Авторы

  • Жанна Назарова
    Центр повышения квалификации медицинских кадров
  • Муниса Бахадырова
    Центр повышения квалификации медицинских кадров
  • Дильбар Хидоятова
    Республиканский научный центр экстренной медицинской помощи
  • Малика Душаева
    Республиканский научный центр экстренной медицинской помощи

DOI:

https://doi.org/10.47689/2181-1415-vol4-iss3-pp92-99

Ключевые слова:

ВИЧ вирусный энцефалит ПЦР спинномозговая жидкость

Аннотация

В работе обследовано 124 пациента с острым вирусным энцефалитом, поступивших в РНЦЭМС в период с 2014 по 2019 г. Изучены результаты ПЦР-диагностики ликвора при вирусных энцефалитах с учетом ВИЧ-статуса пациентов. Результаты показали, что ПЦР-тестирование предлагает более надежный диагноз и, когда позволяют лабораторные ресурсы, его следует использовать для анализа спинномозговой жидкости, чтобы более точно идентифицировать этиологический агент, ответственный за ЦСЖ. Для улучшения выявляемости причинных факторов СВЭ и повышения эффективности таргетной терапии, переходя от эмпирических и симптоматических к этиотропным подходам, рекомендуется повторная ПЦР-диагностика ЦСЖ в режиме реального времени в тех случаях, когда причина СВЭ остается неустановленной.


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Etiological aspects of viral encephalitis explored using
PCR-based cerebrospinal fluid analysis, considering

patients’ HIV status

Janna NAZAROVA

1

, Munisa BAKHADIROVA

2

, Dilbar KHIDOYATOVA

3

Malika DUSHAEVA

4


Center for the Development of Professional Skills of Medical Personnel

Republican Scientific Center for Emergency Medical Care

ARTICLE INFO

ABSTRACT

Article history:

Received April 2023
Received in revised form

15 May 2023
Accepted 15 June 2023

Available online

25 June 2023

This study examined 124 patients suffering from acute viral

encephalitis who were admitted to the Center for the

Development of Professional Skills of Medical Personnel

between 2014 and 2019. The research focused on the outcomes
of cerebrospinal fluid PCR diagnostics in viral encephalitis

cases, taking into account patients’ HIV status. Findings

revealed that PCR testing offers a more reliable diagnosis and,

when lab resources permit, should be employed for CSF
analysis to more accurately identify the etiological agent

responsible for SVE. It is advised to repeat real-time PCR

diagnostics of CSF in cases where the cause of SVE remains

undetermined to improve detection rates of SVE’s causative

factors and enhance targeted therapy effectiveness, shifting
from empirical and symptomatic to etiotropic approaches.

2181-

1415/©

2023 in Science LLC.

DOI:

https://doi.org/10.47689/2181-1415-vol4-iss3-pp9

2-99

This is an open access article under the Attribution 4.0 International
(CC BY 4.0) license (https://creativecommons.org/licenses/by/4.0/deed.ru)

Keywords:

HIV,

viral encephalitis,

PCR,

cerebrospinal fluid.

1

Assistant professor, Department of Neurorehabilitation, Center for the Development of Professional Skills of

Medical Personnel. E-mail: janna804@mail.ru

2

Assistant Professor, Neurorehabilitation Department, Center for the Development of Professional Skills of Medical

Personnel. E-mail: m.bakhadirova@mail.ru

3

Attending Physician, Department of Republican Scientific Center for Emergency Medical Care

4

Republican Scientific Center for Emergency Medical Care


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Patsyentov OIV STATUS hisobga olgan holda PCR asosida

miya omurilik suyuqligi tahlil foydalanish virusli ensefalit
etiolohycheskyh aspektlari o

rganildi

ANNOTATSIYA

Kalit so‘zlar

:

OIV,

virusli ensefalit,

PCR,

miya omurilik suyuqligi.

Ushbu tadqiqot 2014 va 2019-

yillar oralig‘ida RSCEMCga

yotqizilgan o‘tkir virusli ensefalit bilan og‘rigan 124 bemorni

tekshirdi. Tadqiqot bemorlarning OIV holatini hisobga olgan

holda virusli ensefalit holatlarida miya omurilik suyuqligining
PCR diagnostikasi natijalariga qaratilgan. Topilmalar shuni

ko‘rsatdiki, PCR testi yanada ishonchli tashxisni taklif qiladi va

agar laboratoriya resurslari ruxsat bergan bo‘lsa, SVE uchun

javobgar bo‘lgan etiologik agentni aniqroq aniqlash uchun

CSF tahlilidan

foydalanish kerak. SVE qo‘zg‘atuvchi omillarini

aniqlash

tezligini

yaxshilash

va

maqsadli

terapiya

samaradorligini oshirish, empirik va simptomatik yondashuvdan

etiotropik yondashuvlarga o‘tish uchun, SVE sabablari noma’lum
bo‘lgan hollarda CSFning real va

qt rejimida PCR diagnostikasini

takrorlash tavsiya etiladi.

Этиологические аспекты вирусного энцефалита,
изученные

с

использованием

ПЦР

-

анализа

церевроспинной жидкости, с учетом ВИЧ

-

статуса

пациентов

АННОТАЦИЯ

Ключевые слова:

ВИЧ,

вирусный энцефалит,

ПЦР,

спинномозговая жидкость.

В работе обследовано 124 пациента с острым вирусным

энцефалитом, поступивших в РНЦЭМС в период с 2014 по

2019 г. Изучены результаты ПЦР

-

диагностики ликвора при

вирусных энцефалитах с учетом ВИЧ

-

статуса пациентов.

Результаты показали, что ПЦР

-

тестирование предлагает

более надежный диагноз и, когда позволяют лабораторные
ресурсы, его следует использовать для анализа
спинномозговой

жидкости,

чтобы

более

точно

идентифицировать этиологический агент, ответственный

за ЦСЖ. Для улучшения выявляемости причинных

факторов СВЭ и повышения эффективности таргетной

терапии, переходя от эмпирических и симптоматических

к этиотропным подходам, рекомендуется повторная

ПЦР

-

диагностика ЦСЖ в режиме реального времени в тех

случаях, когда причина СВЭ остается неустановленной.

Relevance:

Despite the availability of numerous diagnostic methods at the

beginning of the 21st century, the cause of encephalitis remains undetermined in 62% of
patients, and in 10% of cases, a non-infectious origin is diagnosed (3). According to
Lobzin Y.V. and Zhdanov K.V. in their

Manual of Infectious Diseases

, the following


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statistics are reported:

...out of confirmed or probable cases, 69% were viral, 20% were

bacterial, 7% were related to prions, 3% were parasitic, and 1% were fungal. It is crucial
to acknowledge that the inability to determine the cause in many instances may be due to
challenging diagnostic cases, as well as limited access to suitable samples and suboptimal
sample processing; despite extensive laboratory capabilities, the etiology of encephalitis
remained unclear in 64% of patients

(1).

Presently, scientific studies concentrate on the quality of inflammatory and

immune responses in cerebrospinal fluid (CSF) patients, drawing correlations between
local and systemic immune responses in central nervous system (CNS) infections (2,4).
Numerous publications explore neuroinfections during childhood or specifically address
the prevention, diagnosis, and treatment of tick-borne encephalitis (5,6). However, there
is a scarcity of research focusing on the clinical aspects, diagnosis, inflammatory
response, and immune response parameters of CSF in encephalitis among adults,
depending on the etiological factor.

The aim of the research

is to investigate the outcomes of PCR diagnostics in

cerebrospinal fluid for viral encephalitis, considering the HIV status of the patients
involved.

Data and Methods.

We examined 124 patients with severe viral encephalitis

(SVE). The diagnosis of SVE was based on general infectious, general cerebral and focal
neurological symptoms, and the detection of infectious agents. HIV infection was
diagnosed according to the national clinical protocols for HIV infection in Uzbekistan
according to the WHO classification of clinical stages of HIV infection in adults and
adolescents (2010). All patients with SVE were treated as inpatients at the RSCEMC from
2014 to 2019.

Based on HIV status, the patients were divided into 2 groups: Group I

72 (58.1%)

patients with SVE with HIV seronegative status (HIV-) and Group II

52 (41.9%) patients

with SVE with HIV seropositive status (HIV+). Out of the number of SVE patients with HIV
(+), 41 patients (33.1%) had injecting drug addiction.

In Group I, the average age of the patients was 49.82 ± 2.96 years old; there were

45 (62.5%) and 27 (37.5%) male and female patients, respectively. In Group 2, the

average age of patients was 42.28 ± 2.74 years. There were 29 (55.8%) men and

23 (44.2%) women. The age and sex distribution of Group 2 patients are shown in Table 1.

Table 1.

Distribution of patients by sex and age.

Group I

SVE, HIV (-) patients n=72 (58,1%)

Age

(Years)

men

women

all

n

%

n

%

n

%

18-44

21

29,20%

15

20,80%

36

50,00%

45-59

14

19,40%

9

12,50%

23

31,90%

60-74

10

13,90%

3

4,20%

13

18,10%

TOTAL

45

62,5%**

27

37,50%

72

100,00%

Group II

SVE, HIV (+) patients n=52 (41,9%)

Age

(Years)

men

women

all

n

%

n

%

n

%

18-44

19

36,50%

15

28,80%

34

65,40%


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45-59

8

15,40%

8

15,40%

16

30,80%

60-74

2

3,80%

0

0,00%

2

3,80%

total

29

55,8%*

23

44,20%

52

100,00%

TOTAL

74

59,7%

50

40,3%

124

100,00%


As it was shown in Table 1, the structure of patients in Group I was somehow

different from that in Group II. Thus, males prevailed in both groups (62.5% and 55.8%
in groups I and II, respectively). It should also be noted that there was a percentage shift
toward the younger age of patients in group I compared to group II.

Patients were examined according to the following algorithm: complaints, medical

history; general clinical tests; biochemical tests; diagnostic lumbar puncture (LP) with
the clinical and biochemical examination; cerebrospinal fluid, diagnostic LP with PCR;
MRI of the brain.

Statistical processing of clinical and instrumental materials in accordance with the

recommendations for processing the results of biomedical research at the significance
level of p<0.05 was carried out using a practical statistical package STATISTICA.

Results and discussion.

Diagnostic lumbar puncture is one of the first and

indicative methods for early diagnosis of meningoencephalitis. When performing a
diagnostic lumbar puncture, pleocytosis was determined in all patients under study. With
increasing duration of SVE (according to anamnesis) initially neutrophilic or mixed
pleocytosis became lymphocytic in 4-5 days, the number of cells decreased, and in some
patients there was observed the formation of single erythrocytes in the CSF (Table 2).

Table 2.

CSF study indicators in patients with SVE, (M±σ

)

Studied indicator

Group I (n=72)

Group II (n=52)

Pressure (mm. of water column) (M±σ

)

198,2±12,6

187,9±16,4*

Cytosis (in 1

μl) (M±σ

)

4285±173,6

4762±189,2*

Cellular composition (M±σ)

Lymphocytes

68,2±12,3%

63,8±17,4%

Monocytes

26,7±9,2%

31,6±11,3%*

Erythrocytes

3,2±1,3%

5,4±1,7%

Protein (g/l) (M±σ

)

2,64±0,76

3,17±0,79*

D-Dimer of fibrin mg/

ml (M±σ

)

8,3±1,6

11,4±2,3

Lactate (mmol/l) (M±σ

)

8,7±1,4

10,1±1,6*

Glucose (mmol/l) (M±σ

)

2,5±0,6

2,2±0,8

Note: * significant differences between groups p<0.005.


The cerebrospinal fluid pressure was measured using a U-shaped manometer tube

filled with water in the supine position of the patient. As can be seen from Table 2,
the pressure of CSF in group I was higher than in group II

198.2 mm. of the water

column and 187.9 mm. of the water column, respectively.


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Fig. 1. Etiological factors of SVE in patient groups according to the first

PCR-diagnostics of CSF (abs; %).

The severity of reactive changes in the CSF was stronger in the HIV-negative group

compared to the HIV-positive group, while the overall neurological status and clinical
manifestations were stronger in the HIV-positive group. Thus, in the study of CSF, block
content values were higher in group II patients by 18.7%, D-dimer of fibrin prevailed by
27.2%, and lactate was up by 13.9% compared with group I.

To determine the causative agent of SVE, we used PCR diagnostics of CSF to detect

genetic material of HSV-1, HSV-2, VZV, enteroviruses, and CMV. PCR testing of CSF was
performed in the laboratory of the Research Institute of Virology, in the reference
laboratory of the Republic of Uzbekistan.


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Table 3.

Etiological factors of SVE depending on the presence of HIV infection

Pathogen

Group I (n=72)

p<

Group II (n=52)

n

%

n

%

HSV-1

42

58,3%

0,005

7

13,5%

HSV-2

0

0,0%

0,001

11

21,2%

Enteroviruses

6

8,3%

3

5,8%

VVZ

6

8,3%

5

9,6%

CMV

0

0,0%

0,005

14

26,9%

Non defined

18

25,0%

12

23,1%


PCR diagnostics of cerebrospinal fluid revealed that in Group I HSV-1 prevailed

in 42 patients (58.3%), the pathogen could not be detected in 18 patients (25.0%),
VVZ and enteroviruses were detected in 6 patients (8.3%) in each case. In Group II, CMV
was found in 14 patients (26.9%), which was significantly more frequent than in Group
I patients. In Group II, the frequent causative agents of CVE were also HSV-2 and HSV-1,
in 21.2% and 13.5% of patients, respectively. And in 23.1% of patients, the pathogen
agent of SVE was not identified (Fig. 1, Table 3).

A repeated real-time PCR diagnosis of CSF was performed to clarify the etiological

factor among the unspecified cases of SVE in both groups. The dynamics of the indicators
in the pathogen profiles are shown in Figure 2 and Table 4.

As can be seen from Figures 1 and 2, after a repeated PCR study of CSF for the

presence of viruses, 14 more cases of SVE were identified overall, 9 cases in Group I and
5 cases in Group II.

Dynamics of pathogen profile in group I after repeated PCR diagnostics of

CSF testing of unspecified cases of SVE


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Dynamics of pathogen profile in group II after repeated PCR diagnostics of

CSF testing of unspecified cases of SVE

Figure 2. The dynamics of the profiles of etiological factors of SVE in the groups of

patients after repeated PCR-diagnostics of CSF (abs; %).

As can be seen from Tables 3 and 4, the structure of the etiological factors of SVE in

both groups slightly changed. In group I, repeated PCR diagnostics of CSF additionally
revealed 4 cases of HSV-1, and 4 cases of ZZV, in group II one patient with CMV and one
patient with enterovirus.

Also, in group II, opportunistic infections were detected: cryptococcus

8 cases

(15.3%) and toxoplasma

5 cases (9.6%), Epstein-Barr virus

22 cases (42.3%).

Table 4.

Etiological factors of SVE depending on the presence of HIV infection after repeated

PCR examination of CSF

Pathogen

Group I (n=72)

p<

Group II (n=52)

n

%

n

%

HSV-1

48

66,7%

0,0005

7

13,5%

HSV-2

0

0,0%

0,005

11

21,2%

Enteroviruses

6

8,3%

4

7,7%

VVZ

10

13,9%

5

9,6%

CMV

0

0,0%

0,005

15

28,8%

Non defined

8

11,1%

10

19,2%


In conclusion, PCR-based diagnostics are more dependable, and when lab

resources permit, employing PCR analysis of cerebrospinal fluid is recommended to more
accurately identify the etiological agent responsible for SVE. In cases where the cause of
SVE remains uncertain, repeating real-time PCR diagnostics of CSF is advised to enhance


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detection rates of SVE’s causative factors and subsequently improve the efficacy of

targeted therapy, transitioning from an empirical and symptomatic approach to an
etiotropic one.

REFERENCES:

1.

Rukovodstvo po infeksionnim boleznyam. [Handbook of Infectious Diseases.

In 2 vols. 2 / ed. by Acad. V. Lobzin, Prof. K.V. Zhdanov.

4-th edition, updated and

revised.

Saint-Petersburg: Publishing House Foliant, 2011.

744 p.]

2.

Ensefaliti v klinicheskoy praktike

tak li vse prosto? // Klinicheskaya

mikrobiologiya i antimikrobnaya ximioterapiya [Karpov I.A., Kachanko E.F., Vasilenko
A.I., Y.L. et al. Encephalitis in clinical practice

is it that simple? // Clinical Microbiology

and Antimicrobial Chemotherapy, 2011, Vol. 13, No. 2, pp.104-133.]

3.

Obshaya nevrologiya. [Nikiforov A.S., Gusev E.I. General neurology.

Moscow:

GEOTAR-Media. 2013.]

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Sovremenniye aspekti rasprostranennosti i etiologii razvitiya virusnix

ensefalitov // Sovremenniye problemi nauki i obrazovaniya. [Reveguk E.A., Karpov S.M.,
Zavodnova O.S. Modern aspects of prevalence and etiology of viral encephalitis //
Modern problems of science and education.

2018.

№ 4. –

P.249-254.]

5.

Molecular Virology / S. Modrow, D. Falke, U. Truyen, H. Schätzl. –

B. etc.:

Springer, 2013.

1013 p.

6.

Spatola M, Du Pasquier RA. Immune system’s role in viral encephalit

is. Rev

Neurol (Paris). 2014; 170:577

83.

Библиографические ссылки

Rukovodstvo po infeksionnim boleznyam. [Handbook of Infectious Diseases. In 2 vols. 2 / ed. by Acad. V. Lobzin, Prof. K.V. Zhdanov. - 4-th edition, updated and revised. - Saint-Petersburg: Publishing House Foliant, 2011. - 744 p.]

Ensefaliti v klinicheskoy praktike – tak li vse prosto? // Klinicheskaya mikrobiologiya i antimikrobnaya ximioterapiya [Karpov I.A., Kachanko E.F., Vasilenko A.I., Y.L. et al. Encephalitis in clinical practice - is it that simple? // Clinical Microbiology and Antimicrobial Chemotherapy, 2011, Vol. 13, No. 2, pp.104-133.]

Obshaya nevrologiya. [Nikiforov A.S., Gusev E.I. General neurology. - Moscow: GEOTAR-Media. 2013.]

Sovremenniye aspekti rasprostranennosti i etiologii razvitiya virusnix ensefalitov // Sovremenniye problemi nauki i obrazovaniya. [Reveguk E.A., Karpov S.M., Zavodnova O.S. Modern aspects of prevalence and etiology of viral encephalitis // Modern problems of science and education. - 2018. - № 4. - P.249-254.]

Molecular Virology / S. Modrow, D. Falke, U. Truyen, H. Schätzl. – B. etc.: Springer, 2013. – 1013 p.

Spatola M, Du Pasquier RA. Immune system's role in viral encephalitis. Rev Neurol (Paris). 2014; 170:577–83.

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