Authors

  • Malika Narzilloyeva

DOI:

https://doi.org/10.71337/inlibrary.uz.science-research.73597

Keywords:

obesity epidemiology body mass index steroids insulin

Abstract

Obesity is one of the so-called "diseases of civilization", which also include cardiovascular diseases, malignant tumors, neuropsychiatric disorders, injuries, metabolic diseases (diabetes, gout). Thus, obesity is currently the scourge of developed and developing countries.

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OBESITY PANDEMIC OF THE 21ST CENTURY

Narzilloyeva Malika Shuhrat qizi

Department of Fundamental Medical Sciences of the Asian International University.

Bukhara, Uzbekistan.

https://doi.org/10.5281/zenodo.15093803

Abstract.

Obesity is one of the so-called "diseases of civilization", which also include

cardiovascular diseases, malignant tumors, neuropsychiatric disorders, injuries, metabolic

diseases (diabetes, gout). Thus, obesity is currently the scourge of developed and developing

countries.

Keywords:

obesity, epidemiology, div mass index, steroids, insulin

ПАНДЕМИЯ ОЖИРЕНИЯ XXI ВЕКА

Аннотация.

Ожирение — одна из так называемых «болезней цивилизации», к

которым также относятся сердечно-сосудистые заболевания, злокачественные опухоли,

нервно-психические расстройства, травмы, болезни обмена веществ (сахарный диабет,

подагра). Таким образом, ожирение в настоящее время является бичом развитых и

развивающихся стран.

Ключевые слова:

ожирение, эпидемиология, индекс массы тела, стероиды,

инсулин.

Obesity is a chronic disease characterized by excess accumulation of div fat, which

poses a health risk and is also a major risk factor for a number of other chronic conditions that

make up metabolic syndrome. Obesity increases the risk of developing atherosclerotic changes in

blood vessels, hypertension, heart attacks and strokes, diabetes, cancer, as well as disability in

general and even death.

Excess weight has long ceased to be just an aesthetic problem. Today, obesity is

recognized by the World Health Organization as a non-communicable epidemic of the 21st

century. According to statistics, 2.2 billion people on the planet suffer from obesity - this is

almost ⅓ of the entire population.

Causes of Obesity

Whatever the prerequisites for the occurrence of excess div weight,

almost always the basis is an imbalance between the amount of food entering the div and its

expenditure on energy and anabolic processes. The div always tries to put extra calories "in

reserve" so that, if necessary, they can be used to maintain vital functions.

The fat cell depot is located in the subcutaneous fat tissue and internal organs of the

abdominal cavity. Only 5% of obesity cases are caused by metabolic disorders, all other cases


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occur due to overeating with low physical activity. Obesity can develop for a number of reasons

or a combination of them:

Overeating, especially fatty and carbohydrate foods. Irregular eating habits: rare and large

meals, snacks before bed. Hereditary disorders of the activity of lipolysis and lipogenesis

enzymes.

Diseases of the endocrine glands (hypothyroidism, polycystic ovary disease, pancreatic

tumors, Itsenko-Cushing's disease, etc.).

Low physical activity. Excessive food consumption against the background of

psychoemotional disorders. Taking hormonal drugs (steroids, estrogens, progesterone, insulin).

Degrees of obesity

Currently, there are many classifications of obesity that doctors

around the world use in their practice. The most common way to find out about the presence of

obesity is to calculate the div mass index (BMI). It is applicable to people from 18 to 65 years

old:

BMI <18.5 (low) - indicates underweight;

BMI from 18.5 to 24.9 (normal) - is considered optimal.

BMI from 25.0 to 29.9 (high) - indicates excess div weight and the presence of

predisposition to obesity.

BMI from 30.0 to 34.9 (high) - grade I obesity.

BMI from 35.0 to 39.9 (very high) - grade II obesity.

BMI from 40 and above (excessively high) - grade III obesity (super obesity).

Types of obesity

According to the location of fat deposits in the div, one can talk about the type of

obesity:

Abdominal (upper or android) - typical for the male part of the population and is called

"apple-shaped". It is considered the most dangerous type, because with it, excess fat deposits

occur mainly in the visceral, vital organs, which can lead to damage to the cardiovascular,

respiratory and digestive tracts.

Femorogluteal (lower) - typical for the female part of the population and is called "pear-

shaped". With this type, excess fat is deposited in the gluteal and femoral parts. Not as dangerous

as abdominal, but can lead to the development of arthrosis of the lower extremities, dysfunction

of the spine and lead to venous insufficiency.

Intermediate (mixed) - fat deposits are evenly distributed throughout the div.

Diagnostics for excess weight includes:

Anthropometry — measuring height, chest circumference, waist, hips, and other div

parameters. Helps calculate the div obesity index.


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Bioimpedancemetry — assessing the electrical resistance of div tissues. This is a more

accurate method for calculating the proportion of fat in the div.

Blood tests - general, sugar, biochemistry, hormones. Allows you to evaluate the work of

internal organs, the functions of the endocrine system.

ECG (electrocardiography), blood pressure measurement - to assess the state of the

cardiovascular system.

Treatment

: The following methods are used to correct weight: Diet therapy is the first

stage of treatment. A diet for obesity is created by a nutritionist taking into account the energy

expenditure of the patient's div. It is important to create a diet so that there is a calorie deficit,

but the div receives enough proteins, fats and carbohydrates, as well as vitamins and minerals.

Drug therapy for obesity treatment Treatment of obesity with drugs is an extreme

measure and is aimed at suppressing the feeling of hunger, accelerating the appearance of a

feeling of satiety. In the presence of psychoemotional disorders, appropriate medications that

affect eating behavior may be prescribed. Often, drugs that prevent the div from absorbing fats

and carbohydrates are used. Drug therapy is administered to patients whose div mass index is

greater than 30, and diet and exercise do not produce visible results within 3-4 months.

Surgical treatment of obesity Bariatric (metabolic) surgery has been recognized by

doctors and patients in the fight against excess weight for many decades. The methods of

surgical treatment of obesity are varied and allow the doctor to choose an individual approach to

treatment for each person.

The following are surgical procedures for the treatment of obesity: Intragastric balloon

placement,

gastric bypass,

longitudinal gastrectomy,

gastric bypass,

biliopancreatic bypass.

All these methods of treating obesity have proven themselves to be safe and effective.

Each of them has its own indications and contraindications.

Indications for surgery are a BMI of more than 40 kg/m2 in the absence of an effect from

conservative treatment, as well as the presence of type 2 diabetes mellitus (DM) in combination

with obesity (as directed by an endocrinologist), even if the BMI is 35 kg/m2 or slightly lower.

Preventive measures

Prevention of excess weight is aimed at eliminating the factors that

provoke fat deposition. It is important to change your lifestyle so that the div receives as much

energy as it spends. General recommendations are as follows: Lead an active lifestyle - at least

once a day walk in the fresh air, do light sports. If the patient has a sedentary job, take a break


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every hour for 5 minutes to stretch, do light gymnastics - this will prevent not only obesity, but

also diseases of the joints and spine.

REFERENCES

1.

Development Initiative, Global Nutrition Report 2017: Nourishing the SDGs,

Development Initiatives, Bristol, UK, 2017.

2.

N. D. Ford, S. A. Patel, and K. M. V. Narayan, “Obesity in lowand middle-income

countries: burden, drivers, and emerging challenges,” Annual Review of Public Health,

vol. 38, no. 1, pp. 145–164, 2017.

3.

D. J. Hoffman, Obesity in Developing Countries: Causes and Implications, Rutgers

University, New Brunswick, NY, USA, 2001.

4.

S. Assari and M. Bazargan, “Baseline obesity increases 25-year risk of mortality due to

cerebrovascular disease: role of race,” International Journal of Environmental Research

and Public Health, vol. 16, no. 19, p. 3705, 2019.

5.

M. Agha and R. Agha, “+e rising prevalence of obesity: part a—impact on public health,”

International Journal of Surgery Oncology, vol. 2, no. 7, p. e17, 2017.

6.

G. M. Al Kibria, K. Swasey, M. Z. Hasan, A. Sharmeen, and B. Day, “Prevalence and

factors associated with underweight, overweight and obesity among women of

reproductive age in India,” Global Health Research Policy, vol. 4, no. 1, p. 24, 2019

7.

Al Kibria GM. Prevalence and Factors Affecting Underweight, Overweight and Obesity

Using Asian and World Health Organization Cutoffs Among Adults in Nepal: Analysis of

the Demographic and Health Survey 2016. Obes Res Clin Pract (2019) 13(2):129–36. doi:

10.1016/j.orcp.2019.01.006

8.

Mbogori T, Kimmel K, Zhang M, Kandiah J, Wang Y. Nutrition Transition and Double

Burden of Malnutrition in Africa: A Case Study of Four Selected Countries With Different

Social Economic Development. AIMS Public Health (2020) 7:425–39. doi:

10.3934/publichealth.2020035

9.

Swinburn BA, Sacks G, Hall KD, McPherson K, Finegood DT, Moodie ML, et al. The

GlobalObesity Pandemic: Shaped byGlobalDrivers and Local Environments. Lancet

(2011) 378(9793):804–14. doi: 10.1016/S0140-6736(11)60813-1

10.

WHO Consultation on Obesity (1999: Geneva, Switzerland) & World Health Organization.

Obesity: Preventing and Managing the Global Epidemic. Report of a WHO Consultation.

World Health Organ Tech Rep Ser (2000) 894:i–xii, 1-253.

11.

Bray GA, Kim KK, Wilding JWorld Obesity Federation. Obesity: A Chronic Relapsing

Progressive Disease Process. A Position Statement of the World Obesity Federation. Obes

Rev (2017) 18(7):715–23.doi: 10.1111/obr.12551


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12.

Lee SJ, Shin SW. Mechanisms, Pathophysiology, and Management of Obesity. N Engl J

Med (2017) 376(15):1491–2. doi: 10.1056/NEJMc1701944

13.

Sacks FM, Bray GA, Carey VJ, Smith SR, Ryan DH, Anton SD, et al. Comparison of

Weight-Loss Diets With Different Compositions of Fat, Protein, and Carbohydrates. N

Engl J Med (2009) 360(9):859–73. doi: 10.1056/NEJMoa0804748

14.

Singer-Englar T, Barlow G, Mathur R. Obesity, Diabetes, and the Gut Microbiome: An

Updated Review. Expert Rev Gastroenterol Hepatol (2019) 13(1):3–15. doi:

10.1080/17474124.2019.1543023

15.

Singh RK, Kumar P, Mahalingam K. Molecular Genetics of Human Obesity: A

Comprehensive Review. C R Biol (2017) 340(2):87–108. doi: 10.1016/ j.crvi.2016.11.007

16.

Lopomo A, Burgio E, Migliore L. Epigenetics of Obesity. Prog Mol Biol Transl Sci (2016)

140:151–84. doi: 10.1016/bs.pmbts.2016.02.002

17.

Dubern B. Genetics and Epigenetics of Obesity: Keys to Understand. Rev Prat (2019)

69(9):1016–9.

References

Development Initiative, Global Nutrition Report 2017: Nourishing the SDGs, Development Initiatives, Bristol, UK, 2017.

N. D. Ford, S. A. Patel, and K. M. V. Narayan, “Obesity in lowand middle-income countries: burden, drivers, and emerging challenges,” Annual Review of Public Health, vol. 38, no. 1, pp. 145–164, 2017.

D. J. Hoffman, Obesity in Developing Countries: Causes and Implications, Rutgers University, New Brunswick, NY, USA, 2001.

S. Assari and M. Bazargan, “Baseline obesity increases 25-year risk of mortality due to cerebrovascular disease: role of race,” International Journal of Environmental Research and Public Health, vol. 16, no. 19, p. 3705, 2019.

M. Agha and R. Agha, “+e rising prevalence of obesity: part a—impact on public health,” International Journal of Surgery Oncology, vol. 2, no. 7, p. e17, 2017.

G. M. Al Kibria, K. Swasey, M. Z. Hasan, A. Sharmeen, and B. Day, “Prevalence and factors associated with underweight, overweight and obesity among women of reproductive age in India,” Global Health Research Policy, vol. 4, no. 1, p. 24, 2019

Al Kibria GM. Prevalence and Factors Affecting Underweight, Overweight and Obesity Using Asian and World Health Organization Cutoffs Among Adults in Nepal: Analysis of the Demographic and Health Survey 2016. Obes Res Clin Pract (2019) 13(2):129–36. doi: 10.1016/j.orcp.2019.01.006

Mbogori T, Kimmel K, Zhang M, Kandiah J, Wang Y. Nutrition Transition and Double Burden of Malnutrition in Africa: A Case Study of Four Selected Countries With Different Social Economic Development. AIMS Public Health (2020) 7:425–39. doi: 10.3934/publichealth.2020035

Swinburn BA, Sacks G, Hall KD, McPherson K, Finegood DT, Moodie ML, et al. The GlobalObesity Pandemic: Shaped byGlobalDrivers and Local Environments. Lancet (2011) 378(9793):804–14. doi: 10.1016/S0140-6736(11)60813-1

WHO Consultation on Obesity (1999: Geneva, Switzerland) & World Health Organization. Obesity: Preventing and Managing the Global Epidemic. Report of a WHO Consultation. World Health Organ Tech Rep Ser (2000) 894:i–xii, 1-253.

Bray GA, Kim KK, Wilding JWorld Obesity Federation. Obesity: A Chronic Relapsing Progressive Disease Process. A Position Statement of the World Obesity Federation. Obes Rev (2017) 18(7):715–23.doi: 10.1111/obr.12551

Lee SJ, Shin SW. Mechanisms, Pathophysiology, and Management of Obesity. N Engl J Med (2017) 376(15):1491–2. doi: 10.1056/NEJMc1701944

Sacks FM, Bray GA, Carey VJ, Smith SR, Ryan DH, Anton SD, et al. Comparison of Weight-Loss Diets With Different Compositions of Fat, Protein, and Carbohydrates. N Engl J Med (2009) 360(9):859–73. doi: 10.1056/NEJMoa0804748

Singer-Englar T, Barlow G, Mathur R. Obesity, Diabetes, and the Gut Microbiome: An Updated Review. Expert Rev Gastroenterol Hepatol (2019) 13(1):3–15. doi: 10.1080/17474124.2019.1543023

Singh RK, Kumar P, Mahalingam K. Molecular Genetics of Human Obesity: A Comprehensive Review. C R Biol (2017) 340(2):87–108. doi: 10.1016/ j.crvi.2016.11.007

Lopomo A, Burgio E, Migliore L. Epigenetics of Obesity. Prog Mol Biol Transl Sci (2016) 140:151–84. doi: 10.1016/bs.pmbts.2016.02.002

Dubern B. Genetics and Epigenetics of Obesity: Keys to Understand. Rev Prat (2019) 69(9):1016–9.