Authors

  • Dilora Azimova

DOI:

https://doi.org/10.71337/inlibrary.uz.science-research.137984

Abstract

The increasing prevalence of psychosocial stressors, malnutrition, and environmental factors has significantly contributed to the development of secondary immunodeficiency worldwide. The thymus, as the central organ of T-lymphocyte maturation, is one of the most stress-sensitive structures. Understanding thymic morphological damage under stress is crucial for early diagnosis, prevention, and development of therapeutic strategies in clinical immunology and pathomorphology.

background image

2025

OKTABR

NEW RENAISSANCE

INTERNATIONAL SCIENTIFIC AND PRACTICAL CONFERENCE

VOLUME 2

|

ISSUE 10

200

HISTOLOGICAL REMODELING OF THE THYMUS UNDER STRESS-INDUCED

SECONDARY IMMUNODEFICIENCY

Azimova Dilora Alijon qizi

Asian International University.

diloraxanum1993@gmail.com

https://doi.org/10.5281/zenodo.17449826


Relevance

The increasing prevalence of psychosocial stressors, malnutrition, and environmental

factors has significantly contributed to the development of secondary immunodeficiency
worldwide.

The thymus, as the central organ of T-lymphocyte maturation, is one of the most stress-

sensitive structures. Understanding thymic morphological damage under stress is crucial for early
diagnosis, prevention, and development of therapeutic strategies in clinical immunology and
pathomorphology.

Objective

To investigate stress-induced histological and cellular changes in the thymus based on

experimental models simulating secondary immunodeficiency.

Materials and Methods

Experimental stress conditions were modeled in rats using immobilization, cold exposure

(4–6 °C), corticosteroid injections, and sleep deprivation over 14–30 days. After euthanasia,
thymic samples were collected and analyzed with:

Hematoxylin–eosin staining,

PAS reaction,

Van Gieson method,

Immunohistochemistry (CD3, CD4, CD8, caspase-3),

Light and electron microscopy.

Morphometric measurements were used to quantify cortical and medullary alterations.

Results

Significant degenerative and apoptotic processes were revealed:

Massive lymphocyte apoptosis and necrosis in the cortical layer;

Medullary lymphocyte depletion;

Vacuolization, wrinkling, and shrinkage of Hassall’s corpuscles;

Stromal disintegration and perivascular edema;

Microcirculatory disorders such as congestion, hemorrhage, and stasis;

Fatty infiltration and diffuse thymic atrophy.

The main pathogenic mechanism was linked to glucocorticoid-induced caspase-mediated

apoptosis of thymocytes. Structural reduction of lymphoid reserves can predispose to infection
and autoimmunity.




background image

2025

OKTABR

NEW RENAISSANCE

INTERNATIONAL SCIENTIFIC AND PRACTICAL CONFERENCE

VOLUME 2

|

ISSUE 10

201

References

1.

Sapolsky R. (2018). Stress and Immunosuppression in Mammals. Journal of

Neuroimmunology.

2.

Petrova L. (2021). Morphological Changes of the Thymus Under Stress. Morphology

Journal.

3.

Glaser R., Kiecolt-Glaser J. (2019). Experimental Stress Models in Rats. Brain Behav

Immun.

References

Sapolsky R. (2018). Stress and Immunosuppression in Mammals. Journal of Neuroimmunology.

Petrova L. (2021). Morphological Changes of the Thymus Under Stress. Morphology Journal.

Glaser R., Kiecolt-Glaser J. (2019). Experimental Stress Models in Rats. Brain Behav Immun.