelial cells, as well

as the metabolic disorders associated with

hyperglycemia.

IL-4: increased to 280±0.12 pg/ml by day 5 and

fluctuated between 235±0.13 pg/ml, and decreased to

45±0.2 pg/ml at hospital discharge. Such dynamics can

be explained by the growth and activity of

macrophages, lymphocytes, and, accordingly, a high

level of anti-inflammatory cytokines. IL-4 performs

regulatory functions.

IL-10: ranged from 59.08±0.72 to 6.65±1.33 pg/ml. It is

an antagonist of anti-inflammatory cytokines and

performs a regulatory function, reduces its level,

shows a favorable direction of this process with clinical

improvement.

IFN-

γ: fluctuated from 105.1±0.14 pg/ml to 10.4±0.14

pg/ml with a decrease on day 3. This dynamic can be

explained by clinical recovery. IFN-

γ has a wide

spectrum of immunomodulatory activity, regulates

and protects it, establishes a balance of activity with

fibroblasts and macrophages and monocytes. In

addition, on the first day, high levels of inflammatory

and anti-inflammatory cytokines are observed, their

number increases slightly, and on the fifth day, it

decreases with a clinically favorable level. A high level

of IL-8 indicates activation in response to acute

inflammation and the possibility of the process

becoming chronic. This stage of the research made it

possible to study the state of the cytokine system in

purulent-necrotic lesions of DFS.

The second stage: studying the dynamics of cytokines

in recurrent purulent-necrotic lesions of diabetes.

Materials and methods of the second stage -

comparison group. The second group of the study

included 40 patients (men and women aged 45 to 74).

In 2021-2023, patients with DFS with recurrent

purulent-necrotic lesions of diabetes were taken in the

Department of Purulent Surgery of the Yakkasaroy

District Medical Association. With the neuropathic

form of DFS - 15 people: 5 men and 10 women and the

neuroischemic form of DFS - 25 patients: 9 men and 16

women. The characteristics of operations performed in

this group of patients are presented in Table 4. All

patients, regardless of the severity of the disease, were

treated locally with ozonized physiological solution,

and 200.0 ml of Reosorbilact was administered

intravenously once, the course of treatment was 7

days.

Determination of ozone concentration. To prepare an

ozonated physiological solution, we ozonate 200.0 ml

of physiological solution for 10 minutes at a

concentration of 3-4 mg/l at a concentration of 3-4 mg/l


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and for 10 minutes in the apparatus "Medozons

Beauty" (MEDOZONS LLC, Russia), then its dose is

determined by the following formula:

D

ozone concentration dose;

C

ozone concentration, mg/l;

V - oxygen exit rate, l/min;

t

time, min;

The purulent area of each patient is washed 3 times a day with ozonized physiological solution for 15-20 minutes. The

number of operations is greater than the number of patients. This is explained by the fact that various operations

were performed in one patient to preserve the quality of life and ability to work: necrotomy - 20%, amputation at the

level of fingers - 22.9%, amputations at the level of the foot - 2.9%.

Table 4. Characteristics of operations in group II

Type of operation

Frequency of operations (n=40)

abs.

%

Amputations at the hip level

23

65,7

Amputations at the lower leg

level

0

0

Amputations at the foot level

2

5,0

Amputations at the level of

the fingers

8

22,9

Amputations at the level of

the fingers

5

14,3

Necrectomy

7

20

D=C x V x t;


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However, high amputations were performed when the

tactics of saving vital organs were ineffective or when

there was no prospect of saving the leg at the time of

the initial examination - 65.7%.

Results and discussions in the second group. The

dynamics of the state of the cytokine system in the

blood plasma of patients in this group of patients is

presented in Table 5. It should be noted that 1 day

before the operation, only an increase in the level of IL-

8 up to 182.7 pg/ml is detected. It increased to 245.5

pg/ml by day 5 and decreased to 60.9 pg/ml on days 11-

12. The activity and level of IL-8 is considered to be

related to the activity of IL-

1β, IL

-4, and TNF-

α, as well

as the effects of endotoxins of gram-negative bacterial

microorganisms. In addition, IL-8-producing cells are

monocytes, macrophages, T-lymphocytes, neutrophils,

fibroblasts, keratinocytes, hepatocytes, endothelial

cells, epithelial cells, chondrocytes. IL-8 as a mediator

stimulates

chemotaxis

of

subpopulations

of

neutrophils, T-lymphocytes and basophils; lysosomal

enzymes activate neutrophils.

Table 5. Level of cytokines (in pg/ml) in patients of the second group

Cytokin

es/

Days

Day 1

Day 3

Day 5

Day 7

Day 10

Discharge (11-

12 days)

IL-

10,2±0,

2*

13±1,19

*

7±1,12

*

3,4±0,14

*

5±1,1*

2,2±0,4*

IL-2

0

0

0

0

0

0

IL-4

32±1,14

*

30±1,12

*

31±1,11

*

25±1,13

*

17±1,4*

9±1,1*

IL-6

18,1±0,7

*

15±1,2*

23±1,1

*

15,02±1,3

*

14,9±1,9

*

10,59±1,9*

IL-8

182,7±1,

1*

201,5±1,1

*

245,5±1

,3*

210,1±1,2

*

103,7±1,

4*

70,9±1,9*

IL-10

9,08±0,

7*

23,1±0,9

*

19,3±0,

8*

10,68±1,

3*

14,5±1,1

*

15,65±1,2*

TFN-

α

21,5±

1,14*

19,6±1,11

*

15,6±0,

9*

14,1±0,1

*

12,5±1,1

*

10,2±0,5*


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INF-

γ

0,9±1,1

*

2,6±1,1*

1,1±1,3

*

8,1±1,1*

0,8±1,1*

0,3±0,9*

Note: *

р

<0,05.

In addition, it has receptors that react with monocytes

and neutrophils, which have the ability to cause a

specific appearance, and stops these cells in the

capillaries located in the area of inflammation. Such a

high level of IL-8 indicates that the inflammatory

process in the leg becomes chronic in a positive way.

The dynamics of cytokine levels are more clearly shown

in Figure 2. At the same time, changes in the level of IL-

1β, IL

-2, IL-4, IL-6, IL-10, TNF-

α, INF

-

γ cytokines were

noted, but within normal limits. Locally, in 2 cases,

variants of purulent-necrotic injuries, which included

not only two anatomical areas of the leg, but also with

the spread of the process to the border of the upper

and middle third of the leg, and in 3 cases - ended with

gangrene of the distal parts of the leg. Cytokine

regulation of the inflammatory reaction is insufficiently

activated in patients with chronic DFS and secondary

immunodeficiency with recurrence of the purulent-

necrotic process. This leads to a lack of growth factors

and slow healing. Perhaps this condition can be

considered as a weak proliferative response.

Accordingly, as a local process, the functional status of

functional

mononuclear

phagocytes,

platelets,

macrophages, fibroblasts, keratocytes, endothelial

cells and other cells slows down with slow resorption

of the infiltrate and the formation of necrosis.

Materials and methods of the third stage

main group.

Taking into account the information obtained about

the state of the cytokine system in patients with

purulent-necrotic processes of DFS, it was decided to

study the antioxidant effect of ozone therapy and the

drug Reomannisol. The research group consists of 40

patients. With the neuropathic form of DFS - 16 people:

4 men and 12 women, and the neuroischemic form of

DFS - 24 patients: 8 men and 16 women, aged 50 to 75

years. In the anamnesis, there were multiple

recurrences of purulent-necrotic complications (from 2

to 4 times). The duration of diabetes is from 5 to 20

years. The goal of surgery and conservative treatment

algorithm is to preserve the function of the leg or

reduce the level of amputation. Reomannisol was used

from the first day of hospitalization to study the

antioxidant and peripheral blood circulation effects of

the drug in complex treatment. According to the

scheme, 200.0 ml of solution was administered

intravenously 1 time per day for 10 days. Plasma

sampling and cytokine levels were performed on the

first day before and/or after surgery, and then on days


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3, 5, 7, 10, and 12 again blood plasma was taken and the

level of cytokines was determined.

Results and discussions in the third group. Table 6 lists

the procedures performed in patients of group III.

Table 6. Characteristics of operations in group III

Type of operation

Frequency of operations (n=35)

abs.

%

Amputations at the hip level

10

28,6

Amputations at the lower leg

level

1

2,9

Amputations at the foot level

1

2,9

Amputations at the level of

the fingers

15

42,8

Amputations at the level of

the fingers

11

31,3

Necrectomy

15

42,9

Operations performed to save the leg: necroctomies - 42.9%, sequestrectomy - up to 31.3%, amputations at the level of

toes - 42.8%, amputations at the level of the heel - 34.3%. In addition, the number of high amputations: at the hip level

- 28.6%, at the heel level - 2.9%. Compared to the second group, the number of upper amputations decreased from

65.7% to 28.6%. In addition, the antioxidant effect of Reomannisol, manifested in the induction of inflammatory and

anti-inflammatory cytokines, was determined. Cytokine levels in patients of study group III are presented in Table 7.

Table 7. The amount of cytokines after ozonotherapy + reomannisol treatment in group III patients, pg/ml

Cytokines/

Days

Day 1

Day 3

Day 5

Day 7

Day 10

Discharge (11-

12 days)

IL-

64,59±3,

3*

74,48

±1,9*

95,27±2,2

*

71,48±

1,8*

31,49

±1,9*

20±1,8*

IL-2

1,2±0,3*

1,7±0,

13*

1,1±0,2*

0,54±

0,15*

0,2±0,

11*

0

IL-4

41,56±2,3

43,96

39,29±4,1

30,7±3

32,9±

20,58±2,2*


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*

±3,1*

*1

,2*

3,5*

IL-6

228,92±5

,1*

138±3

,2*

50,8±2,1*

42,6±3

,1*

38±2,

1*

8,5±1,1*

IL-8

181,14±4,

1*

197,31

±3,1*

245,47±5,9

*

222,6±

4,5*

109,6

±5,1*

53±1,1*

IL-10

48,8±2,8

*

25,81

±1,6*

34,2±2,1*

40,2±3

,2*

38,7±

2,5*

21,2±1,3*

TFN-

α

147,86±4

,6*

150,6

±5,3*

121,96±5,4

*

91,58±

4,3*

41,1±2

,1*

21,87±1,1*

INF-

γ

194±3,4

*

205,9

±6,5*

89±4,1*

47,8±2

,1*

7,8±0

,4*

5,39±0,3*

Note: *

р<0,05.

IL-

1β: high level of cytokine up to 64.59±3.3 pg/ml on

the first postoperative day, increased to 95.27±2.2

pg/ml on the fifth day and decreased to 20 ± 1.8 pg/ml.

IL-1b promotes the proliferation of monocytes,

macrophages, neutrophils, endothelium, smooth

muscle cells, fibroblasts, keratinocytes, T- and B-

lymphocytes, along with other cytokines; increases the

stimulation of receptors for IL-2; activates endothelial

cells; induces an acute phasic response.

IL-2: increases to 1.2±0.3 pg/ml on the first

postoperative day, to 1.7±0.13 pg/ml on the third day,

and to 0 at discharge. After an increase in IL-2, the

active implementation of the immune response and

the activation of T cells show.

IL-4: fluctuations were noted within normal limits and

an increase was observed in only one case, that is, 76

pg/ml in a patient with an allergic background, which

increased due to the development of postoperative

traumatic dermatitis on the limbs after surgery. . It is

possible that reomannisol stimulates the production of

this cytokine by mononuclear cells in the peripheral

blood of allergic patients. However, as a result of

antioxidant activity, there are fluctuations in the group

of cytokines with pro-inflammatory effects, but only

within normal limits.

IL-6: a high level of cytokine up to 228.92±5.1 pg/ml was

recorded on the first postoperative day. Then it

gradually returns to normal, and by the time of

discharge from the hospital, it was back to normal. IL-


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6 is produced by T-lymphocytes, monocytes,

macrophages, fibroblasts. Hematopoiesis causes the

differentiation of progenitor cells, stimulates the

development and production of megakaryocytes,

promotes the growth and differentiation of platelets,

T- and B-lymphocytes, and stimulates the production.

IL-8: peak cytokine levels increased to 181.14 ± 4.1 pg/ml

on the first postoperative day, 245.47 ± 5.9 pg/ml on

the fifth day, and 53 ± 1.1 pg/ml at hospital discharge.

decreased to ml. As revealed in the study of the

previous two groups, the growth of this cytokine

characterizes the transition of the process to a chronic

form.

IL-10: An anti-inflammatory cytokine produced by T-

lymphocytes, macrophages, keratinocytes, and B-

lymphocytes. It blocks the functional activity of

macrophages, the production of anti-inflammatory

cytokines by monocytes and macrophages. It increases

the proliferation of B-lymphocytes and the secretion of

immunoglobulins IgE. However, IL-10 can stimulate the

synthesis of IL-4 in a balanced state. By preventing the

systemic response to the inflammatory process, IL-10

ensures a balanced level of inflammatory and anti-

inflammatory cytokines, controls the immune

response, which has a positive effect on the prevention

of purulent-necrotic recurrence.

TNF-

α: produced by macrophages, monocytes,

keratinocytes,

T-lymphocytes,

B-lymphocytes,

neutrophils, endothelial cells. TNF-

α has a variety of

effects, occurs due to the modulation of the

expression of genes for cell growth factors, cytokines,

transcription factors, cell surface receptors and acute

phase proteins, and plays an important role in

protection against them. The increase of this cytokine

on the third day up to 150.6±5.3 pg/ml after the

operation is included in the high activity and the

cytokine itself.

INF-

γ: on the first day, a high level was recorded to

194±3.4 pg/ml, on the third day it increased to

205.9±6.5 pg/ml and decreased to 5.39±0.3 pg/ml, this

indicates

its

immunomodulatory

value

and

involvement in the regulation of the anti-inflammatory

response.

CONCLUSION

In the case of purulent-necrotic processes in DFS, the

adequate introduction of the cytokine system is

characterized by the slowing down of the local

process, corresponding to acute inflammation, but

increases the tendency of the process to go into a

chronic state. With the recurrence of purulent-necrotic

inflammation, there is a chronization of the process

and a secondary immune deficiency, which does not

allow the cytokine system to adequately join the anti-

inflammatory system. Reomannisol allows us to

consider it a physiologically active compound with

clear antioxidant activity. Reomannisol has an


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antioxidant effect on the cytokine system, correcting

the cytokine balance. It can decrease the function of

pro-inflammatory and anti-inflammatory cytokines,

interferons. Reomannisol is effective in preventing

recurrence of purulent-necrotic lesions of DFS in

conditions of secondary immunodeficiency, so it is a

necessary drug in the main therapy complex.

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(ISSN

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AGES

:

38-52

OCLC

1121105677
















































Publisher:

Oscar Publishing Services

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Эрназаров Х. и др. ПАТОМОРФОЛОГИЧЕСКАЯ

КАРТИНА ЖИЗНЕННО ВАЖНЫХ ОРГАНОВ ПРИ

ЭКСПЕРИМЕНТАЛЬНОЙ

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84-95.

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Эргашев

У.

Ю.

и

др.

ИЗУЧЕНИЕ

ПАТОМОРФОЛОГИИ

ПЕЧЕНИ

ПРИ

ЭКСПЕРИМЕНТАЛЬНОМ

СИНДРОМЕ

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Зохиров А. Р., Набиева А. Ш. ИЗУЧЕНИЕ

ПАТОМОРФОЛОГИЧЕСКИХ

ОСОБЕННОСТЕЙ

СОВРЕМЕННОГО

ЛЕЧЕНИЯ

ГНОЙНО

-

НЕКРОТИЧЕСКИХ ПРОЦЕССОВ ПРИ САХАРНОМ

ДИАБЕТЕ //Interpretation and researches. –

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Т. 1. –

№. 2. –

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Эрназаров Х. И. и др. ЭФФЕКТИВНОСТЬ

ИСПОЛЬЗОВАНИЕ ПРЕПАРАТА РЕОМАННИСОЛ

В ЛЕЧЕНИИ ЭКСПЕРИМЕНТАЛЬНОЙ МОДЕЛИ

ДИАБЕТИЧЕСКОЙ СТОПЫ. –

2022.

18.

Зохиров А. Р., Эрназаров Х. И., Эргашев У. Ю.

ПАТОМОРФОЛОГИЧЕСКИЕ

ОСОБЕННОСТИ

ЗАЖИВЛЕНИЯ РАН ПРИ ЭКСПЕРИМЕНТАЛЬНОЙ

МОДЕЛИ ДИАБЕТИЧЕСКОЙ СТОПЫ. –

64-

ОЙ

НАУЧНО

-

ПРАКТИЧЕСКОЙ

КОНФЕРЕНЦИИ

ОБУЧАЮЩИХСЯ

«НАУКА

И

ЗДОРОВЬЕ»

ПОСВЯЩЕННАЯ ДНЮ НАУКИ РЕСПУБЛИКИ

КАЗАХСТАН С МЕЖДУНАРОДНЫМ УЧАСТИЕМ,

2022.

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Yusufjanovich E. U. et al. PRINCIPLES OF STUDYING

LIVER MORPHOLOGY IN EXPERIMENTAL DIABETIC

FOOT SYNDROME //World Bulletin of Public Health.

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Т. 19. –

С. 63

-65.

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Rafiqovich Z. A. et al. IMPROVING THE DETECTION

OF MORPHOLOGICAL CHANGES IN PURULENT

WOUNDS //E Conference Zone.

2023.

С. 51

-57.

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Yusufjanovich E. U. et al. Assessment of the Process of Epithelialization After Complex Treatment of Diabetic Foot Syndrome //Texas Journal of Medical Science. – 2023. – Т. 16. – С. 19-23.

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Yusufjanovich E. U. et al. Treatment of purulent-necrotic lesions of the lower extremities with modern drugs //Conferencea. – 2023. – С. 88-94.

Ergashev U. Y. et al. THE STUDY OF DIAGNOSTICS AND PREVENTION OF PATHOPHYSIOLOGICAL PARAMETERS AFTER MODERN TREATMENT OF PURULENT-NECROTIC PROCESSES IN DIABETIC. – 2022.

Ergashev U. Y. et al. THE STUDY OF PATHOMORPHOLOGICAL DIAGNOSIS OF VITAL ORGANS AFTER MODERN TREATMENT OF DIABETIC FOOT SYNDROME. – 2022.

Rafiqovich Z. A. CONTROL OF INDICATORS OF ENDOTOXICOSIS IN DIABETIC FOOT SYNDROME //Conferencea. – 2023. – С. 83-90.

Rafiqovich Z. A. STUDY OF THE EFFECT OF LIPID PEROXIDASE ANALYSIS ON THE BODY IN DIABETIC FOOT SYNDROME //Conferencea. – 2023. – С. 76-82.

Rafiqovich Z. A. MONITORING OF THE REGENERATION PROCESS IN PURULENT-NECROTIC PROCESSES OF THE LOWER EXTREMITIES //Conferencea. – 2023. – С. 189-194.

Зохиров А. Р. ОБОСНОВАНИЕ ПРОЦЕССОВ ЭПИТЕЛИЗАЦИИ И РЕГЕНЕРАЦИИ ПРИ ГНОЙНО-НЕКРОТИЧЕСКИХ ПРОЦЕССАХ НИЖНИХ КОНЕЧНОСТЕЙ ПРИ САХАРНОМ ДИАБЕТЕ //Conferencea. – 2023. – С. 174-180.

Ergashev U. Y. Ernazarov Kh. I., Zohirov AR, Alzabni ID 2022. Complex Treatment of Experimental Model of Diabetic Foot Syndrome //American Journal of Medicine and Medical Sciences. – 2022. – Т. 12. – №. 5. – С. 471-480.

Rafiqovich Z. A. OBSERVATION OF BIOCHEMICAL RESULTS IN EXPERIMENTAL DIABETIC FOOT SYNDROME //Conferencea. – 2023. – С. 181-188.

Ergashev U. Y., Zokhirov A. R., Minavarkhujaev R. R. Determination of changes in the lipid peroxidase index in purulent-necrotic lesions of the lower extremities. – 2022.

Эрназаров Х. и др. ПАТОМОРФОЛОГИЧЕСКАЯ КАРТИНА ЖИЗНЕННО ВАЖНЫХ ОРГАНОВ ПРИ ЭКСПЕРИМЕНТАЛЬНОЙ МОДЕЛИ ДИАБЕТИЧЕСКОЙ СТОПЫ. – 2022.

Ergashev U. Y. et al. IMPROVING METHODS FOR DIAGNOSING AND MONITORING ENDOTOXICOSIS IN EXPERIMENTAL DIAETIC FOOT SYNDROME //World Bulletin of Public Health. – 2023. – Т. 19. – С. 84-95.

Эргашев У. Ю. и др. ИЗУЧЕНИЕ ПАТОМОРФОЛОГИИ ПЕЧЕНИ ПРИ ЭКСПЕРИМЕНТАЛЬНОМ СИНДРОМЕ ДИАБЕТИЧЕСКОЙ СТОПЫ //European Journal of Interdisciplinary Research and Development. – 2023. – Т. 12. – С. 27-31.

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Зохиров А. Р., Эрназаров Х. И., Эргашев У. Ю. ПАТОМОРФОЛОГИЧЕСКИЕ ОСОБЕННОСТИ ЗАЖИВЛЕНИЯ РАН ПРИ ЭКСПЕРИМЕНТАЛЬНОЙ МОДЕЛИ ДИАБЕТИЧЕСКОЙ СТОПЫ. – 64-ОЙ НАУЧНО-ПРАКТИЧЕСКОЙ КОНФЕРЕНЦИИ ОБУЧАЮЩИХСЯ «НАУКА И ЗДОРОВЬЕ» ПОСВЯЩЕННАЯ ДНЮ НАУКИ РЕСПУБЛИКИ КАЗАХСТАН С МЕЖДУНАРОДНЫМ УЧАСТИЕМ, 2022.

Yusufjanovich E. U. et al. PRINCIPLES OF STUDYING LIVER MORPHOLOGY IN EXPERIMENTAL DIABETIC FOOT SYNDROME //World Bulletin of Public Health. – 2023. – Т. 19. – С. 63-65.

Rafiqovich Z. A. et al. IMPROVING THE DETECTION OF MORPHOLOGICAL CHANGES IN PURULENT WOUNDS //E Conference Zone. – 2023. – С. 51-57.