Авторы

  • Akhmadjonov J U., Hoshimov I.M.
    Andijan State Medical Institute.

DOI:

https://doi.org/10.71337/inlibrary.uz.iqro.72100

Ключевые слова:

superficial venous thrombosis varicose vein etiology pathogenesis epidemiology.

Аннотация

 Superficial thrombophlebitis or superficial vein thrombosis (SVT)results from thrombus formation in a superficial vein with associated inflammation of the vessel wall. SVT is most often observed in the lower extremities, with greater saphenous vein (GSV) involvement in 60-80% of affected individuals. SVT is 6-fold more common than venous thromboembolism (VTE) with a yearly incidence rate of 0.64%. It is important to note that SVT is different from thrombus within the superficial femoral vein which is a deep vein and requires the same approach to management as deep vein thrombosis (DVT) in other deep veins. Risk factors for SVT are similar to those for deep vein thrombosis (DVT) and pulmonary embolism (PE) and include active malignancy or cancer therapy, surgery, venous procedures, trauma/injury, immobilization, obesity, estrogen use/pregnancy (particularly in the first month postpartum), a personal or family history of VTE, and inherited thrombophilia. In addition, SVT often occurs in the presence of varicose veins (present in up to 80% of SVT patients) and, in the upper extremities, is often associated with intravenous catheters. SVT is a risk factor for concomitant and future VTE.


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JOURNAL OF IQRO – ЖУРНАЛ ИҚРО – IQRO JURNALI – volume 14, issue 02, 2025

ISSN: 2181-4341, IMPACT FACTOR ( RESEARCH BIB ) – 7,245, SJIF – 5,431

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ILMIY METODIK JURNAL

Akhmadjonov J U., Hoshimov I.M.

Andijan State Medical Institute.

Email :Doctor_invincible@mail.ru

ORCID ID: horcid.org/0000-0003-0362-8357

MODERN APPROACHES TO THE PREVENTION OF THROMBOPHLEBITIS:

LITERATURE REVIEW

Annotation.

Superficial thrombophlebitis or superficial vein thrombosis (SVT)results from

thrombus formation in a superficial vein with associated inflammation of the vessel wall. SVT is

most often observed in the lower extremities, with greater saphenous vein (GSV) involvement in

60-80% of affected individuals. SVT is 6-fold more common than venous thromboembolism

(VTE) with a yearly incidence rate of 0.64%. It is important to note that SVT is different from

thrombus within the superficial femoral vein which is a deep vein and requires the same

approach to management as deep vein thrombosis (DVT) in other deep veins. Risk factors for

SVT are similar to those for deep vein thrombosis (DVT) and pulmonary embolism (PE) and

include active malignancy or cancer therapy, surgery, venous procedures, trauma/injury,

immobilization, obesity, estrogen use/pregnancy (particularly in the first month postpartum), a

personal or family history of VTE, and inherited thrombophilia. In addition, SVT often occurs in

the presence of varicose veins (present in up to 80% of SVT patients) and, in the upper

extremities, is often associated with intravenous catheters. SVT is a risk factor for concomitant

and future VTE.

Keywords:

superficial venous thrombosis, varicose vein, etiology, pathogenesis, epidemiology.

Introduction.

Superficial venous thrombosis (SVT), an inflammatory–thrombotic process of a

superficial vein, is a relatively common event that may have several different underlying causes.

This phenomenon has been generally considered benign, and its prevalence has been historically

underestimated; the estimated incidence ranges from about 0.3 to 1.5 event per 1000 person-

years, while the prevalence is approximately 3 to 11%, with different reports depending on the

population studied. However, such pathology is not free of complications; indeed, it could extend

to the deep circulation and embolize to pulmonary circulation. For this reason, an ultrasound

examination is recommended to evaluate the extension of SVT and to exclude the involvement

of deep circulation. Also, SVT may be costly, especially in the case of recurrence. Therefore,

accurate management is necessary to prevent sequelae and costs related to the disease. This

review aims to analyse the epidemiology of SVT, its complications, optimal medical treatment,

and open questions with future perspectives. SVT is a relatively common disease that mostly

affects the lower limbs. The incidence is estimated to be 0.3–0.6 events per 1000 person-years in

young people and 0.7–1.5 events per 1000 person-years in older patients [1], not so different than

DVT, which is estimated to be about 1 in every 1000 cases [2]. However, the real incidence of

STV is probably underestimated.

Materials and methods

. The STEPH study was a descriptive, multicentre, community-based

study conducted over 1 year in the adult resident population of an urban area in France. The

study included 265,687 adults and 171 of them had symptomatic SVT, confirmed with

ultrasound performed by vascular specialists. The measured annual diagnosis rate of SVT was

0.64% of adults [95% confidence interval (CI) 0.55% to 0.74%]; however, it is possible that the

primary care setting underestimated the true prevalence [7].

Another study analysed the


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JOURNAL OF IQRO – ЖУРНАЛ ИҚРО – IQRO JURNALI – volume 14, issue 02, 2025

ISSN: 2181-4341, IMPACT FACTOR ( RESEARCH BIB ) – 7,245, SJIF – 5,431

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ILMIY METODIK JURNAL

prevalence of clinically diagnosed SVT in the Swiss population. The prevalence reported was

about 3–11% depending on the population analysed . Notably, the latter two studies are old and

based only on clinical diagnoses; therefore, the real prevalence could be underestimated. The

venous thrombosis prevalence rate was found to be 2.9% in women and 0.8% in men aged ≥49

in an Italian study (Anemone) that used a self-managed questionnaire to investigate the

prevalence of the condition in blood donors. After testing for confounding potentials, a

significant and independent association was found between a history of venous thrombosis and

age. Another interesting point is the seasonality of SVT. A small retrospective study showed an

increase in the summer months; a possible explanation could be the poor compliance of patients

to pharmacologic therapy and elastic stockings . Other reports did not confirm such an

observation, although a small increase was observed in summer .

Results.

It is worth noting that most data about patients with SVT are derived from old studies

with methodological limitations. New studies using diagnostic technologies such as ultrasound

could allow for a better epidemiological definition of pathology and complications.

The risk

factors for SVT and DVT are similar: advanced age, varicose veins, pregnancy, post-operative

states, immobilization, malignant neoplasms, autoimmune diseases, obesity, trauma,

hypercoagulable states, use of oral contraceptives or hormonal therapies, previous episodes of

DVT/PE, vascular access, infusion of hypertonic solutions or endothelial damaging substances,

and autoimmune diseases [4].

Unlike DVT, varicose veins are the primary risk factor for lower-limb SVT, and they are found

in 90% of cases [8,9]. Among autoimmune disorders, Behcet’s disease has been associated with

SVT onset. Also, patients with Buerger’s disease are particularly susceptible to SVT.

Interestingly, in such cases a peculiar inflammation of the three layers of the vessel has been

described [4,10]. Superficial thrombophlebitis starts with microscopic thrombosis. When venous

turbulence or stasis, vessel wall injuries, abnormal coagulability, or vessel wall injuries,

microthrombi could propagate and subsequently form macroscopic thrombi. Vascular endothelial

injury reliably results in thrombus formation by triggering an inflammatory response that results

in immediate platelet adhesion. Platelet aggregation is mediated by thrombin and thromboxane

A2.

Predisposition to the occurrence of varicose veins, working conditions that contribute to this, as

well as the appearance of symptoms of the disease are sufficient grounds for compression

therapy, it seems to be the most reliable and affordable way to correct venous outflow and the

work of the musculo-venous pump of the lower leg. Elastic bandages or therapeutic knitwear

(knee socks, stockings or tights) can be used.[6] Compression hosiery prevents stretching of

superficial veins and accelerates blood flow through the deep venous system.

The simplest method of elastic compression is leg bandaging. In this case, bandages of medium

and short extensibility are used. Compression bandage should be applied in the morning, before

the patient gets out of bed. The doctor must teach the patient the technical features of this

procedure. The bandage begins to be applied from the foot, immediately at the base of the toes,

then it is wound in a spiral so that each subsequent turn covers the previous one by 2/3 of the

width Bandage. It is especially important that the heel is tightly bandaged - for this purpose, the

"hammock" technique is used. It is necessary to inform the patient that the maximum stretch and,

accordingly, pressure of the bandage should be exerted on the foot and in the ankle area, they

should be gradually reduced towards the hip.

Convenient for the patient and the most adequate from the point of view of normalizing venous

outflow from the lower extremities, undoubtedly, is the use of special compression hosiery

.

With its help, a fixed, graduated pressure is created, gradually decreasing from the periphery to

the center

.

The task of the doctor is to recommend the necessary type of product (knee socks,


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JOURNAL OF IQRO – ЖУРНАЛ ИҚРО – IQRO JURNALI – volume 14, issue 02, 2025

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ILMIY METODIK JURNAL

stockings or tights) and the class of compression. [5] The patient should choose the product

strictly according to his size and use it regularly. Currently, there is a sufficient selection of high-

quality medical knitwear in a wide price range on the Russian market.

To prevent the progression of chronic venous insufficiency and the development of

varicothrombophlebitis, it is advisable to resort to pharmacotherapy. The patient needs to know

that drug treatment solves several problems

:

increasing the tone of the venous wall, improving

microcirculation and lymphatic outflow, relieving inflammation, correcting disorders of the fluid

properties of the blood.

Conclusion.

Finally, in cases of valvular insufficiency of the subcutaneous venous trunks and

perforating veins, patients should be persuaded of the need for timely surgical intervention

.

They

should understand that a properly performed operation protects them from the occurrence of

varicothrombophlebitis and the dangers associated with it.

References

1. Di Nisio M, Wichers IM, Middeldorp S. Treatment for superficial thrombophlebitis of the

leg. Cochrane Database Syst Rev. 2018 Feb 25;2(2):CD004982.

2. Nasr H, Scriven JM. Superficial thrombophlebitis (superficial venous thrombosis). BMJ. 2015

Jun 22;350:h2039.

4. Décousus H, Bertoletti L, Frappé P. Spontaneous acute superficial vein thrombosis of the legs:

do we really need to treat? J Thromb Haemost. 2015 Jun;13 Suppl 1:S230-7.

5. Di Minno MN, Ambrosino P, Ambrosini F, Tremoli E, Di Minno G, Dentali F. Prevalence of

deep vein thrombosis and pulmonary embolism in patients with superficial vein thrombosis: a

systematic review and meta-analysis. J Thromb Haemost. 2016 May;14(5):964-72.

6. Tait C, Baglin T, Watson H, Laffan M, Makris M, Perry D, Keeling D., British Committee for

Standards in Haematology. Guidelines on the investigation and management of venous

thrombosis at unusual sites. Br J Haematol. 2012 Oct;159(1):28-38.

7. Scott G, Mahdi AJ, Alikhan R. Superficial vein thrombosis: a current approach to

management. Br J Haematol. 2015 Mar;168(5):639-45.

8. Maddox RP, Seupaul RA. What Is the Most Effective Treatment of Superficial

Thrombophlebitis? Ann Emerg Med. 2016 May;67(5):671-2.

9.Di Nisio M, Peinemann F, Porreca E, Rutjes AW. Treatment for superficial infusion

thrombophlebitis of the upper extremity. Cochrane Database Syst Rev. 2015 Nov

20;2015(11):CD011015.

10.Varki A. Trousseau's syndrome: multiple definitions and multiple mechanisms. Blood. 2007

Sep 15;110(6):1723-9.

Библиографические ссылки

Di Nisio M, Wichers IM, Middeldorp S. Treatment for superficial thrombophlebitis of the leg. Cochrane Database Syst Rev. 2018 Feb 25;2(2):CD004982.

Nasr H, Scriven JM. Superficial thrombophlebitis (superficial venous thrombosis). BMJ. 2015 Jun 22;350:h2039.

Décousus H, Bertoletti L, Frappé P. Spontaneous acute superficial vein thrombosis of the legs: do we really need to treat? J Thromb Haemost. 2015 Jun;13 Suppl 1:S230-7.

Di Minno MN, Ambrosino P, Ambrosini F, Tremoli E, Di Minno G, Dentali F. Prevalence of deep vein thrombosis and pulmonary embolism in patients with superficial vein thrombosis: a systematic review and meta-analysis. J Thromb Haemost. 2016 May;14(5):964-72.

Tait C, Baglin T, Watson H, Laffan M, Makris M, Perry D, Keeling D., British Committee for Standards in Haematology. Guidelines on the investigation and management of venous thrombosis at unusual sites. Br J Haematol. 2012 Oct;159(1):28-38.

Scott G, Mahdi AJ, Alikhan R. Superficial vein thrombosis: a current approach to management. Br J Haematol. 2015 Mar;168(5):639-45.

Maddox RP, Seupaul RA. What Is the Most Effective Treatment of Superficial Thrombophlebitis? Ann Emerg Med. 2016 May;67(5):671-2.

Di Nisio M, Peinemann F, Porreca E, Rutjes AW. Treatment for superficial infusion thrombophlebitis of the upper extremity. Cochrane Database Syst Rev. 2015 Nov 20;2015(11):CD011015.

Varki A. Trousseau's syndrome: multiple definitions and multiple mechanisms. Blood. 2007 Sep 15;110(6):1723-9.