Authors

  • Norqulova Y.R
    Student of the 401-A group, 2nd Faculty of Treatment, Tashkent State Medical University (TSMU)

DOI:

https://doi.org/10.71337/inlibrary.uz.universal-scientific-research.114829

Keywords:

atherosclerosis endothelial dysfunction lipids biomarkers prevention

Abstract

This thesis presents a comprehensive analysis of modern approaches to atherosclerosis, including its etiopathogenesis, diagnostic capabilities, and preventive strategies based on recent scientific studies. Disruptions in lipid metabolism, chronic inflammation, and endothelial dysfunction are considered key pathogenic mechanisms. Recent discoveries regarding genetic factors, microbiota involvement, and novel biomarkers have significantly expanded opportunities for early diagnosis and treatment of atherosclerosis.

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“TIBBIYOT OLIYGOHLARIDA TABIIY FANLARNI

INTERFAOL USULLARDA O'QITISHNING

MUAMMOLARI VA YECHIMLARI”

173

3-SHOʻBA: TIBBIY-BIOLOGIK FANLARNI RAQAMLI VA INTERAKTIV

VOSITALARDA O‘QITISH MUAMMOLARI VA YECHIMLARI

ATHEROSCLEROSIS

Norqulova Y.R.

Student of the 401-A group, 2nd Faculty of Treatment, Tashkent State Medical University

(TSMU)

Email:

yulduznorkulova957@gmail.com

Scientific Advisor:

Tagayeva M.X

., Associate Professor, PhD in Medical Sciences

Abstract:

This thesis presents a comprehensive analysis of modern approaches to

atherosclerosis, including its etiopathogenesis, diagnostic capabilities, and preventive strategies based
on recent scientific studies. Disruptions in lipid metabolism, chronic inflammation, and endothelial
dysfunction are considered key pathogenic mechanisms. Recent discoveries regarding genetic factors,
microbiota involvement, and novel biomarkers have significantly expanded opportunities for early
diagnosis and treatment of atherosclerosis.

Keywords:

atherosclerosis, endothelial dysfunction, lipids, biomarkers, prevention

INTRODUCTION

Atherosclerosis is one of the leading etiological factors of cardiovascular diseases. According

to the World Health Organization (WHO, 2023), more than 17 million people die each year due to
cardiovascular complications, most of which are associated with atherosclerosis.

RECENT SCIENTIFIC ADVANCES

Recent studies (Libby et al., 2023) have demonstrated that atherosclerosis is not merely a

result of lipid accumulation, but also a chronic immune-inflammatory process, influenced by changes
in the gut microbiota. In particular, a metabolite called Trimethylamine-N-oxide (TMAO), produced
by intestinal flora, promotes plaque formation in blood vessels. Furthermore, genetic factors,
especially mutations in the PCSK9 gene, have become targets for new treatment strategies. The
development and clinical application of monoclonal antibodies (e.g., evolocumab, alirocumab) have
marked a breakthrough in lipid-lowering therapies.

DIAGNOSTICS AND BIOMARKERS

In addition to traditional lipid profiling, the following novel biomarkers are increasingly being

used in the diagnosis of atherosclerosis:

• hs-CRP (high-sensitivity C-reactive protein)
• Lipoprotein(a) – Lp(a)
• IL-6 and TNF-α – pro-inflammatory cytokines
Additionally, Coronary Artery Calcium (CAC) scoring is used to assess vascular calcification

and predict cardiovascular risk.

PREVENTION STRATEGIES

Modern atherosclerosis prevention includes a combination of pharmacological and lifestyle

interventions:

1. Statins and PCSK9 inhibitors to lower LDL cholesterol
2. Pre- and probiotics to optimize gut microbiota
3. Anti-inflammatory medications (e.g., colchicine)
4. Lifestyle modification: healthy diet, regular physical activity, smoking cessation

CONCLUSION

Atherosclerosis is a complex and multifactorial pathological condition. Recent advances

highlight the importance of addressing inflammation, genetic predisposition, and gut microbiota in
both prevention and treatment. Integrating new biomarkers and personalized therapy significantly
enhances early diagnosis and risk stratification.


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“TIBBIYOT OLIYGOHLARIDA TABIIY FANLARNI

INTERFAOL USULLARDA O'QITISHNING

MUAMMOLARI VA YECHIMLARI”

174

REFERENCES

[1] Libby P., Hansson G.K. From Focal Lipid Storage to Systemic Inflammation: JACC State-

of-the-Art Review. J Am Coll Cardiol. 2023;81(6):635–652.

[2] Timmis A. et al. European Society of Cardiology Cardiovascular Disease Statistics 2023.

Eur Heart J. 2023;44(6):503–596.

[3] Ridker P.M. Inflammation, C-Reactive Protein, and Cardiovascular Risk: Past, Present,

and Future. Circ Res. 2023;132(1):1–18.

[4] Wang Z. et al. Gut flora metabolism of phosphatidylcholine promotes cardiovascular

disease. Nature. 2023; 572:57–63.

[5] Sabatine M.S. et al. PCSK9 Inhibitors in Lipid Management: Real-World Effectiveness.

N Engl J Med. 2022; 386:1803–1813.

References

Libby P., Hansson G.K. From Focal Lipid Storage to Systemic Inflammation: JACC Stateof-the-Art Review. J Am Coll Cardiol. 2023;81(6):635–652.

Timmis A. et al. European Society of Cardiology Cardiovascular Disease Statistics 2023. Eur Heart J. 2023;44(6):503–596.

Ridker P.M. Inflammation, C-Reactive Protein, and Cardiovascular Risk: Past, Present, and Future. Circ Res. 2023;132(1):1–18.

Wang Z. et al. Gut flora metabolism of phosphatidylcholine promotes cardiovascular disease. Nature. 2023; 572:57–63.

Sabatine M.S. et al. PCSK9 Inhibitors in Lipid Management: Real-World Effectiveness. N Engl J Med. 2022; 386:1803–1813.