The effect of a decoction of liquorice root and an infusion of Helichrysum flowers on the processes of lipid peroxidation in chronic toxic hepatitis was studied. It has been established that a decoction of liquorice root and an infusion of Sandy immortelle flower in chronic poisoning with heliotrin has an antioxidant effect. In terms of hepatoprotective action, herbal preparations are not inferior to the well-known hepatoprotector carsil.
The influence of stimusol (extract radix Glycyrrhiza+mumie) on lipid peroxidation processes and
synthesis of gall m the liver at chronic toxic hepatitis were researched. It was determined that stimusol during
chronic poisoning with heliotrine made antioxidant and choleretic action. Normalize of gall contains. It didn't
give up to famous, silibor by hepatoprotector action
In experiments on white rats it was established that administration of heliotrine during a month induced severe damage to the liver characterized by enhancement of lipid peroxidation, a decrease of enzyme activities - superoxide dismutase and catalase. Liquorice, immortelle flowers and mumie inhibited manifestation of heliotrine hepatitis and exerted antioxidant action. In combined use of natural compounds their antioxidant action increases.
The effect of a decoction of liquorice root and an infusion of Helichrysum flowers on the processes of lipid peroxidation in chronic toxic hepatitis was studied. It has been established that a decoction of liquorice root and an infusion of Sandy immortelle flower in chronic poisoning with heliotrin has an antioxidant effect. In terms of hepatoprotective action, herbal preparations are not inferior to the well-known hepatoprotector silibor.
The effect of immunomodulin and phytin substances on lipid peroxidation was studied in experimental toxic hepatitis in rats. The results showed that the tested substances showed an antioxidant effect when poisoned with tetrachloromethane. The combined use of these substances led to an increase in the antioxidant effect.
Жигарнинг углеводлар алмашинувида қатнашиши, ўзига хос аҳамиятга эга. Унинг асосий фаолиятидан ири бу гепатоцитларда глюкозани гликогенга айлантириб тўплаш ва гликоген заҳиралари камайганда уни қонга ажратиш ҳисобланади. Жигар касалликларида эса, фермент системаларининг фаолликларини сусайиши ўз навбатида гликоген ҳосил бўлишини камайишига, сут кислотасини миқдорини ортишига олиб келиб углеводлар алмашинувини издан чиқишига сабаб бўлади. Гепатопротектор хусусиятга эга бўлган дори воситаларини излаш, самарали таъсирини ўрганиш ва амалиётга тадбиқ этиш долзарб муаммолардан бири бўлиб ҳисобланади.