Authors

  • Dilora Azimova

DOI:

https://doi.org/10.71337/inlibrary.uz.science-research.137931

Keywords:

To assess the morphological changes in lymph nodes under experimentally induced stress-related secondary immunosuppression.

Abstract

Chronic stress disrupts immune homeostasis through neuroendocrine pathways and leads to increased susceptibility to infectious, inflammatory, and autoimmune disorders. Lymph nodes, as the main peripheral immune organs, are directly affected by stress hormones such as glucocorticoids and catecholamines. Identifying histological alterations in lymph nodes is essential for predicting immunodepression and designing prophylactic measures.

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Oktabr, 2025-Yil

293

STRUCTURAL AND FUNCTIONAL ALTERATIONS OF LYMPH NODES IN STRESS-

RELATED IMMUNODEFICIENCY

Azimova Dilora Alijon qizi

Asian International University.

diloraxanum1993@gmail.com

https://doi.org/10.5281/zenodo.17446969

Reasurch relevance:

Chronic stress disrupts immune homeostasis through neuroendocrine

pathways and leads to increased susceptibility to infectious, inflammatory, and autoimmune
disorders. Lymph nodes, as the main peripheral immune organs, are directly affected by stress
hormones such as glucocorticoids and catecholamines. Identifying histological alterations in lymph
nodes is essential for predicting immunodepression and designing prophylactic measures.

Objective:

To assess the morphological changes in lymph nodes under experimentally

induced stress-related secondary immunosuppression.

Materials and Methods

Rodent models were exposed to:

Noise and light stress,

Nutritional restriction,

High-dose corticosteroids,

Sleep disturbance.

The duration of exposure varied between 7 and 28 days.
Lymph nodes were examined using:

Hematoxylin–eosin staining,

Sudan III and Van Gieson methods,

PAS reaction,

Phase-contrast and electron microscopy,

Immunohistochemical markers (CD8+, caspase-3, Ki-67).

Results

The following pathological alterations were identified:

Regression or disappearance of cortical follicles

Reduced germinal center proliferation

Atrophy of T-cell zones in the paracortex

Delay in plasma cell differentiation

Early stromal sclerosis in the reticular framework

Decreased numbers of macrophages in lymphatic sinuses

Venous and capillary stasis

Partial replacement of lymphoid follicles by adipose tissue.
These changes confirm the suppressive effect of stress mediators on lymphopoiesis, immune

surveillance, and lymphatic microcirculation. The findings indicate a direct role of apoptosis, stromal
collapse, and inhibited proliferation in the pathogenesis of secondary immunodeficiency.




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Oktabr, 2025-Yil

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References

1.

Khaitov R., Pinegin B. (2020). Pathogenesis of Stress-Induced Immunodeficiency.
Immunology Journal.

2.

Karimova D. (2022). Lymph Node Histology and Stress Impact. Uzbek Medical Journal.

3.

Sapolsky R. (2018). Neuroendocrine Stress Mechanisms. J Neuroimmunol.

References

Khaitov R., Pinegin B. (2020). Pathogenesis of Stress-Induced Immunodeficiency. Immunology Journal.

Karimova D. (2022). Lymph Node Histology and Stress Impact. Uzbek Medical Journal.

Sapolsky R. (2018). Neuroendocrine Stress Mechanisms. J Neuroimmunol.