One of the many problems of modern allergology, pulmonology and occupational pathology is alveolitis of various origins, often leading to diagnostic errors (they are mistaken for asthma or COPD) and, as a result, to incorrect treatment [2, 8,14,18,20]. To date, the number of patients with exogenous allergic alveolitis (EAA) is increasing significantly and is about 3% of all pulmonological patients, which requires a special study and selection of the most informative methods for verifying various variants of this disease [1, 3,5,6,24]. Allergic pneumopathies under the general name - EAA arise as a result of an allergic reaction of the lung tissue to intense and prolonged inhalations of a certain kind of antigens (AH) [2,4,6,9,11]. EAA is a diffuse granulomatous inflammatory process of the alveoli and interstitial tissue of the lungs, developing under the influence of intense and prolonged inhalation of predominantly organic water-insoluble antigens.