Authors

  • A. Mengliyev
    Тermez State University Of Engineering And Agrotechnology
  • I. Salimov
    Veterinary Research Institute
  • I. Siddikov
    Veterinary Research Institute
  • I. Kho'jakulov
    Samarkand Agroinnovations And Research Institute.
  • D. Sodiqova
    Samarkand Agroinnovations And Research Institute.
  • D. Daliyev
    Samarkand Agroinnovations And Research Institute.

DOI:

https://doi.org/10.71337/inlibrary.uz.ijai.102761

Abstract

This article outlines the clinical signs, diagnostics, and prevention of nodular dermatitis in cattle, a disease that has been poorly studied by our scientists in Uzbekistan and is currently a pressing problem.

 

 

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UDK.: 619.616.9 – 036.2:576.807.3

CLINICAL SIGNS, DIAGNOSIS AND PREVENTION OF NODULAR

DERMATITIS IN CATTLE

A.S.Mengliyev

Тermez State University Of Engineering And Agrotechnology. (Ph.D)

Alimengliyev1972@Gmail.Com

I.Kh.Salimov,

Veterinary Research Institute, Senior Researcher, Deputy Doctor Of Science.

I.N. Siddikov

,

Senior Lecturer (Veterinary Research Institute, “Food Safety And Technology”), Samarkand

Agroinnovations And Research Institute. (Ph.D)

I.S.Kho'jakulov, D.S.Sodiqova., D.A.Daliyev.

Students Of Termez State University Of Engineering And Agrotechnology.

Annotatsiya.

Ushbu maqolada O’zbekistonda hozirgi kungacha olimlarimiz tomonidan кам

o’ganilgan bugungi kunda dolzarb muommo bo’lib kelayotgan kasalliklardan biri bo’lgan

qoramollarning nodelyarniy dermatit kasalligining klinik belgilari,diagnostikasi va oldini

olish vazifalari belgilab berilgan.

Аннотация

. В статье изложены клинические признаки, диагностика и профилактика

нодулярного дерматита крупного рогатого скота – заболевания, которое мало изучено

нашими учеными в Узбекистане и в настоящее время является актуальной проблемой.

Annotation.

This article outlines the clinical signs, diagnostics, and prevention of nodular

dermatitis in cattle, a disease that has been poorly studied by our scientists in Uzbekistan and

is currently a pressing problem.

Kalit so'zlar:

qoramol,infeksiya,virus,isitma,teri tugun, yallig’lanish,teri bo’rtmasi,davolash

va oldini olish, сўлак, уруғ, сут, кўз ва бурун,суюқлик, трансмиссив йўл, ҳашаротлар,

ўчоқли тери қопламида бўртмалар, думалоқ, сон, қўлтиқ, кўз атрофи, тумшуқ, елин.

Ключевые слова:

крупный рогатый скот, инфекция, вирус, лихорадка, узелок на коже,

воспаление, кожная сыпь, лечение и профилактика, слюна, сперма, молоко, глаза и нос,

жидкость, путь передачи, насекомые, локализованная кожная сыпь, пах, бедро,

подмышечная впадина, вокруг глаз, морда, крестец.


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Key words:

cattle, infection, virus, fever, skin nodule, inflammation, skin rash, treatment and

prevention, saliva, semen, milk, eyes and nose, fluid, transmission route, insects, localized

skin rash, groin, thigh, armpit, around the eyes, snout, rump.

Introduction.

Lumbar dermatitis of cattle (Dermatitis nodularis bovum; contagious nodular

dermatitis) is a contagious viral disease that knows no borders, characterized by persistent

fever, necrotizing focal skin rashes (lumpy swelling), widespread lymphadenitis, lesions of

the mucous membranes of the eyes, respiratory and digestive organs. Sheep, goats and

buffaloes can also sometimes be infected with this disease.
The somewhat complicated epizootic situation with lumpy dermatitis of cattle in the world,

the proximity of its distribution area to the border regions of our country and the potential

danger require the provision of information about this disease.

Relevance of the topic.

Lumpy skin disease (LSD) of cattle was recorded in Central and

Southern African countries, Madagascar, and India. Later, in the Middle East, during 2014-

2016, LSD of cattle was recorded in Turkey, Azerbaijan, Lebanon, Iraq, Iran, Egypt, Cyprus,

the Russian Federation, the Republics of Dagestan, Chechnya, Armenia, Greece, Bulgaria,

Macedonia, Serbia, Montenegro, Albania, and Kazakhstan. This disease also causes serious

damage in Uzbekistan.

Course and clinical signs.

The incubation period of the disease is 3-30 days, the incubation

period in natural conditions is 2-4 weeks, and in experimental conditions it is 7-8 days.

Usually, when the virus suspension is infected subcutaneously and between the skin, in cattle,

after 4-7 days, the inflammatory process begins at the site of damage and spreads to the

epidermis, dermis, and nearby muscles. Exudate accumulates in the bumps that appear, then

necrosis develops. Injured areas of the skin will be painful. Intensification of these processes

occurs 7-19 days after the animal is infected, and fever is observed. The virus can be detected

in the blood 3-4 days after the div temperature rises and the rash appears. The virus spreads

through the blood throughout the animal's div over 1-2 weeks, penetrating the tissues of the

oral cavity, nose, eyes, vagina, prepuce, salivary and mammary glands, testicles, and other

organs, causing necrotic inflammation.

In the acute course of the disease, the div temperature rises to +40°C and remains at this

level for 4-14 days, there is a decrease in appetite, discharge from the eyes, mucous or

purulent discharge from the nose and mouth, and after 48 hours, the formation of focal skin

rashes is observed. These rashes are slightly raised above the skin surface, round, clearly

demarcated, 0.2-7.0 cm in size, and depending on the course of the disease, their number can

range from a few to several hundred. They are located throughout the div, mainly on the

thighs, armpits, around the eyes, on the muzzle, and on the groin.

In severe cases, the lesions may also be located on the mucous membranes of the mouth and

nose, and on the labia minora. Nodular lesions may form on the eyelids, causing blurred

vision and partial or complete blindness. 1-3 weeks after the lesions appear, the tissue inside

them becomes completely necrotic, forming sequestrations. Later, the lesions rupture,

releasing a foul-smelling, viscous mucus.


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As the swellings rupture and heal, the bumps and their signs of inflammation disappear

(within 4-6 weeks). The wool in their place falls out. The bumps sometimes harden and can

remain in this state for up to a year. Eventually, they either resorb or, more often, undergo

necrosis, forming a dry scab, under which granulation tissue forms. During the healing and

scarring process, these wounds are often contaminated with various secondary microflora,

which leads to complications. Lymph nodes, especially the prescapular and axillary lymph

nodes, enlarge. Sick animals lose weight quickly, their productivity decreases. In dairy cows,

due to injuries to the udders, the milk thickens, acquires a pinkish color, is milked in drops,

and when heated, turns into a jelly-like state.

The disease progresses with damage to the respiratory, digestive, reproductive organs and

joints, and may be accompanied by abdominal-type difficulty breathing, profuse salivation,

mucous or mucopurulent conjunctivitis, clouding of the conjunctiva, and enlarged lymph

nodes. In cows, abortion, mastitis, and impaired reproductive function may occur, and in

bulls, temporary impotence or complete infertility may occur.

In calves, ND can occur without visible lesions of the skin. In this case, the disease is

characterized by fever, diarrhea with mucus and blood. In the subacute course, no significant

lesions of the skin are observed. The disease is accompanied by short-term fever (2-5 days),

loss of appetite. The disease can also occur without clinical signs, in which case the disease is

diagnosed only by detecting the DNA of the causative virus in the polymerase chain reaction

(PCR) or by the presence of virus-neutralizing antibodies. Infected animals without clinical

signs in the affected herd can reach up to 50%.

Diagnosis.

A preliminary diagnosis is made based on clinical signs (clearly demarcated

lesions on the skin, in severe cases, lesions on the mucous membranes, lesions clearly

demarcated from healthy skin, superficial lymph nodes are involved), analysis of

epizootological data (the disease occurs suddenly on several farms at once, the number of

infected animals increases rapidly, sometimes reaching 70%), and pathological changes.

The final diagnosis is made on the basis of laboratory tests. Rashes are used as pathological

material for virus isolation, viral nucleic acid in rash scabies can be detected by PCR for 3

months. Cell culture and neutralization reaction are used to isolate and identify the virus.

Currently, molecular genetic methods are used for diagnosis. In cases where the virus or

antigen, genome of ND is detected, the final diagnosis is considered to be made. For this

purpose, reactions such as PCR, IFA and RSK (RDSK) are used.

Differential diagnosis

. Animal ND should be differentiated from dermatophilosis (a chronic

skin lesion characterized by the formation of scaly, raised papules on the surface of the skin);

from the cutaneous form of tuberculosis (the lesions are located under the skin along the

joints and cervical lymph nodes and persist for a long time); from the skin reaction to insect

bites (painful lesions are well felt, the inflammation is not limited to the papule, the lesions

are soft and spread), from protein, sheep bluetongue, demodicosis, smallpox, infectious

rhinotracheitis, and helminth larvae lesions.


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Specific prophylaxis.

Animals that have recovered from the disease do not become infected

with ND again. According to some scientists, immunity in animals that have recovered from

the disease lasts for 11 months.

For active specific prophylaxis, a homologous live attenuated virus vaccine from the

Neethling strain and a heterologous live attenuated virus vaccine from strains of

capripoxviruses isolated from sheep and goats are used. All strains of capripoxviruses used as

vaccines can cause a strong local reaction at the injection site. The vaccination dose with the

nodular dermatitis virus vaccine is 2.5 lg50/cm3, and the vaccination dose with the vaccine

prepared from the sheep and goat pox virus is 5 lg50/cm3.

For specific prophylaxis, the first scheduled vaccination is carried out on 3-month-old young

animals. Revaccination is carried out after 12 months. In unhealthy areas and farms in risk

zones, all healthy animals are vaccinated, regardless of the period of previous immunization.

Young animals up to 6 months of age are vaccinated 2 times with an interval of 14 days.

Prevention and control measures

. There is no ND in Uzbekistan and has never been.

However, as noted above, this disease is present in some countries of Asia, Europe, and

Africa, so the risk of the disease entering the country remains. The way to protect susceptible

animals is to strictly monitor the dangerous border area, cattle crossing the border and their

products, immediately report any suspicions, and conduct clinical and laboratory

examinations of newly arrived animals during preventive quarantine. The main attention

should be paid to preventing the introduction of the pathogen from foreign countries. Cattle

imported into the country, their meat, dairy products, and semen should be purchased from

countries that are healthy in terms of ND. Cattle should be tested for ND during a 30-day

preventive quarantine, and only healthy animals should be allowed onto the farm. It is not

advisable to keep cattle and sheep and goats in border areas with foreign countries.

Organizing farms as a closed enterprise, organizing entrances to buildings through

disinfectants, feeding animals with nutritious feed, housing them in accordance with

zoohygienic standards, timely isolating, keeping and treating sick animals, keeping the farm

area clean, and conducting disinfection and disinfestation measures on the farm based on a

plan will help prevent this disease.

If this disease is detected in cattle by one of the clinical, pathological and laboratory methods,

within the framework of the Veterinary Law, the farm or settlement is declared unhealthy by

the decision of the khokim based on the act of the chief veterinary inspector of the district

(city) and quarantined as an exotic disease. All quarantine measures are taken in the

unhealthy settlement and measures are taken to prevent the spread of the disease. All types of

animals, unauthorized persons, and mixing groups of animals are prohibited from entering

and leaving the farm.

When ND is first detected among cattle, sick animals and those in direct contact with them

are isolated in the epizootic focus, they are killed by a bloodless method, and the carcasses

are destroyed. Sick animals that died, uneaten feed and bedding are burned in the territory of

the unhealthy point. Milk obtained from animals in the epizootic focus is sold without


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restrictions after boiling for more than 5 minutes on site or pasteurization at a temperature of

+850C for 30 minutes.

In the epizootic focus, buildings and pastures where sick animals are kept, as well as vehicles

with manure, are disinfected and disinfested every week. Special clothing and rubber boots of

farm workers are disinfected with formaldehyde vapors in a special chamber.

The border of the danger zone (3 km) is determined, and disinfection, disinfestation and

vaccination of conditionally healthy animals are carried out there to prevent the spread of the

disease. Until the quarantine is lifted, susceptible animals are clinically examined daily in all

livestock farms, regardless of their form of ownership. In the surveillance zone (10 km),

cattle are clinically examined daily and disinfested weekly.

Quarantine is lifted 30 days after the death or loss of the last sick or suspected animal from a

farm (population settlement) that is not healthy according to the ND of cattle, after all

measures specified in the current regulations have been carried out and a commission report

has been submitted confirming their completeness and quality.Карантин бекор қилингач 1

йилга чеклов қўйилади ва шу муддатда қорамолларни гўштга топширишдан ташқари,

носоғлом пункт ҳудудидан чиқариш ва реализация қилиш тақиқланади.

REFERENCES:

1. Salimov X.S., Qambarov A.A., Salimov I.X. Darslik. Epizootologiya va infeksion

kasalliklar - Toshkent, 2021.

2. Х.С.Салимов., С.Х.Абдалимов., Р.А.Исматова “Қорамолларнинг нодуляр дерматит.

2018 йил 26-июнь.Ветеринария маслаҳати.

3. Арипов А.И., Фесенко Л.М., Арипов О.А., Исмоилова Н.И., Муҳамедиярова Р.Г.

Клиник Лаборатория Диагностика бўйича қўлланма Тошкент -2007.- Б. 12-56.

4. Архипов Н.И., Чевелев С.Ф., Брагин Г.И.и соавт. Нодулярный дерматит/

Патологоанатомическая диагностика вирусных инфекций животных: Справочное

издание// М.: Нодулярный дерматит: ДИВА-стратегия // БИО.- 2017. - № 4.- С. 14-

16.

5. Борисевич С.В., Сизикова Т.Е., Петров А.А., Карулин А.В., Лебедев В.Н.

“Нодулярный дерматит: появление новой поксвирусной

инфекции

в

россии”/ФГБУ

«48

Центральный

научно-исследовательский

институт»

Министерства обороны Российской Федерации, Сергиев Посад, Российская

Федерация 2018. - С. 5-11.

6. Вацаев Ш.В., Черных О.Ю., Лысенко А.А., Хахов Л.А. Эпизоотологическая

ситуация по нодулярному дерматиту крупного рогатого скота в Чеченской

Республике /- Текст: непосредственный // Труды, Кубанский государственный

аграрный университет.- Вып. 5.- 2016.-С. 140-145.


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page 1184

7. Закутский Н. И., Балышев В. М., Юрков С. Г. [и др.]. Нодулярный дерматит

крупного рогатого скота: характеристика возбудителя болезни, распространение,

диагностика и меры борьбы (обзор литературы) /- Текст: непосредственный //

Ветеринарный врач. - 2016.- № 4. С. 3-12.

References

Salimov X.S., Qambarov A.A., Salimov I.X. Darslik. Epizootologiya va infeksion kasalliklar - Toshkent, 2021.

Х.С.Салимов., С.Х.Абдалимов., Р.А.Исматова “Қорамолларнинг нодуляр дерматит. 2018 йил 26-июнь.Ветеринария маслаҳати.

Арипов А.И., Фесенко Л.М., Арипов О.А., Исмоилова Н.И., Муҳамедиярова Р.Г. Клиник Лаборатория Диагностика бўйича қўлланма Тошкент -2007.- Б. 12-56.

Архипов Н.И., Чевелев С.Ф., Брагин Г.И.и соавт. Нодулярный дерматит/ Патологоанатомическая диагностика вирусных инфекций животных: Справочное издание// М.: Нодулярный дерматит: ДИВА-стратегия // БИО.- 2017. - № 4.- С. 14-16.

Борисевич С.В., Сизикова Т.Е., Петров А.А., Карулин А.В., Лебедев В.Н. “Нодулярный дерматит: появление новой поксвирусной инфекции в россии”/ФГБУ «48 Центральный научно-исследовательский институт» Министерства обороны Российской Федерации, Сергиев Посад, Российская Федерация 2018. - С. 5-11.

Вацаев Ш.В., Черных О.Ю., Лысенко А.А., Хахов Л.А. Эпизоотологическая ситуация по нодулярному дерматиту крупного рогатого скота в Чеченской Республике /- Текст: непосредственный // Труды, Кубанский государственный аграрный университет.- Вып. 5.- 2016.-С. 140-145.

Закутский Н. И., Балышев В. М., Юрков С. Г. [и др.]. Нодулярный дерматит крупного рогатого скота: характеристика возбудителя болезни, распространение, диагностика и меры борьбы (обзор литературы) /- Текст: непосредственный // Ветеринарный врач. - 2016.- № 4. С. 3-12.