Асфиксия, перенесенная в родах и в периоде новорожденности, не проходит бесследно и оказывает негативное воздействие на развивающийся мозг ребенка различной степени тяжести (H.Boskabadi ,2010). Гипоксия внутриутробно вызывает компенсационный ответ в виде усиленного эритропоза, что приводит к высвобождению незрелых эритроцитов и большого количества нейтрофилов.
Во всем мире частота анемии колеблется от 25% до 50%, в развивающихся странах – от 35% до 75%, в развитых странах составляет 18%-20% Из-за высокой распространенности в Центральноазиатском регионе железодефицитную анемию относят к краевой патологии. У беременных ЖДА диагностируется у 40-90% и часто является причиной развития тяжелых осложнений гестации и течения родов, критических ситуаций, угрожающих жизни матери и ребенка.
This study investigated the comparative use of Cerebrolysin and Cortexin in the treatment of children with periventricular leukomalacia. Sick children were divided into two groups, and for 10 days, the drugs of patients of the first group were used Cortexin, and those of the second group were Cerebrolysin. As a result of the treatment, the patients underwent neurosonography and Doppler studies, there was an improvement in the general dynamics and M-echo of the brain structure in patients taking Cerebrolysin.
To study the characteristic features of the cellular composition of blood in patients with acute purulent odontogenic ostitis of the jaw. Material and methods: The data obtained during the examination and treatment of 42 patients with acute purulent odontogenic ostitis at the age of 7 to 9 years, who applied to the TGSI clinic, were studied. Results: In the patients, the indices of the red part of the blood were significantly lower than in the comparison group. A low level of hemoglobin can cause hemic tissue hypoxia and affect the state of the acid-base state of the blood, expressed in the form of acidosis, which activates anaerobic glycolysis. An increase in the level of lactic acid in cells can be one of the reasons for the disruption of local microcirculationand the accumulation of endogenous toxins. Conclusions: This pathology is accompanied by a shift in the indicators of the white part of the blood, the observed hemic hypoxia leads to an increase in the level of lactic acid in the cells, which may be one of the reasons for the disruption of local microcirculation and the accumulation of endogenous toxins.
The monograph presents modern aspects of syndialysis arterial hypotension: solved and unresolved problems of predicting, preventing and diagnosing this complication during dialysis. The characteristics of clinical and pathogenetic features, diagnosis, treatment, prognosis and prevention of syndialysis arterial hypotension are given. The data of the dynamics of clinical, functional and laboratory studies of syndialytic arterial hypotension in patients on dialysis are presented. An algorithm for verifying the status of hydration and preventing syndialysis hypotension is presented. The monograph is intended for nephrologists, doctors of related specialties, masters and students of medical institutes.
Целью работы является исследование состояния системы гемостаза и сосудистого эндотелия у новорожденных с гипоксическими поражениями нервной системы.