This study assesses the activity of autoimmune reactions in adolescent children with HIV encephalopathy of varying severity. According to the results of the study, it was found that in children with HIV encephalopathy, symptomatic neurocognitive impairments are associated with the activation of cellular and humoral immunity - mainly natural killers, apoptosis markers, antibody-producing and reactive lymphocytes. In this regard, a hypothesis was put forward about the role of autoimmune reactions in the formation of HIV encephalopathy. Possible ways of damage to the CNS can be both vasculitis and autoimmune damage to the nervous tissue itself. Based on this hypothesis, during the study, a study was made of the concentration of autoantibodies - Abs to myelin, APA (vasculitis induction), Abs to cell nuclei (ANA) and their components - 1- and 2-stranded DNA.
This article presents the results of a study of patients with diabetic encephalopathy, which were divided into 2 groups. Patients of group 1 received the drug Dialipon Turbo on the background of traditional therapy, patients of group 2 received only traditional therapy. In the dynamics of therapy, the scales used to study the function of the autonomic system were used.
Проблема инсульта имеет большую медико социальную значимость из-за их широкой распространенности, высокой частотой смертности и инвалидизации. Токсическая энцефалопатия часто возникает у людей, злоупотребляющих алкоголь. Считается, что умеренное употребление алкоголя снижает риск развития инфаркта мозга, но много пьющие люди предрасположены к ишемическим инсультам вследствие развития у них ангиопатии, кардиомиопатий, аритмий и наклонности к гиперкоагуляции. Течение и восстановление утраченных неврологических функций после перенесенного инсульта при токсической энцефалопатии зависит от сложного взаимодействия морфологических, гемодинамических, психологических и иммунологических изменений. Все эти изменения в настояшее время изучены
недостаточно.
The largest number of drug-resistant epilepsies occur in childhood, which is associated primarily with early maturation of excitatory glutamatergic and delay the formation of the brake GABAergic systems. This can cause the development of resistance due to the predominance of excitatory neuron pool of brake and confirmation of neural network theory. The theory of neural networks is confirmed not only in the study of drug-resistant epilepsies of childhood.
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